PD-1/B7-H1 interaction contribute to the spontaneous acceptance of mouse liver allograft - Wikidata - wikimedia - TriplyDB

PD-1/B7-H1 interaction contribute to the spontaneous acceptance of mouse liver allograft

PD-1/B7-H1 interaction contribute to the spontaneous acceptance of mouse liver allograft

http://www.wikidata.org/entity/Q33921085

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Role of the PD-1 pathway in the immune response T-cell exhaustion in allograft rejection and tolerance The CD8 T-cell response during tolerance induction in liver transplantation The role of co-inhibitory signals in spontaneous tolerance of weakly mismatched transplants DAP12 deficiency in liver allografts results in enhanced donor DC migration, augmented effector T cell responses and abrogation of transplant tolerance.Programmed death-1/B7-H1 negative costimulation protects mouse liver against ischemia and reperfusion injury.PD-L1(hi) retinal pigment epithelium (RPE) cells elicited by inflammatory cytokines induce regulatory activity in uveitogenic T cells.Hepatic B7 homolog 1 expression is essential for controlling cold ischemia/reperfusion injury after mouse liver transplantation CD39 deficiency in murine liver allografts promotes inflammatory injury and immune-mediated rejection.MiR-152 may silence translation of CaMK II and induce spontaneous immune tolerance in mouse liver transplantation.The role of coinhibitory signaling pathways in transplantation and tolerance Rejection triggers liver transplant tolerance: Involvement of mesenchyme-mediated immune control mechanisms in mice.Tolerance-inducing strategies in islet transplantation CD28 family and chronic rejection: "to belatacept...And beyond!".Tolerance induction using nanoparticles bearing HY peptides in bone marrow transplantation.Liver transplantation in the mouse: Insights into liver immunobiology, tissue injury, and allograft tolerance PD-1/PD-L1 Interaction Maintains Allogeneic Immune Tolerance Induced by Administration of Ultraviolet B-Irradiated Immature Dendritic Cells.Lessons and limits of mouse models.Identification of microRNAs involved in acute rejection and spontaneous tolerance in murine hepatic allografts.Therapeutic targeting of B7-H1 in breast cancer.The nature of activatory and tolerogenic dendritic cell-derived signal II.Immunology in the liver--from homeostasis to disease.High-pressure carbon monoxide preserves rat kidney grafts from apoptosis and inflammation.Graft-infiltrating PD-L1(hi) cross-dressed dendritic cells regulate anti-donor T cell responses in mouse liver transplant tolerance.PD-1 expression on CD8+ T cells regulates their differentiation within lung allografts and is critical for tolerance induction.Adoptive transfer of allogeneic liver sinusoidal endothelial cells specifically inhibits T-cell responses to cognate stimuli.PD-1 Blockade Prevents the Development and Progression of Carcinogen-Induced Oral Premalignant Lesions.Orthotopic mouse liver transplantation to study liver biology and allograft tolerance.Mechanisms of Mixed Chimerism-Based Transplant Tolerance.Essential role of PDL1 expression on nonhematopoietic donor cells in acquired tolerance to vascularized cardiac allografts.Galectin-1 prolongs survival of mouse liver allografts from Flt3L-pretreated donors.The STATus of PD-L1 (B7-H1) on tolerogenic APCs.Polymorphism of costimulatory molecules (CTLA4, ICOS, PD.1 and CD28) and allogeneic hematopoietic stem cell transplantation in Iranian patients Identification of a novel biomarker gene set with sensitivity and specificity for distinguishing between allograft rejection and tolerance Checkpoint inhibitor use in two heart transplant patients with metastatic melanoma and review of high-risk populations

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PD-1/B7-H1 interaction contribute to the spontaneous acceptance of mouse liver allograft

http://www.wikidata.org/entity/Q33921085

description

2009 nî lūn-bûn

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2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2009 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

PD-1/B7-H1 interaction contribute to the spontaneous acceptance of mouse liver allograft

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PD-1/B7-H1 interaction contribute to the spontaneous acceptance of mouse liver allograft

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PD-1/B7-H1 interaction contribute to the spontaneous acceptance of mouse liver allograft

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PD-1/B7-H1 interaction contribute to the spontaneous acceptance of mouse liver allograft

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PD-1/B7-H1 interaction contribute to the spontaneous acceptance of mouse liver allograft

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PD-1/B7-H1 interaction contribute to the spontaneous acceptance of mouse liver allograft

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J.1600-6143.2009.02859.X

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American Journal of Transplantation

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PD-1/B7-H1 interaction contribute to the spontaneous acceptance of mouse liver allograft

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A Sugioka

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G Jiang

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H Yagita

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L Xie

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M Fujino

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P2860

Induced expression of PD-1, a novel member of the immunoglobulin gene superfamily, upon programmed cell death

Tumor-associated B7-H1 promotes T-cell apoptosis: a potential mechanism of immune evasion

PD-L2 is a second ligand for PD-1 and inhibits T cell activation

Apoptosis within spontaneously accepted mouse liver allografts: evidence for deletion of cytotoxic T cells and implications for tolerance induction

PD-1 inhibits antiviral immunity at the effector phase in the liver.

Insulin-induced remission in new-onset NOD mice is maintained by the PD-1-PD-L1 pathway.

Propagation of dendritic cell progenitors from normal mouse liver using granulocyte/macrophage colony-stimulating factor and their maturational development in the presence of type-1 collagen

The immunology of experimental liver transplantation in the rat.

Inhibition of T-cell responses by hepatic stellate cells via B7-H1-mediated T-cell apoptosis in mice.

Induction of immunological tolerance by porcine liver allografts.

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40-46

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