Altered responsiveness to chemokines due to targeted disruption of SHIP. - Wikidata - wikimedia - TriplyDB

Altered responsiveness to chemokines due to targeted disruption of SHIP.

Altered responsiveness to chemokines due to targeted disruption of SHIP.

http://www.wikidata.org/entity/Q33903127

about

Subcellular localization of Grb2 by the adaptor protein Dok-3 restricts the intensity of Ca2+ signaling in B cells Overexpression of SH2-containing inositol phosphatase 2 results in negative regulation of insulin-induced metabolic actions in 3T3-L1 adipocytes via its 5'-phosphatase catalytic activity The inositol polyphosphate 5-phosphatase, PIPP, Is a novel regulator of phosphoinositide 3-kinase-dependent neurite elongation.SHIP1, an SH2 domain containing polyinositol-5-phosphatase, regulates migration through two critical tyrosine residues and forms a novel signaling complex with DOK1 and CRKL A mutation in synaptojanin 2 causes progressive hearing loss in the ENU-mutagenised mouse strain Mozart.Effect of monocyte chemoattractant protein-1 on murine bone marrow cells: proliferation, colony-forming ability and signal transduction pathway involved.SH2-inositol phosphatase 1 negatively influences early megakaryocyte progenitors Regulation of cell polarity during eukaryotic chemotaxis: the chemotactic compass.Inositol phospholipid signaling and the biology of natural killer cells.Aberrant regulation of hematopoiesis by T cells in BAZF-deficient mice Characterization of AQX-1125, a small-molecule SHIP1 activator: Part 1. Effects on inflammatory cell activation and chemotaxis in vitro and pharmacokinetic characterization in vivo Characterization of AQX-1125, a small-molecule SHIP1 activator: Part 2. Efficacy studies in allergic and pulmonary inflammation models in vivo.The inositol phosphatase SHIP controls Salmonella enterica serovar Typhimurium infection in vivo SHIP represses lung inflammation and inhibits mammary tumor metastasis in BALB/c mice.Modulation of Hematopoietic Chemokine Effects In Vitro and In Vivo by DPP-4/CD26.Survival of monocytes and macrophages and their role in health and disease.CXCL12 induces hepatic stellate cell contraction through a calcium-independent pathway.Role of inositol phospholipid signaling in natural killer cell biology.Role of inositol poly-phosphatases and their targets in T cell biology.CXCL12/CXCR4 axis in the pathogenesis of acute lymphoblastic leukemia (ALL): a possible therapeutic target.Inflammation in Alzheimer's Disease and Molecular Genetics: Recent Update.Negative regulation of chemokine receptor signaling and B-cell chemotaxis by p66Shc.Visualization of negative signaling in B cells by quantitative confocal microscopy.SHIP1-expressing mesenchymal stem cells regulate hematopoietic stem cell homeostasis and lineage commitment during aging.SHIP deficiency enhances HSC proliferation and survival but compromises homing and repopulation.IL-6 increases B-cell IgG production in a feed-forward proinflammatory mechanism to skew hematopoiesis and elevate myeloid production.Compound heterozygosity for Pten and SHIP augments T-dependent humoral immune responses and cytokine production by CD(4+) T cells.

P2860

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P2860

Altered responsiveness to chemokines due to targeted disruption of SHIP.

http://www.wikidata.org/entity/Q33903127

description

1999 nî lūn-bûn

@nan

1999 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

1999 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

1999年の論文

@ja

1999年論文

@yue

1999年論文

@zh-hant

1999年論文

@zh-hk

1999年論文

@zh-mo

1999年論文

@zh-tw

1999年论文

@wuu

name

Altered responsiveness to chemokines due to targeted disruption of SHIP.

@ast

Altered responsiveness to chemokines due to targeted disruption of SHIP.

@en

type

label

Altered responsiveness to chemokines due to targeted disruption of SHIP.

@ast

Altered responsiveness to chemokines due to targeted disruption of SHIP.

@en

prefLabel

Altered responsiveness to chemokines due to targeted disruption of SHIP.

@ast

Altered responsiveness to chemokines due to targeted disruption of SHIP.

@en

P1433

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P1433

Journal of Clinical Investigation

P1476

Altered responsiveness to chemokines due to targeted disruption of SHIP.

@en

P2093

C D Helgason

http://www.w3.org/2001/XMLSchema#string

C H Kim

http://www.w3.org/2001/XMLSchema#string

G Hangoc

http://www.w3.org/2001/XMLSchema#string

G Krystal

http://www.w3.org/2001/XMLSchema#string

H E Broxmeyer

http://www.w3.org/2001/XMLSchema#string

R K Humphries

http://www.w3.org/2001/XMLSchema#string

S Cooper

http://www.w3.org/2001/XMLSchema#string

S Yew

http://www.w3.org/2001/XMLSchema#string

P2860

RANTES-activated human T lymphocytes. A role for phosphoinositide 3-kinase

The lymphocyte chemoattractant SDF-1 is a ligand for LESTR/fusin and blocks HIV-1 entry

The 145-kDa protein induced to associate with Shc by multiple cytokines is an inositol tetraphosphate and phosphatidylinositol 3,4,5-triphosphate 5-phosphatase

A highly efficacious lymphocyte chemoattractant, stromal cell-derived factor 1 (SDF-1)

Interleukin-3 induces the association of the inositol 5-phosphatase SHIP with SHP2

Human chemokines: an update

CK beta-11/macrophage inflammatory protein-3 beta/EBI1-ligand chemokine is an efficacious chemoattractant for T and B cells

p150Ship, a signal transduction molecule with inositol polyphosphate-5-phosphatase activity

Macrophage-inflammatory protein-3 beta/EBI1-ligand chemokine/CK beta-11, a CC chemokine, is a chemoattractant with a specificity for macrophage progenitors among myeloid progenitor cells

Multiple forms of an inositol polyphosphate 5-phosphatase form signaling complexes with Shc and Grb2

P304

1751-1759

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scholarly article

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10.1172/JCI7310

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P407

P433

12

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P478

104

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1999-12-01T00:00:00Z

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10606629

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409879

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