Association of hypometabolism and amyloid levels in aging, normal subjects
about
Multimodality imaging of Alzheimer disease and other neurodegenerative dementiasReduced glucose uptake and Aβ in brain regions with hyperintensities in connected white matterRegional Fluid-Attenuated Inversion Recovery (FLAIR) at 7 Tesla correlates with amyloid beta in hippocampus and brainstem of cognitively normal elderly subjects.Mapping 3-year changes in gray matter and metabolism in Aβ-positive nondemented subjects.Imaging brain effects of APOE4 in cognitively normal individuals across the lifespan.Early-Phase 11C-PiB PET in Amyloid Angiopathy-Related Symptomatic Cerebral Hemorrhage: Potential Diagnostic Value?β-Amyloid Deposition Is Associated with Decreased Right Prefrontal Activation during Task Switching among Cognitively Normal Elderly.Arterial spin labeling imaging reveals widespread and Aβ-independent reductions in cerebral blood flow in elderly apolipoprotein epsilon-4 carriers.Regional brain hypometabolism is unrelated to regional amyloid plaque burden.VoxelStats: A MATLAB Package for Multi-Modal Voxel-Wise Brain Image Analysis.Is amyloid-β harmful to the brain? Insights from human imaging studiesAmyloid-β and hyperphosphorylated tau synergy drives metabolic decline in preclinical Alzheimer's disease.Peripheral apoE isoform levels in cognitively normal APOE ε3/ε4 individuals are associated with regional gray matter volume and cerebral glucose metabolism.FDG-PET Contributions to the Pathophysiology of Memory Impairment.Comparison of [18F]Flutemetamol and [11C]Pittsburgh Compound-B in cognitively normal young, cognitively normal elderly, and Alzheimer's disease dementia individuals.Hippocampal and Clinical Trajectories of Mild Cognitive Impairment with Suspected Non-Alzheimer's Disease Pathology.Alzheimer Disease Signature Neurodegeneration and APOE Genotype in Mild Cognitive Impairment With Suspected Non-Alzheimer Disease Pathophysiology.Dynamic relationships between age, amyloid-β deposition, and glucose metabolism link to the regional vulnerability to Alzheimer's diseaseLocal and distant relationships between amyloid, tau and neurodegeneration in Alzheimer's Disease.Widespread brain tau and its association with ageing, Braak stage and Alzheimer's dementia.Alzheimer's Disease Biomarkers and Future Decline in Cognitive Normal Older Adults.Cerebral Amyloid Deposition and Dual-Tasking in Cognitively Normal, Mobility Unimpaired Older Adults.Longitudinal Positron Emission Tomography in Preventive Alzheimer's Disease Drug Trials, Critical Barriers from Imaging Science Perspective.Amyloid and tau signatures of brain metabolic decline in preclinical Alzheimer's disease.The impact of pulse pressure on cerebrovascular events leading to age-related cognitive decline.Does posterior cingulate hypometabolism result from disconnection or local pathology across preclinical and clinical stages of Alzheimer's disease?Metabolic brain networks in aging and preclinical Alzheimer's disease.Modulation of glucose metabolism and metabolic connectivity by β-amyloid.Longitudinal influence of microglial activation and amyloid on neuronal function in Alzheimer's disease.Asymmetries of amyloid-β burden and neuronal dysfunction are positively correlated in Alzheimer's disease.Elevated medial temporal lobe and pervasive brain tau-PET signal in normal participants.Synergistic interaction between APOE and family history of Alzheimer's disease on cerebral amyloid deposition and glucose metabolism
P2860
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P2860
Association of hypometabolism and amyloid levels in aging, normal subjects
description
2014 nî lūn-bûn
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2014 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年学术文章
@wuu
2014年学术文章
@zh-cn
2014年学术文章
@zh-hans
2014年学术文章
@zh-my
2014年学术文章
@zh-sg
2014年學術文章
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name
Association of hypometabolism and amyloid levels in aging, normal subjects
@ast
Association of hypometabolism and amyloid levels in aging, normal subjects
@en
type
label
Association of hypometabolism and amyloid levels in aging, normal subjects
@ast
Association of hypometabolism and amyloid levels in aging, normal subjects
@en
prefLabel
Association of hypometabolism and amyloid levels in aging, normal subjects
@ast
Association of hypometabolism and amyloid levels in aging, normal subjects
@en
P2093
P2860
P50
P1433
P1476
Association of hypometabolism and amyloid levels in aging, normal subjects
@en
P2093
Bradley Boeve
Lennon Jordan
Matthew L Senjem
Prashanthi Vemuri
Stephen D Weigand
Val J Lowe
P2860
P304
P356
10.1212/WNL.0000000000000467
P407
P577
2014-05-02T00:00:00Z