A hierarchical role for classical pathway complement proteins in the clearance of apoptotic cells in vivo. - Wikidata - wikimedia - TriplyDB

A hierarchical role for classical pathway complement proteins in the clearance of apoptotic cells in vivo.

A hierarchical role for classical pathway complement proteins in the clearance of apoptotic cells in vivo.

http://www.wikidata.org/entity/Q33912964

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Cytokine-associated neutrophil extracellular traps and antinuclear antibodies in Plasmodium falciparum infected children under six years of age Complement classical pathway components are all important in clearance of apoptotic and secondary necrotic cells Elastase-mediated phosphatidylserine receptor cleavage impairs apoptotic cell clearance in cystic fibrosis and bronchiectasis Paths reunited: Initiation of the classical and lectin pathways of complement activation PPAR-delta senses and orchestrates clearance of apoptotic cells to promote tolerance Epidermal growth factor-like domain repeat of stabilin-2 recognizes phosphatidylserine during cell corpse clearance C1q and mannose binding lectin engagement of cell surface calreticulin and CD91 initiates macropinocytosis and uptake of apoptotic cells ATP-binding cassette transporter A7 enhances phagocytosis of apoptotic cells and associated ERK signaling in macrophages Phosphatidylserine receptor and apoptosis: consequences of a non-ingested meal Links between complement deficiency and apoptosis.Genetic epidemiology: systemic lupus erythematosus Complement in Lupus Nephritis: New Perspectives Systemic Lupus Erythematosus and Deficiencies of Early Components of the Complement Classical Pathway Cell death in the pathogenesis of immune-mediated diseases: the role of HMGB1 and DAMP-PAMP complexes C1q binds phosphatidylserine and likely acts as a multiligand-bridging molecule in apoptotic cell recognition Neutrophils regulate tissue Neutrophilia in inflammation via the oxidant-modified lipid lysophosphatidylserine Signaling via macrophage G2A enhances efferocytosis of dying neutrophils by augmentation of Rac activity.A new triggering receptor expressed on myeloid cells (Trem) family member, Trem-like 4, binds to dead cells and is a DNAX activation protein 12-linked marker for subsets of mouse macrophages and dendritic cells The endothelial deprotection hypothesis for lupus pathogenesis: the dual role of C1q as a mediator of clearance and regulator of endothelial permeability The role of nucleotides in apoptotic cell clearance: implications for disease pathogenesis.ACAT inhibition reduces the progression of preexisting, advanced atherosclerotic mouse lesions without plaque or systemic toxicity.Complement receptor 3 (CD11b/CD18) is implicated in the elimination of β-amyloid peptides.Complement in glomerular injury.C1q differentially modulates phagocytosis and cytokine responses during ingestion of apoptotic cells by human monocytes, macrophages, and dendritic cells.Complement C4 maintains peripheral B-cell tolerance in a myeloid cell dependent manner.Isoflurane promotes phagocytosis of apoptotic neutrophils through AMPK-mediated ADAM17/Mer signaling Endogenous NO upon estradiol-17β stimulation and NO donor differentially regulate mitochondrial S-nitrosylation in endothelial cells.Plasminogen promotes macrophage phagocytosis in mice Gene expression profiling of dendritic cells reveals important mechanisms associated with predisposition to Staphylococcus infections A polymorphism in the complement component C1qA correlates with prolonged response following rituximab therapy of follicular lymphoma Apoptotic cell-based therapies against transplant rejection: role of recipient's dendritic cells.Wound-healing defect of CD18(-/-) mice due to a decrease in TGF-beta1 and myofibroblast differentiation.The complement cascade as a mediator of tissue growth and regeneration.Native polymeric forms of properdin selectively bind to targets and promote activation of the alternative pathway of complement Characterization of the effects of cross-linking of macrophage CD44 associated with increased phagocytosis of apoptotic PMN.Regulation of phagocytosis by TAM receptors and their ligands.Pathogenesis of systemic lupus erythematosus (SLE).The alternative pathway is critical for pathogenic complement activation in endotoxin- and diet-induced atherosclerosis in low-density lipoprotein receptor-deficient mice.The phosphatidylserine receptor: a crucial molecular switch?The macrophage and the apoptotic cell: an innate immune interaction viewed simplistically?

P2860

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P2860

A hierarchical role for classical pathway complement proteins in the clearance of apoptotic cells in vivo.

http://www.wikidata.org/entity/Q33912964

description

2000 nî lūn-bûn

@nan

2000 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2000 թվականի օգոստոսին հրատարակված գիտական հոդված

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2000年の論文

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2000年論文

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2000年論文

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2000年論文

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2000年論文

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2000年論文

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2000年论文

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name

A hierarchical role for classi ...... ce of apoptotic cells in vivo.

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A hierarchical role for classi ...... ce of apoptotic cells in vivo.

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A hierarchical role for classi ...... ce of apoptotic cells in vivo.

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A hierarchical role for classi ...... ce of apoptotic cells in vivo.

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A hierarchical role for classi ...... ce of apoptotic cells in vivo.

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A hierarchical role for classi ...... ce of apoptotic cells in vivo.

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Journal of Experimental Medicine

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A hierarchical role for classi ...... ce of apoptotic cells in vivo.

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Andrews M

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Botto M

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Carroll MC

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Carugati A

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Cook HT

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Taylor PR

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P2860

Human CD14 mediates recognition and phagocytosis of apoptotic cells

Immunogenicity of apoptotic cells in vivo: role of antigen load, antigen-presenting cells, and cytokines

Immature dendritic cells phagocytose apoptotic cells via alphavbeta5 and CD36, and cross-present antigens to cytotoxic T lymphocytes

Macrophages that have ingested apoptotic cells in vitro inhibit proinflammatory cytokine production through autocrine/paracrine mechanisms involving TGF-beta, PGE2, and PAF

Exposure of phosphatidylserine on the surface of apoptotic lymphocytes triggers specific recognition and removal by macrophages

Complement-dependent clearance of apoptotic cells by human macrophages.

Autoantigens as substrates for apoptotic proteases: implications for the pathogenesis of systemic autoimmune disease.

Studies of group B streptococcal infection in mice deficient in complement component C3 or C4 demonstrate an essential role for complement in both innate and acquired immunity.

Recognition of oxidatively damaged and apoptotic cells by an oxidized low density lipoprotein receptor on mouse peritoneal macrophages: role of membrane phosphatidylserine.

Thrombospondin cooperates with CD36 and the vitronectin receptor in macrophage recognition of neutrophils undergoing apoptosis.

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359-366

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10.1084/JEM.192.3.359

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3

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192

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12386899

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10934224

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apoptotic process

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2193213

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