Suppression of class I and II histone deacetylases blunts pressure-overload cardiac hypertrophy. - Wikidata - wikimedia - TriplyDB

Suppression of class I and II histone deacetylases blunts pressure-overload cardiac hypertrophy.

Suppression of class I and II histone deacetylases blunts pressure-overload cardiac hypertrophy.

http://www.wikidata.org/entity/Q33929939

about

Histone deacetylase signaling in cardioprotection Histone deacetylase (HDAC) inhibitors attenuate cardiac hypertrophy by suppressing autophagy Regulation of cardiac gene expression by KLF15, a repressor of myocardin activity The many roles of histone deacetylases in development and physiology: implications for disease and therapy Hadp1, a newly identified pleckstrin homology domain protein, is required for cardiac contractility in zebrafish HDAC and HDAC Inhibitor: From Cancer to Cardiovascular Diseases Epigenetic regulation in cardiac fibrosis Signaling effectors underlying pathologic growth and remodeling of the heart Metabolic reprogramming by class I and II histone deacetylases Autophagy in cardiovascular biology Epigenetics: a new mechanism of regulation of heart failure?Emerging roles for histone deacetylases in pulmonary hypertension and right ventricular remodeling (2013 Grover Conference series)Targeting histone deacetylases: perspectives for epigenetic-based therapy in cardio-cerebrovascular disease Protein quality control and metabolism: bidirectional control in the heart Proteotoxicity: an underappreciated pathology in cardiac disease Dynamic link between histone H3 acetylation and an increase in the functional characteristics of human ESC/iPSC-derived cardiomyocytes Histone deacetylase-2 is a key regulator of diabetes- and transforming growth factor-beta1-induced renal injury HDAC4 and PCAF bind to cardiac sarcomeres and play a role in regulating myofilament contractile activity Histone deacetylases facilitate sodium/calcium exchanger up-regulation in adult cardiomyocytes Sequencing of mRNA identifies re-expression of fetal splice variants in cardiac hypertrophy.Histone deacetylase activity selectively regulates notch-mediated smooth muscle differentiation in human vascular cells.NET silencing by let-7i in postural tachycardia syndrome.Trichostatin a inhibits corneal haze in vitro and in vivo.EED orchestration of heart maturation through interaction with HDACs is H3K27me3-independent.Antifibrotic activity of an inhibitor of histone deacetylases in DOCA-salt hypertensive rats Redundant control of adipogenesis by histone deacetylases 1 and 2 HDAC class I inhibitor, Mocetinostat, reverses cardiac fibrosis in heart failure and diminishes CD90+ cardiac myofibroblast activation gp-91 mediates histone deacetylase inhibition-induced cardioprotection.Targeted deletion of NF-kappaB p50 diminishes the cardioprotection of histone deacetylase inhibition Chromatin regulation by Brg1 underlies heart muscle development and disease Class I HDACs regulate angiotensin II-dependent cardiac fibrosis via fibroblasts and circulating fibrocytes.Specific inhibition of HDAC4 in cardiac progenitor cells enhances myocardial repairs.HDAC inhibition attenuates inflammatory, hypertrophic, and hypertensive responses in spontaneously hypertensive rats.Disparate impact of butyroyloxymethyl diethylphosphate (AN-7), a histone deacetylase inhibitor, and doxorubicin in mice bearing a mammary tumor.Epigenetic mechanisms in heart failure pathogenesis Roles and targets of class I and IIa histone deacetylases in cardiac hypertrophy.Tubulin hyperacetylation is adaptive in cardiac proteotoxicity by promoting autophagy.Pressure overload-induced cardiac hypertrophy response requires janus kinase 2-histone deacetylase 2 signaling.HDAC inhibition elicits myocardial protective effect through modulation of MKK3/Akt-1.Epigenetic regulation of vascular smooth muscle cell proliferation and neointima formation by histone deacetylase inhibition.

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P2860

Suppression of class I and II histone deacetylases blunts pressure-overload cardiac hypertrophy.

http://www.wikidata.org/entity/Q33929939

description

2006 nî lūn-bûn

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2006 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2006 թվականի մայիսին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

Suppression of class I and II ...... -overload cardiac hypertrophy.

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Suppression of class I and II ...... -overload cardiac hypertrophy.

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type

label

Suppression of class I and II ...... -overload cardiac hypertrophy.

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Suppression of class I and II ...... -overload cardiac hypertrophy.

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prefLabel

Suppression of class I and II ...... -overload cardiac hypertrophy.

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Suppression of class I and II ...... -overload cardiac hypertrophy.

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Suppression of class I and II ...... -overload cardiac hypertrophy.

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Beverly A Rothermel

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Eric N Olson

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Guangrong Lu

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Joseph A Hill

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Kambeez Berenji

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Paul Tannous

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Yongli Kong

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P2860

Translating the Histone Code

HDAC6 is a microtubule-associated deacetylase

The neuron-restrictive silencer element-neuron-restrictive silencer factor system regulates basal and endothelin 1-inducible atrial natriuretic peptide gene expression in ventricular myocytes

Myosin heavy chain gene expression in human heart failure

Signal-dependent activation of the MEF2 transcription factor by dissociation from histone deacetylases

Cardiac hypertrophy and histone deacetylase-dependent transcriptional repression mediated by the atypical homeodomain protein Hop

Sequence-specific recruitment of transcriptional co-repressor Cabin1 by myocyte enhancer factor-2

Myosin heavy chain isoform expression in the failing and nonfailing human heart

MEF2: a calcium-dependent regulator of cell division, differentiation and death

Class II histone deacetylases act as signal-responsive repressors of cardiac hypertrophy

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