Reconsolidation of a cocaine-associated stimulus requires amygdalar protein kinase A. - Wikidata - wikimedia - TriplyDB

Reconsolidation of a cocaine-associated stimulus requires amygdalar protein kinase A.

Reconsolidation of a cocaine-associated stimulus requires amygdalar protein kinase A.

http://www.wikidata.org/entity/Q33897247

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Post-retrieval extinction as reconsolidation interference: methodological issues or boundary conditions?Anisomycin in the medial prefrontal cortex reduces reconsolidation of cocaine-associated memories in the rat self-administration model A Memory Retrieval-Extinction Procedure to Prevent Drug Craving and Relapse Amygdala Dopamine Receptors Are Required for the Destabilization of a Reconsolidating Appetitive Memory(1,2).Exercise as a potential treatment for drug abuse: evidence from preclinical studies Critical role for protein kinase A in the acquisition of gregarious behavior in the desert locust.D-cycloserine reduces the context specificity of pavlovian extinction of cocaine cues through actions in the nucleus accumbens.Dopamine D1 and D3 receptors mediate reconsolidation of cocaine memories in mouse models of drug self-administration Activation of exchange protein activated by cAMP in the rat basolateral amygdala impairs reconsolidation of a memory associated with self-administered cocaine.Learning to forget: manipulating extinction and reconsolidation processes to treat addiction Epigenetic mechanisms of memory formation and reconsolidation.Aberrant learning and memory in addiction.PKMζ maintains drug reward and aversion memory in the basolateral amygdala and extinction memory in the infralimbic cortex.β-Arrestin-biased signaling mediates memory reconsolidation.Brain pathways to recovery from alcohol dependence A novel UCS memory retrieval-extinction procedure to inhibit relapse to drug seeking.Reconsolidation of a cocaine associated memory requires DNA methyltransferase activity in the basolateral amygdala.Neurobiological dissociation of retrieval and reconsolidation of cocaine-associated memory Dopamine D3 receptors regulate reconsolidation of cocaine memory.Extracellular signal-regulated kinase in the basolateral amygdala, but not the nucleus accumbens core, is critical for context-response-cocaine memory reconsolidation in rats.Addiction: a drug-induced disorder of memory reconsolidation.Targeting extinction and reconsolidation mechanisms to combat the impact of drug cues on addiction.Phosphoproteomic Analysis Reveals a Novel Mechanism of CaMKIIα Regulation Inversely Induced by Cocaine Memory Extinction versus Reconsolidation The reinstatement model of drug relapse: recent neurobiological findings, emerging research topics, and translational research.Involvement of amygdalar protein kinase A, but not calcium/calmodulin-dependent protein kinase II, in the reconsolidation of cocaine-related contextual memories in rats.Post-retrieval extinction attenuates cocaine memories.Reconsolidation of drug memories.Neural and psychological mechanisms underlying compulsive drug seeking habits and drug memories--indications for novel treatments of addiction.Knockdown of zif268 in the posterior dorsolateral striatum does not enduringly disrupt a response memory of a rewarded T-maze task.Inhibition of protein synthesis but not β-adrenergic receptors blocks reconsolidation of a cocaine-associated cue memory.The Naturally Occurring Compound Garcinia Indica Selectively Impairs the Reconsolidation of a Cocaine-Associated Memory.Distinctive roles for amygdalar CREB in reconsolidation and extinction of fear memory.Pavlovian conditioned approach, extinction, and spontaneous recovery to an audiovisual cue paired with an intravenous heroin infusion.Glycogen synthase kinase 3β in the basolateral amygdala is critical for the reconsolidation of cocaine reward memory.Activation of adenylate cyclase-cyclic AMP-protein kinase A signaling by corticotropin-releasing factor within the dorsolateral bed nucleus of the stria terminalis is involved in pain-induced aversion.The effect of postretrieval extinction of nicotine pavlovian memories in rats trained to self-administer nicotine.Post-retrieval extinction training enhances or hinders the extinction of morphine-induced conditioned place preference in rats dependent on the retrieval-extinction interval.

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P2860

Reconsolidation of a cocaine-associated stimulus requires amygdalar protein kinase A.

http://www.wikidata.org/entity/Q33897247

description

2010 nî lūn-bûn

@nan

2010 թուականի Մարտին հրատարակուած գիտական յօդուած

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2010 թվականի մարտին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

@zh-hk

2010年論文

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2010年論文

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2010年论文

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name

Reconsolidation of a cocaine-associated stimulus requires amygdalar protein kinase A.

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Reconsolidation of a cocaine-associated stimulus requires amygdalar protein kinase A.

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type

label

Reconsolidation of a cocaine-associated stimulus requires amygdalar protein kinase A.

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Reconsolidation of a cocaine-associated stimulus requires amygdalar protein kinase A.

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prefLabel

Reconsolidation of a cocaine-associated stimulus requires amygdalar protein kinase A.

@ast

Reconsolidation of a cocaine-associated stimulus requires amygdalar protein kinase A.

@en

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JNEUROSCI.3149-09.2010

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Journal of Neuroscience

P1476

Reconsolidation of a cocaine-associated stimulus requires amygdalar protein kinase A.

@en

P2093

Hayde Sanchez

http://www.w3.org/2001/XMLSchema#string

Jane R Taylor

http://www.w3.org/2001/XMLSchema#string

Jennifer J Quinn

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Mary M Torregrossa

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P2860

Impulsivity resulting from frontostriatal dysfunction in drug abuse: implications for the control of behavior by reward-related stimuli.

Both protein kinase A and mitogen-activated protein kinase are required in the amygdala for the macromolecular synthesis-dependent late phase of long-term potentiation.

Renewal of drug seeking by contextual cues after prolonged extinction in rats.

The neurobiology of learning and memory: some reminders to remember.

Reconsolidation: the advantage of being refocused.

Applying extinction research and theory to cue-exposure addiction treatments.

Noradrenergic signaling in the amygdala contributes to the reconsolidation of fear memory: treatment implications for PTSD.

Molecular mechanisms of memory reconsolidation.

Drug addiction and the memory systems of the brain.

Regulated transcription of the immediate-early gene Zif268: mechanisms and gene dosage-dependent function in synaptic plasticity and memory formation.

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4401-4407

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10.1523/JNEUROSCI.3149-09.2010

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2010-03-01T00:00:00Z

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