Critical role for PAR1 in kallikrein 6-mediated oligodendrogliopathy.
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Involvement of Kallikrein-Related Peptidases in Normal and Pathologic ProcessesThe progeroid gene BubR1 regulates axon myelination and motor function.Protease-activated receptor-1 activation by granzyme B causes neurotoxicity that is augmented by interleukin-1βAstrocyte heterogeneity across the brain and spinal cord occurs developmentally, in adulthood and in response to demyelination.Kallikrein cascades in traumatic spinal cord injury: in vitro evidence for roles in axonopathy and neuron degeneration.Differential expression of multiple kallikreins in a viral model of multiple sclerosis points to unique roles in the innate and adaptive immune response.The thrombin receptor is a critical extracellular switch controlling myelination.Genetic targeting of protease activated receptor 2 reduces inflammatory astrogliosis and improves recovery of function after spinal cord injuryKallikreins - The melting pot of activity and functionInterplay between exercise and dietary fat modulates myelinogenesis in the central nervous system.Identification of brain-enriched proteins in the cerebrospinal fluid proteome by LC-MS/MS profiling and mining of the Human Protein Atlas.Astrocyte barriers to neurotoxic inflammation.Reactive gliosis and the multicellular response to CNS damage and disease.Kallikrein Promotes Inflammation in Human Dental Pulp Cells Via Protease-Activated Receptor-1.Targeting the thrombin receptor modulates inflammation and astrogliosis to improve recovery after spinal cord injuryInterference with protease-activated receptor 1 does not reduce damage to subventricular zone cells of immature rodent brain following exposure to blood or blood plasma.Technical comments on rodent spinal cord injuries models.Kallikrein-related peptidase 6 exacerbates disease in an autoimmune model of multiple sclerosis.Protease activated receptor 2 controls myelin development, resiliency and repair.Nexilin Regulates Oligodendrocyte Progenitor Cell Migration and Remyelination and Is Negatively Regulated by Protease-Activated Receptor 1/Ras-Proximate-1 Signaling Following Subarachnoid Hemorrhage.The Thrombin Receptor Restricts Subventricular Zone Neural Stem Cell Expansion and Differentiation.Kallikrein-related peptidase 6 orchestrates astrocyte form and function through proteinase activated receptor-dependent mechanisms
P2860
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P2860
Critical role for PAR1 in kallikrein 6-mediated oligodendrogliopathy.
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2013 nî lūn-bûn
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2013 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Critical role for PAR1 in kallikrein 6-mediated oligodendrogliopathy.
@ast
Critical role for PAR1 in kallikrein 6-mediated oligodendrogliopathy.
@en
type
label
Critical role for PAR1 in kallikrein 6-mediated oligodendrogliopathy.
@ast
Critical role for PAR1 in kallikrein 6-mediated oligodendrogliopathy.
@en
prefLabel
Critical role for PAR1 in kallikrein 6-mediated oligodendrogliopathy.
@ast
Critical role for PAR1 in kallikrein 6-mediated oligodendrogliopathy.
@en
P2093
P2860
P356
P1433
P1476
Critical role for PAR1 in kallikrein 6-mediated oligodendrogliopathy
@en
P2093
Hyesook Yoon
Isobel A Scarisbrick
Maja Radulovic
P2860
P304
P356
10.1002/GLIA.22534
P577
2013-07-08T00:00:00Z