Central amylin acts as an adiposity signal to control body weight and energy expenditure - Wikidata - wikimedia - TriplyDB

Central amylin acts as an adiposity signal to control body weight and energy expenditure

Central amylin acts as an adiposity signal to control body weight and energy expenditure

http://www.wikidata.org/entity/Q33976258

about

The brain and brown fat Amylin at the interface between metabolic and neurodegenerative disorders Gut hormones such as amylin and GLP-1 in the control of eating and energy expenditure.Associations of amylin with inflammatory markers and metabolic syndrome in apparently healthy Chinese.Neuronal receptor activity-modifying protein 1 promotes energy expenditure in mice.Ovariectomy and overeating palatable, energy-dense food increase subcutaneous adipose tissue more than intra-abdominal adipose tissue in rats.Ablation of the transcriptional regulator Id1 enhances energy expenditure, increases insulin sensitivity, and protects against age and diet induced insulin resistance, and hepatosteatosis.Genetic Similarities between Compulsive Overeating and Addiction Phenotypes: A Case for "Food Addiction"?Pharmacological characterization of rat amylin receptors: implications for the identification of amylin receptor subtypes.Islet Amyloid Polypeptide: Structure, Function, and Pathophysiology.Human islet amyloid polypeptide at the air-aqueous interface: a Langmuir monolayer approach.Endogenous VMH amylin signaling is required for full leptin signaling and protection from diet-induced obesity.Acute peripheral GLP-1 receptor agonism or antagonism does not alter energy expenditure in rats after Roux-en-Y gastric bypass Islet amyloid: from fundamental biophysics to mechanisms of cytotoxicity.The interaction of amylin with other hormones in the control of eating.Role of Amylin in Type 1 and Type 2 Diabetes.Amylin-mediated control of glycemia, energy balance, and cognition.A novel dual amylin and calcitonin receptor agonist, KBP-089, induces weight loss through a reduction in fat, but not lean mass, while improving food preference.The dual amylin- and calcitonin-receptor agonist KBP-042 increases insulin sensitivity and induces weight loss in rats with obesity.KBP-088, a novel DACRA with prolonged receptor activation, is superior to davalintide in terms of efficacy on body weight.The Receptor for Advanced Glycation Endproducts is a mediator of toxicity by IAPP and other proteotoxic aggregates: Establishing and Exploiting Common Ground for Novel Amyloidosis Therapies.

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Central amylin acts as an adiposity signal to control body weight and energy expenditure

http://www.wikidata.org/entity/Q33976258

description

2010 nî lūn-bûn

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2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2010 թվականի ապրիլին հրատարակված գիտական հոդված

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2010年の論文

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年學術文章

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name

Central amylin acts as an adiposity signal to control body weight and energy expenditure

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Central amylin acts as an adiposity signal to control body weight and energy expenditure

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Central amylin acts as an adiposity signal to control body weight and energy expenditure

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Central amylin acts as an adiposity signal to control body weight and energy expenditure

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Central amylin acts as an adiposity signal to control body weight and energy expenditure

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Central amylin acts as an adiposity signal to control body weight and energy expenditure

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P356

J.PHYSBEH.2010.04.012

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Physiology and Behavior

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Central amylin acts as an adiposity signal to control body weight and energy expenditure

@en

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Christian Löwenstein

http://www.w3.org/2001/XMLSchema#string

Manuela Munz

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Peter Y Wielinga

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Sabine Muff

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Stephen C Woods

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Leptin responsiveness restored by amylin agonism in diet-induced obesity: evidence from nonclinical and clinical studies

Amylin receptor blockade stimulates food intake in rats

UCP1: the only protein able to mediate adaptive non-shivering thermogenesis and metabolic inefficiency.

Adiposity signals and the control of energy homeostasis.

Effects of meal ingestion on plasma amylin concentration in NIDDM and nondiabetic humans.

New methods for calculating metabolic rate with special reference to protein metabolism.

Central amylin signaling and the regulation of energy homeostasis.

Endocrine controls of eating: CCK, leptin, and ghrelin.

Biological importance of the peptides of the calcitonin family as revealed by disruption and transfer of corresponding genes.

Pancreatic amylin as a centrally acting satiating hormone.

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45-52

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10.1016/J.PHYSBEH.2010.04.012

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1

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20416330

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2900396

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