Growth inhibition and DNA damage induced by Cre recombinase in mammalian cells - Wikidata - wikimedia - TriplyDB

Growth inhibition and DNA damage induced by Cre recombinase in mammalian cells

Growth inhibition and DNA damage induced by Cre recombinase in mammalian cells

http://www.wikidata.org/entity/Q33930407

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ATR suppresses endogenous DNA damage and allows completion of homologous recombination repair Stable and efficient cassette exchange under non-selectable conditions by combined use of two site-specific recombinases.Taspase1 functions as a non-oncogene addiction protease that coordinates cancer cell proliferation and apoptosis Efficient delivery of Cre-recombinase to neurons in vivo and stable transduction of neurons using adeno-associated and lentiviral vectors Enhanced cell-permeant Cre protein for site-specific recombination in cultured cells Rearranging the centromere of the human Y chromosome with phiC31 integrase From Gene Targeting to Genome Editing: Transgenic animals applications and beyond Cilia/Ift protein and motor -related bone diseases and mouse models Expanding the scope of site-specific recombinases for genetic and metabolic engineering Strategies to achieve conditional gene mutation in mice Cre-mediated stress affects sirtuin expression levels, peroxisome biogenesis and metabolism, antioxidant and proinflammatory signaling pathways Mutants of Cre recombinase with improved accuracy Genetically manipulated mouse models of lung disease: potential and pitfalls Aurora-A kinase is essential for bipolar spindle formation and early development The primitive endoderm lineage of the mouse blastocyst: sequential transcription factor activation and regulation of differentiation by Sox17.Reversible suppression of an essential gene in adult mice using transgenic RNA interference Specificity protein 2 (Sp2) is essential for mouse development and autonomous proliferation of mouse embryonic fibroblasts Palb2 synergizes with Trp53 to suppress mammary tumor formation in a model of inherited breast cancer Conditional, genetic disruption of ciliary neurotrophic factor receptors reveals a role in adult motor neuron survival p63 deficiency activates a program of cellular senescence and leads to accelerated aging Functional characterization of the human mariner transposon Hsmar2 Development of a chromosomally integrated metabolite-inducible Leu3p-alpha-IPM "off-on" gene switch Neural crest origins of the neck and shoulder p53 pathway is involved in cell competition during mouse embryogenesis Gfi1Cre mice have early onset progressive hearing loss and induce recombination in numerous inner ear non-hair cells.PPARgamma deficiency results in reduced lung elastic recoil and abnormalities in airspace distribution.Transient expression of the Arf tumor suppressor during male germ cell and eye development in Arf-Cre reporter mice Disruption of Foxg1 expression by knock-in of cre recombinase: effects on the development of the mouse telencephalon.Notch1 loss of heterozygosity causes vascular tumors and lethal hemorrhage in mice.Highly efficient cell-type-specific gene inactivation reveals a key function for the Drosophila FUS homolog cabeza in neurons A pRb-independent mechanism preserves the postmitotic state in terminally differentiated skeletal muscle cells.A Cre-lox approach for transient transgene expression in neural precursor cells and long-term tracking of their progeny in vitro and in vivo.Characterization of parameters required for effective use of tamoxifen-regulated recombination.Efficient CPP-mediated Cre protein delivery to developing and adult CNS tissues Transcriptome analysis reveals absence of unintended effects in drought-tolerant transgenic plants overexpressing the transcription factor ABF3 Cre recombination-mediated cassette exchange for building versatile transgenic human embryonic stem cells lines.A p53-dependent mechanism underlies macrocytic anemia in a mouse model of human 5q- syndrome.Age- and gene-dosage-dependent cre-induced abnormalities in the retinal pigment epithelium Prevention of senescence progression in reversibly immortalized human ensheathing glia permits their survival after deimmortalization.Integrated genetic approaches identify the molecular mechanisms of Sox4 in early B-cell development: intricate roles for RAG1/2 and CK1ε.

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Growth inhibition and DNA damage induced by Cre recombinase in mammalian cells

http://www.wikidata.org/entity/Q33930407

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2001 nî lūn-bûn

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2001 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2001 թվականի հուլիսին հրատարակված գիտական հոդված

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2001年の論文

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2001年論文

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2001年論文

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2001年論文

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2001年論文

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2001年論文

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2001年论文

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name

Growth inhibition and DNA damage induced by Cre recombinase in mammalian cells

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Growth inhibition and DNA damage induced by Cre recombinase in mammalian cells

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Growth inhibition and DNA damage induced by Cre recombinase in mammalian cells

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Growth inhibition and DNA damage induced by Cre recombinase in mammalian cells

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Growth inhibition and DNA damage induced by Cre recombinase in mammalian cells

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Growth inhibition and DNA damage induced by Cre recombinase in mammalian cells

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Proceedings of the National Academy of Sciences of the United States of America

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Growth inhibition and DNA damage induced by Cre recombinase in mammalian cells

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Allak BA

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Berns A

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Beverloo HB

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Jonkers J

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Kanaar R

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Loonstra A

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Vooijs M

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van Drunen E

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P2860

Ligand-activated site-specific recombination in mice

Site-specific DNA recombination in mammalian cells by the Cre recombinase of bacteriophage P1

Structure of Cre recombinase complexed with DNA in a site-specific recombination synapse

Generalized lacZ expression with the ROSA26 Cre reporter strain

Analysis of C-MYC function in normal cells via conditional gene-targeted mutation

Bacteriophage P1 site-specific recombination. I. Recombination between loxP sites

Bacteriophage P1 site-specific recombination. Purification and properties of the Cre recombinase protein

Inducible gene targeting in mice using the Cre/lox system

Simplified mammalian DNA isolation procedure

Engineering the mouse genome by site-specific recombination.

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10.1073/PNAS.161269798

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