Differential activation of stress-response signaling in load-induced cardiac hypertrophy and failure. - Wikidata - wikimedia - TriplyDB

Differential activation of stress-response signaling in load-induced cardiac hypertrophy and failure.

Differential activation of stress-response signaling in load-induced cardiac hypertrophy and failure.

http://www.wikidata.org/entity/Q33986250

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Phenotyping of left and right ventricular function in mouse models of compensated hypertrophy and heart failure with cardiac MRI Hic-5 is required for fetal gene expression and cytoskeletal organization of neonatal cardiac myocytes CaMKII inhibition reduces isoproterenol-induced ischemia and arrhythmias in hypertrophic mice.The experimental model of transition from compensated cardiac hypertrophy to failure created by transverse aortic constriction in mice.The CCAAT/enhancer binding protein beta (C/EBPbeta) cooperates with NFAT to control expression of the calcineurin regulatory protein RCAN1-4.Remodeling of early-phase repolarization: a mechanism of abnormal impulse conduction in heart failure Cardiac pressure overload hypertrophy is differentially regulated by β-adrenergic receptor subtypes.Nitric oxide synthase 3 deficiency limits adverse ventricular remodeling after pressure overload in insulin resistance.Cardiac autophagy is a maladaptive response to hemodynamic stress CaMKII inhibition in heart failure, beneficial, harmful, or both Effects of increased preload on the force-frequency response and contractile kinetics in early stages of cardiac muscle hypertrophy Variable phenotype in murine transverse aortic constriction.Targets, trafficking, and timing of cardiac autophagy Autophagy is an adaptive response in desmin-related cardiomyopathy.Ca2+/calmodulin-dependent protein kinase II-dependent remodeling of Ca2+ current in pressure overload heart failure.Diminished cardiac fibrosis in heart failure is associated with altered ventricular arrhythmia phenotype.Intracellular protein aggregation is a proximal trigger of cardiomyocyte autophagy.Substrain specific response to cardiac pressure overload in C57BL/6 mice.Autophagy in load-induced heart disease.Ventricular remodeling and function: insights using murine echocardiography.HACE1-dependent protein degradation provides cardiac protection in response to haemodynamic stress.Calcineurin signaling in the heart: The importance of time and place.Sustained hemodynamic stress disrupts normal circadian rhythms in calcineurin-dependent signaling and protein phosphorylation in the heart.Foxo transcription factors blunt cardiac hypertrophy by inhibiting calcineurin signaling Chronic doxycycline exposure accelerates left ventricular hypertrophy and progression to heart failure in mice after thoracic aorta constriction.Temporal dynamics of cardiac hypertrophic growth in response to pressure overload.Ultrasound biomicroscopy validation of a murine model of cardiac hypertrophic preconditioning: comparison with a hemodynamic assessment.Pathophysiological Mapping of Experimental Heart Failure: Left and Right Ventricular Remodeling in Transverse Aortic Constriction Is Temporally, Kinetically and Structurally Distinct.Dual inhibition of sodium–glucose linked cotransporters 1 and 2 exacerbates cardiac dysfunction following experimental myocardial infarction.Endogenous inhibitors of hypertrophy in concentric versus eccentric hypertrophy

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P2860

Differential activation of stress-response signaling in load-induced cardiac hypertrophy and failure.

http://www.wikidata.org/entity/Q33986250

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2005 nî lūn-bûn

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2005 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2005 թվականի հուլիսին հրատարակված գիտական հոդված

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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name

Differential activation of str ...... rdiac hypertrophy and failure.

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Differential activation of str ...... rdiac hypertrophy and failure.

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Differential activation of str ...... rdiac hypertrophy and failure.

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Differential activation of str ...... rdiac hypertrophy and failure.

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Differential activation of str ...... rdiac hypertrophy and failure.

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Differential activation of str ...... rdiac hypertrophy and failure.

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PHYSIOLGENOMICS.00061.2005

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Physiological Genomics

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Differential activation of str ...... rdiac hypertrophy and failure.

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Asim Dey

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Barry Cabuay

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Beverly A Rothermel

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Bridgid Nolan

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Elaine Demetroulis

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Joseph A Hill

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Kambeez Berenji

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Ki-Dong Yoo

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Michael Gimbel

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Mohsen Karimi

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P2860

Activated MEK5 induces serial assembly of sarcomeres and eccentric cardiac hypertrophy.

The phosphoinositide 3-kinase pathway

A protein encoded within the Down syndrome critical region is enriched in striated muscles and inhibits calcineurin signaling

Synthesis and function of 3-phosphorylated inositol lipids

Hypertension, cardiac hypertrophy, and sudden death in mice lacking natriuretic peptide receptor A

A small molecular activator of cardiac hypertrophy uncovered in a chemical screen for modifiers of the calcineurin signaling pathway

PKC-alpha regulates cardiac contractility and propensity toward heart failure

Cardiac hypertrophy: the good, the bad, and the ugly

Calcineurin/NFAT coupling participates in pathological, but not physiological, cardiac hypertrophy.

Left ventricular hypertrophy: pathogenesis, detection, and prognosis.

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18-27

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10.1152/PHYSIOLGENOMICS.00061.2005

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1

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23

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7711137

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16033866

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4118287

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