Autonomous CaMKII can promote either long-term potentiation or long-term depression, depending on the state of T305/T306 phosphorylation - Wikidata - wikimedia - TriplyDB

Autonomous CaMKII can promote either long-term potentiation or long-term depression, depending on the state of T305/T306 phosphorylation

Autonomous CaMKII can promote either long-term potentiation or long-term depression, depending on the state of T305/T306 phosphorylation

http://www.wikidata.org/entity/Q33991390

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Calcium input frequency, duration and amplitude differentially modulate the relative activation of calcineurin and CaMKII The NMDA receptor complex: a multifunctional machine at the glutamatergic synapse Mechanisms of CaMKII action in long-term potentiation CASK and CaMKII function in Drosophila memory In search of a recognition memory engram Improving a natural CaMKII inhibitor by random and rational design Excitotoxic insult results in a long-lasting activation of CaMKIIα and mitochondrial damage in living hippocampal neurons Fast Decay of CaMKII FRET Sensor Signal in Spines after LTP Induction Is Not Due to Its Dephosphorylation.Autonomous CaMKII requires further stimulation by Ca2+/calmodulin for enhancing synaptic strength CaMKII control of spine size and synaptic strength: role of phosphorylation states and nonenzymatic action.Temporal profiling of gene networks associated with the late phase of long-term potentiation in vivo.Genetic inhibition of CaMKII in dorsal striatal medium spiny neurons reduces functional excitatory synapses and enhances intrinsic excitability.Single-cell juxtacellular transfection and recording technique.SynGAP regulates protein synthesis and homeostatic synaptic plasticity in developing cortical networks.Role of the CaMKII/NMDA receptor complex in the maintenance of synaptic strength Temporal and region-specific requirements of αCaMKII in spatial and contextual learning.MAGUKs, synaptic development, and synaptic plasticity Catalytically Dead αCaMKII K42M Mutant Acts as a Dominant Negative in the Control of Synaptic Strength.Activity-dependent regulation of synaptic strength by PSD-95 in CA1 neurons Loss of α1,6-Fucosyltransferase Decreases Hippocampal Long Term Potentiation: IMPLICATIONS FOR CORE FUCOSYLATION IN THE REGULATION OF AMPA RECEPTOR HETEROMERIZATION AND CELLULAR SIGNALING.Chronic intermittent ethanol and withdrawal differentially modulate basolateral amygdala AMPA-type glutamate receptor function and trafficking Substrate-selective and calcium-independent activation of CaMKII by α-actinin.Wip1 phosphatase modulates both long-term potentiation and long-term depression through the dephosphorylation of CaMKII CaMKII regulation in information processing and storage.Kalirin and Trio proteins serve critical roles in excitatory synaptic transmission and LTP.Metabolic regulation of CaMKII protein and caspases in Xenopus laevis egg extracts.CaMKII-dependent phosphorylation of the GTPase Rem2 is required to restrict dendritic complexity.Phosphoproteomic Analysis Reveals a Novel Mechanism of CaMKIIα Regulation Inversely Induced by Cocaine Memory Extinction versus Reconsolidation Multiple CaMKII Binding Modes to the Actin Cytoskeleton Revealed by Single-Molecule Imaging.The interplay between neuronal activity and actin dynamics mimic the setting of an LTD synaptic tag CaMKII: claiming center stage in postsynaptic function and organization.Synaptic Plasticity and Memory: New Insights from Hippocampal Left-Right Asymmetries.Diverse impact of neuronal activity at θ frequency on hippocampal long-term plasticity.CaMKII Autophosphorylation Is Necessary for Optimal Integration of Ca2+ Signals during LTP Induction, but Not Maintenance.Memory Takes Time.Quantitative estimates of the cytoplasmic, PSD, and NMDAR-bound pools of CaMKII in dendritic spines.Proteomic Analysis of Postsynaptic Protein Complexes Underlying Neuronal Plasticity.Emerging Role for Nucleus Accumbens Medium Spiny Neuron Subtypes in Depression.Measuring CaMKII concentration in dendritic spines.Early-life stress affects the structural and functional plasticity of the medial prefrontal cortex in adolescent rats.

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Autonomous CaMKII can promote either long-term potentiation or long-term depression, depending on the state of T305/T306 phosphorylation

http://www.wikidata.org/entity/Q33991390

description

2010 nî lūn-bûn

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2010 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հունիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Autonomous CaMKII can promote ...... e of T305/T306 phosphorylation

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Autonomous CaMKII can promote ...... e of T305/T306 phosphorylation

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Autonomous CaMKII can promote ...... e of T305/T306 phosphorylation

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JNEUROSCI.0133-10.2010

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Journal of Neuroscience

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Autonomous CaMKII can promote ...... e of T305/T306 phosphorylation

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P2093

David Lemelin

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Hyun Jae Pi

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John Lisman

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Nikolai Otmakhov

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Paul De Koninck

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P2860

Homosynaptic long-term depression in area CA1 of hippocampus and effects of N-methyl-D-aspartate receptor blockade

CaMKII tethers to L-type Ca2+ channels, establishing a local and dedicated integrator of Ca2+ signals for facilitation

The molecular basis of CaMKII function in synaptic and behavioural memory

Autophosphorylation at Thr286 of the alpha calcium-calmodulin kinase II in LTP and learning

CaMKII regulates the frequency-response function of hippocampal synapses for the production of both LTD and LTP

Dynamic control of CaMKII translocation and localization in hippocampal neurons by NMDA receptor stimulation

Inhibition of postsynaptic PKC or CaMKII blocks induction but not expression of LTP

Postnatal synaptic potentiation: delivery of GluR4-containing AMPA receptors by spontaneous activity

A simple method for organotypic cultures of nervous tissue

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8704-8709

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10.1523/JNEUROSCI.0133-10.2010

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26

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44888950

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20592192

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phosphorylation

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2903435

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