H2AX is required for chromatin remodeling and inactivation of sex chromosomes in male mouse meiosis. - Wikidata - wikimedia - TriplyDB

H2AX is required for chromatin remodeling and inactivation of sex chromosomes in male mouse meiosis.

H2AX is required for chromatin remodeling and inactivation of sex chromosomes in male mouse meiosis.

http://www.wikidata.org/entity/Q33965393

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A high incidence of meiotic silencing of unsynapsed chromatin is not associated with substantial pachytene loss in heterozygous male mice carrying multiple simple robertsonian translocations Polo-like kinase is required for synaptonemal complex disassembly and phosphorylation in mouse spermatocytes WSTF regulates the H2A.X DNA damage response via a novel tyrosine kinase activity Surveillance of different recombination defects in mouse spermatocytes yields distinct responses despite elimination at an identical developmental stage.An essential role for a mammalian SWI/SNF chromatin-remodeling complex during male meiosis Choreographing the Double Strand Break Response: Ubiquitin and SUMO Control of Nuclear Architecture Progress in epigenetic histone modification analysis by mass spectrometry for clinical investigations Double-strand break repair on sex chromosomes: challenges during male meiotic prophase Molecular turnover, the H3.3 dilemma and organismal aging (hypothesis)Sperm nuclear proteome and its epigenetic potential Every amino acid matters: essential contributions of histone variants to mammalian development and disease Phylogenomics of unusual histone H2A Variants in Bdelloid rotifers Meiotic pairing and segregation of achiasmate sex chromosomes in eutherian mammals: the role of SYCP3 protein Dicer1 depletion in male germ cells leads to infertility due to cumulative meiotic and spermiogenic defects Male meiosis in Crustacea: synapsis, recombination, epigenetics and fertility in Daphnia magna.SPO11-independent DNA repair foci and their role in meiotic silencing TAF4b is required for mouse spermatogonial stem cell development.Mammalian BTBD12 (SLX4) protects against genomic instability during mammalian spermatogenesis Hormad1 mutation disrupts synaptonemal complex formation, recombination, and chromosome segregation in mammalian meiosis Genome-wide mapping of DNA strand breaks AGO4 regulates entry into meiosis and influences silencing of sex chromosomes in the male mouse germline Localization and regulation of murine Esco2 during male and female meiosis Polycomb protein SCML2 associates with USP7 and counteracts histone H2A ubiquitination in the XY chromatin during male meiosis Mouse MAELSTROM: the link between meiotic silencing of unsynapsed chromatin and microRNA pathway?A mammal-specific Doublesex homolog associates with male sex chromatin and is required for male meiosis Silencing of unpaired chromatin and histone H2A ubiquitination in mammalian meiosis Rapid phosphorylation of histone H2A.X following ionotropic glutamate receptor activation Meiotic DNA double-strand breaks and chromosome asynapsis in mice are monitored by distinct HORMAD2-independent and -dependent mechanisms Antagonistic roles of ubiquitin ligase HEI10 and SUMO ligase RNF212 regulate meiotic recombination RNF8-dependent histone modifications regulate nucleosome removal during spermatogenesis HORMAD2 is essential for synapsis surveillance during meiotic prophase via the recruitment of ATR activity HORMAD1-dependent checkpoint/surveillance mechanism eliminates asynaptic oocytes GammaH2AX and cancer Histone variants and epigenetics.Mass spectrometry analysis of Arabidopsis histone H3 reveals distinct combinations of post-translational modifications.DNA double-strand breaks, recombination and synapsis: the timing of meiosis differs in grasshoppers and flies.Changes in chromatin structure and mobility in living cells at sites of DNA double-strand breaks Physiologic brain activity causes DNA double-strand breaks in neurons, with exacerbation by amyloid-β Histone H2AX is phosphorylated at sites of retroviral DNA integration but is dispensable for postintegration repair.Expressed sequence tag analysis of Lilium longiflorum generative cells.

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P2860

H2AX is required for chromatin remodeling and inactivation of sex chromosomes in male mouse meiosis.

http://www.wikidata.org/entity/Q33965393

description

2003 nî lūn-bûn

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2003 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2003 թվականի ապրիլին հրատարակված գիտական հոդված

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年论文

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name

H2AX is required for chromatin ...... mosomes in male mouse meiosis.

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H2AX is required for chromatin ...... mosomes in male mouse meiosis.

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type

label

H2AX is required for chromatin ...... mosomes in male mouse meiosis.

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H2AX is required for chromatin ...... mosomes in male mouse meiosis.

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prefLabel

H2AX is required for chromatin ...... mosomes in male mouse meiosis.

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H2AX is required for chromatin ...... mosomes in male mouse meiosis.

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H2AX is required for chromatin ...... mosomes in male mouse meiosis.

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P2093

André Nussenzweig

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Arkady Celeste

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Katia Manova

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Oscar Fernandez-Capetillo

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Paul Burgoyne

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Peter J Romanienko

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R Daniel Camerini-Otero

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Shantha K Mahadevaiah

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William M Bonner

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497-508

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10.1016/S1534-5807(03)00093-5

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4

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