Roles for Nox4 in the contractile response of bovine pulmonary arteries to hypoxia - Wikidata - wikimedia - TriplyDB

Roles for Nox4 in the contractile response of bovine pulmonary arteries to hypoxia

Roles for Nox4 in the contractile response of bovine pulmonary arteries to hypoxia

http://www.wikidata.org/entity/Q33917083

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Pulmonary endothelial cell NOX Oxygen-coupled redox regulation of the skeletal muscle ryanodine receptor-Ca2+ release channel by NADPH oxidase 4 The Nox4 inhibitor GKT137831 attenuates hypoxia-induced pulmonary vascular cell proliferation Contribution of NADPH oxidase to membrane CD38 internalization and activation in coronary arterial myocytes Roles for redox mechanisms controlling protein kinase G in pulmonary and coronary artery responses to hypoxia Key Role of ROS in the Process of 15-Lipoxygenase/15-Hydroxyeicosatetraenoiccid-Induced Pulmonary Vascular Remodeling in Hypoxia Pulmonary Hypertension.NADPH oxidase-derived ROS and the regulation of pulmonary vessel tone Contribution of Nrf2 to Atherogenic Phenotype Switching of Coronary Arterial Smooth Muscle Cells Lacking CD38 Gene.Role of nicotinamide adenine dinucleotide phosphate-reduced oxidase proteins in Pseudomonas aeruginosa-induced lung inflammation and permeability.Estrogen normalizes perinatal nicotine-induced hypertensive responses in adult female rat offspring.Molecular mechanisms of the crosstalk between mitochondria and NADPH oxidase through reactive oxygen species-studies in white blood cells and in animal models.Redox regulation of guanylate cyclase and protein kinase G in vascular responses to hypoxia.Differential roles of NADPH oxidases in vascular physiology and pathophysiology Reactive oxygen species as therapeutic targets in pulmonary hypertension.The human Nox4: gene, structure, physiological function and pathological significance.NADPH oxidases-do they play a role in TRPC regulation under hypoxia?ICAM-1 cytoplasmic tail regulates endothelial glutathione synthesis through a NOX4/PI3-kinase-dependent pathway.Dehydroepiandrosterone promotes pulmonary artery relaxation by NADPH oxidation-elicited subunit dimerization of protein kinase G 1α.Redox regulation of responses to hypoxia and NO-cGMP signaling in pulmonary vascular pathophysiology.Recent advances in oxygen sensing and signal transduction in hypoxic pulmonary vasoconstriction.High insulin-induced down-regulation of Erk-1/IGF-1R/FGFR-1 signaling is required for oxidative stress-mediated apoptosis of adipose-derived stem cells.Biphasic augmentation of alpha-adrenergic contraction by plumbagin in rat systemic arteries.Evidence for novel aspects of Nox4 oxidase regulation of mitochondrial function and peroxide generation in an endothelial cell model of senescence.Hypoxia-augmented constriction of deep femoral artery mediated by inhibition of eNOS in smooth muscle.Hypoxic pulmonary vasoconstriction

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Roles for Nox4 in the contractile response of bovine pulmonary arteries to hypoxia

http://www.wikidata.org/entity/Q33917083

description

2010 nî lūn-bûn

@nan

2010 թուականի Մարտին հրատարակուած գիտական յօդուած

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2010 թվականի մարտին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

@zh-hk

2010年論文

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2010年論文

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2010年论文

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name

Roles for Nox4 in the contractile response of bovine pulmonary arteries to hypoxia

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Roles for Nox4 in the contractile response of bovine pulmonary arteries to hypoxia

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type

label

Roles for Nox4 in the contractile response of bovine pulmonary arteries to hypoxia

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Roles for Nox4 in the contractile response of bovine pulmonary arteries to hypoxia

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Roles for Nox4 in the contractile response of bovine pulmonary arteries to hypoxia

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Roles for Nox4 in the contractile response of bovine pulmonary arteries to hypoxia

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AJPHEART.01228.2009

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American Journal of Physiology - Heart and Circulatory Physiology

P1476

Roles for Nox4 in the contractile response of bovine pulmonary arteries to hypoxia

@en

P2093

Mansoor Ahmad

http://www.w3.org/2001/XMLSchema#string

Melissa R Kelly

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Michael S Wolin

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Xiangmin Zhao

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P2860

A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

Effects of hypoxia on relationships between cytosolic and mitochondrial NAD(P)H redox and superoxide generation in coronary arterial smooth muscle

Prolonged hypoxia increases ROS signaling and RhoA activation in pulmonary artery smooth muscle and endothelial cells.

ROS-dependent signaling mechanisms for hypoxic Ca(2+) responses in pulmonary artery myocytes.

Inhibition of Nox-4 activity by plumbagin, a plant-derived bioactive naphthoquinone.

Hypoxic pulmonary vasoconstriction: redox regulation of O2-sensitive K+ channels by a mitochondrial O2-sensor in resistance artery smooth muscle cells.

O2 sensing is preserved in mice lacking the gp91 phox subunit of NADPH oxidase.

Peroxide generation by p47phox-Src activation of Nox2 has a key role in protein kinase C-induced arterial smooth muscle contraction.

Heme oxygenase-1 induction modulates hypoxic pulmonary vasoconstriction through upregulation of ecSOD.

Mitochondrial-derived hydrogen peroxide inhibits relaxation of bovine coronary arterial smooth muscle to hypoxia through stimulation of ERK MAP kinase.

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10.1152/AJPHEART.01228.2009

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6

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