Role of amygdalo-nigral circuitry in conditioning of a visual stimulus paired with food. - Wikidata - wikimedia - TriplyDB

Role of amygdalo-nigral circuitry in conditioning of a visual stimulus paired with food.

Role of amygdalo-nigral circuitry in conditioning of a visual stimulus paired with food.

http://www.wikidata.org/entity/Q33947551

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Dopamine in motivational control: rewarding, aversive, and alerting The primate amygdala represents the positive and negative value of visual stimuli during learning.The effect of anticipation and the specificity of sex differences for amygdala and hippocampus function in emotional memory Neural correlates of two different types of extinction learning in the amygdala central nucleus The habenula encodes negative motivational value associated with primary punishment in humans Neural mechanisms of extinction learning and retrieval Dorsolateral striatum is critical for the expression of surprise-induced enhancements in cue associability.An update on the connections of the ventral mesencephalic dopaminergic complex.Dissociation of attention in learning and action: effects of lesions of the amygdala central nucleus, medial prefrontal cortex, and posterior parietal cortex.Appetitive behavioral traits and stimulus intensity influence maintenance of conditioned fear.Inputs to the midbrain dopaminergic complex in the rat, with emphasis on extended amygdala-recipient sectors.Dissociable effects of disconnecting amygdala central nucleus from the ventral tegmental area or substantia nigra on learned orienting and incentive motivation A developmental neurobiological model of motivated behavior: anatomy, connectivity and ontogeny of the triadic nodes.Substantial similarity in amygdala neuronal activity during conditioned appetitive and aversive emotional arousal.Homeostatic regulation of memory systems and adaptive decisions.Control of appetitive and aversive taste-reactivity responses by an auditory conditioned stimulus in a devaluation task: a FOS and behavioral analysis.Multiple timescales of memory in lateral habenula and dopamine neurons.Differential involvement of the central amygdala in appetitive versus aversive learning.Temporally limited role of substantia nigra-central amygdala connections in surprise-induced enhancement of learning.Beyond the Classic VTA: Extended Amygdala Projections to DA-Striatal Paths in the Primate Interactions between amygdala central nucleus and the ventral tegmental area in the acquisition of conditioned cue-directed behavior in rats.Pulvinar projections to the striatum and amygdala in the tree shrew.Basal ganglia orient eyes to reward.Neural mechanisms of acquired phasic dopamine responses in learning.The nucleus accumbens and Pavlovian reward learning.Amygdalo-striatal interaction in the enhancement of stimulus salience in associative learning.The central amygdala projection to the substantia nigra reflects prediction error information in appetitive conditioning.Role of lateral hypothalamus in two aspects of attention in associative learning.Role of amygdala central nucleus in aversive learning produced by shock or by unexpected omission of food.Differential recruitment of distinct amygdalar nuclei across appetitive associative learning Functional internal complexity of amygdala: focus on gene activity mapping after behavioral training and drugs of abuse.Effects of ventral striatal lesions on first- and second-order appetitive conditioning.Cognitive versus stimulus-response theories of learning Updating appetitive memory during reconsolidation window: critical role of cue-directed behavior and amygdala central nucleus.Toward a neurobiology of delusions.Regulator of G protein signaling 14 (RGS14) is expressed pre- and postsynaptically in neurons of hippocampus, basal ganglia, and amygdala of monkey and human brain.Putting desire on a budget: dopamine and energy expenditure, reconciling reward and resources.Role of amygdala central nucleus in the potentiation of consuming and instrumental lever-pressing for sucrose by cues for the presentation or interruption of sucrose delivery in rats.Effects of lesions of the amygdala central nucleus on autoshaped lever pressing.Effects of reward timing information on cue associability are mediated by amygdala central nucleus.

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P2860

Role of amygdalo-nigral circuitry in conditioning of a visual stimulus paired with food.

http://www.wikidata.org/entity/Q33947551

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2005 թվականի ապրիլին հրատարակված գիտական հոդված

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2005年の論文

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2005年论文

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Role of amygdalo-nigral circuitry in conditioning of a visual stimulus paired with food.

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Role of amygdalo-nigral circuitry in conditioning of a visual stimulus paired with food.

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Role of amygdalo-nigral circuitry in conditioning of a visual stimulus paired with food.

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Role of amygdalo-nigral circuitry in conditioning of a visual stimulus paired with food.

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Role of amygdalo-nigral circuitry in conditioning of a visual stimulus paired with food.

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Role of amygdalo-nigral circuitry in conditioning of a visual stimulus paired with food.

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JNEUROSCI.0416-05.2005

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Journal of Neuroscience

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Role of amygdalo-nigral circuitry in conditioning of a visual stimulus paired with food.

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P2093

Frank Groshek

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Gorica D Petrovich

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Hongjoo J Lee

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Joseph P Cantalini

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Michela Gallagher

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Peter C Holland

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P2860

Emotion circuits in the brain

Damage to the lateral and central, but not other, amygdaloid nuclei prevents the acquisition of auditory fear conditioning.

Amygdala circuitry in attentional and representational processes.

Expression of c-fos protein in brain: metabolic mapping at the cellular level.

Efferent connections of the substantia nigra and ventral tegmental area in the rat.

New vistas on amygdala networks in conditioned fear.

Ventral tegmental (A10) system: neurobiology. 1. Anatomy and connectivity.

Removal of cholinergic input to rat posterior parietal cortex disrupts incremental processing of conditioned stimuli.

The amygdala central nucleus and appetitive Pavlovian conditioning: lesions impair one class of conditioned behavior.

Conditioned stimulus as a determinant of the form of the Pavlovian conditioned response.

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10.1523/JNEUROSCI.0416-05.2005

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