Reconsolidation of a cocaine-associated stimulus requires amygdalar protein kinase A.
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Post-retrieval extinction as reconsolidation interference: methodological issues or boundary conditions?Anisomycin in the medial prefrontal cortex reduces reconsolidation of cocaine-associated memories in the rat self-administration modelA Memory Retrieval-Extinction Procedure to Prevent Drug Craving and RelapseAmygdala Dopamine Receptors Are Required for the Destabilization of a Reconsolidating Appetitive Memory(1,2).Exercise as a potential treatment for drug abuse: evidence from preclinical studiesCritical role for protein kinase A in the acquisition of gregarious behavior in the desert locust.D-cycloserine reduces the context specificity of pavlovian extinction of cocaine cues through actions in the nucleus accumbens.Dopamine D1 and D3 receptors mediate reconsolidation of cocaine memories in mouse models of drug self-administrationActivation of exchange protein activated by cAMP in the rat basolateral amygdala impairs reconsolidation of a memory associated with self-administered cocaine.Learning to forget: manipulating extinction and reconsolidation processes to treat addictionEpigenetic mechanisms of memory formation and reconsolidation.Aberrant learning and memory in addiction.PKMζ maintains drug reward and aversion memory in the basolateral amygdala and extinction memory in the infralimbic cortex.β-Arrestin-biased signaling mediates memory reconsolidation.Brain pathways to recovery from alcohol dependenceA novel UCS memory retrieval-extinction procedure to inhibit relapse to drug seeking.Reconsolidation of a cocaine associated memory requires DNA methyltransferase activity in the basolateral amygdala.Neurobiological dissociation of retrieval and reconsolidation of cocaine-associated memoryDopamine D3 receptors regulate reconsolidation of cocaine memory.Extracellular signal-regulated kinase in the basolateral amygdala, but not the nucleus accumbens core, is critical for context-response-cocaine memory reconsolidation in rats.Addiction: a drug-induced disorder of memory reconsolidation.Targeting extinction and reconsolidation mechanisms to combat the impact of drug cues on addiction.Phosphoproteomic Analysis Reveals a Novel Mechanism of CaMKIIα Regulation Inversely Induced by Cocaine Memory Extinction versus ReconsolidationThe reinstatement model of drug relapse: recent neurobiological findings, emerging research topics, and translational research.Involvement of amygdalar protein kinase A, but not calcium/calmodulin-dependent protein kinase II, in the reconsolidation of cocaine-related contextual memories in rats.Post-retrieval extinction attenuates cocaine memories.Reconsolidation of drug memories.Neural and psychological mechanisms underlying compulsive drug seeking habits and drug memories--indications for novel treatments of addiction.Knockdown of zif268 in the posterior dorsolateral striatum does not enduringly disrupt a response memory of a rewarded T-maze task.Inhibition of protein synthesis but not β-adrenergic receptors blocks reconsolidation of a cocaine-associated cue memory.The Naturally Occurring Compound Garcinia Indica Selectively Impairs the Reconsolidation of a Cocaine-Associated Memory.Distinctive roles for amygdalar CREB in reconsolidation and extinction of fear memory.Pavlovian conditioned approach, extinction, and spontaneous recovery to an audiovisual cue paired with an intravenous heroin infusion.Glycogen synthase kinase 3β in the basolateral amygdala is critical for the reconsolidation of cocaine reward memory.Activation of adenylate cyclase-cyclic AMP-protein kinase A signaling by corticotropin-releasing factor within the dorsolateral bed nucleus of the stria terminalis is involved in pain-induced aversion.The effect of postretrieval extinction of nicotine pavlovian memories in rats trained to self-administer nicotine.Post-retrieval extinction training enhances or hinders the extinction of morphine-induced conditioned place preference in rats dependent on the retrieval-extinction interval.
P2860
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P2860
Reconsolidation of a cocaine-associated stimulus requires amygdalar protein kinase A.
description
2010 nî lūn-bûn
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2010 թուականի Մարտին հրատարակուած գիտական յօդուած
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2010 թվականի մարտին հրատարակված գիտական հոդված
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2010年の論文
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2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
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2010年论文
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name
Reconsolidation of a cocaine-associated stimulus requires amygdalar protein kinase A.
@ast
Reconsolidation of a cocaine-associated stimulus requires amygdalar protein kinase A.
@en
type
label
Reconsolidation of a cocaine-associated stimulus requires amygdalar protein kinase A.
@ast
Reconsolidation of a cocaine-associated stimulus requires amygdalar protein kinase A.
@en
prefLabel
Reconsolidation of a cocaine-associated stimulus requires amygdalar protein kinase A.
@ast
Reconsolidation of a cocaine-associated stimulus requires amygdalar protein kinase A.
@en
P2093
P2860
P1476
Reconsolidation of a cocaine-associated stimulus requires amygdalar protein kinase A.
@en
P2093
Hayde Sanchez
Jane R Taylor
Jennifer J Quinn
Mary M Torregrossa
P2860
P304
P356
10.1523/JNEUROSCI.3149-09.2010
P407
P577
2010-03-01T00:00:00Z