The PPAR alpha agonist gemfibrozil is an ineffective treatment for spinal cord injured mice. - Wikidata - wikimedia - TriplyDB

The PPAR alpha agonist gemfibrozil is an ineffective treatment for spinal cord injured mice.

The PPAR alpha agonist gemfibrozil is an ineffective treatment for spinal cord injured mice.

http://www.wikidata.org/entity/Q33919749

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Systemic iron chelation results in limited functional and histological recovery after traumatic spinal cord injury in rats.Spinal cord injury causes chronic liver pathology in rats.PPAR agonists as therapeutics for CNS trauma and neurological diseases Opioid administration following spinal cord injury: implications for pain and locomotor recovery.Role of PPAR γ in the Differentiation and Function of Neurons Macrophage Transcriptional Profile Identifies Lipid Catabolic Pathways That Can Be Therapeutically Targeted after Spinal Cord Injury.Palmitoylethanolamide is a disease-modifying agent in peripheral neuropathy: pain relief and neuroprotection share a PPAR-alpha-mediated mechanism.Neuroprotective Effect of Ginsenoside Rd in Spinal Cord Injury Rats.

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The PPAR alpha agonist gemfibrozil is an ineffective treatment for spinal cord injured mice.

http://www.wikidata.org/entity/Q33919749

description

2011 nî lūn-bûn

@nan

2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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The PPAR alpha agonist gemfibr ...... for spinal cord injured mice.

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P356

J.EXPNEUROL.2011.09.023

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Experimental Neurology

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The PPAR alpha agonist gemfibr ...... for spinal cord injured mice.

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P2093

A Todd Lash

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Akshata Almad

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Amy E Lovett-Racke

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Dana M McTigue

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Ping Wei

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P2860

CD8+ T cells cause disability and axon loss in a mouse model of multiple sclerosis

Safety issues and prospects for future generations of PPAR modulators

B cells produce pathogenic antibodies and impair recovery after spinal cord injury in mice.

PPAR-alpha and PPAR-gamma activators induce cholesterol removal from human macrophage foam cells through stimulation of the ABCA1 pathway.

Biphasic regulation of intracellular calcium by gemfibrozil contributes to inhibiting L6 myoblast differentiation: implications for clinical myotoxicity.

PPARalpha: energy combustion, hypolipidemia, inflammation and cancer

Transcriptional modulation of the immune response by peroxisome proliferator-activated receptor-{alpha} agonists in autoimmune disease.

Effects of chronic systemic treatment with peroxisome proliferator-activated receptor α activators on neuroinflammation induced by intracerebral injection of lipopolysaccharide in adult mice.

Molecular control of physiological and pathological T-cell recruitment after mouse spinal cord injury

Degenerative and regenerative mechanisms governing spinal cord injury.

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10.1016/J.EXPNEUROL.2011.09.023

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