Glia are critical for the neuropathology of complex I deficiency in Drosophila - Wikidata - wikimedia - TriplyDB

Glia are critical for the neuropathology of complex I deficiency in Drosophila

Glia are critical for the neuropathology of complex I deficiency in Drosophila

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Leigh syndrome: neuropathology and pathogenesis.Drosophila as a model to study the role of glia in neurodegeneration.Nerve-racking - Apoptotic and non-apoptotic roles of caspases in the nervous system of Drosophila.Defective Phagocytic Corpse Processing Results in Neurodegeneration and Can Be Rescued by TORC1 Activation A Conserved Cytoskeletal Signaling Cascade Mediates Neurotoxicity of FTDP-17 Tau Mutations In Vivo.Glial lipid droplets and neurodegeneration in a Drosophila model of complex I deficiency.Mito-Nuclear Interactions Affecting Lifespan and Neurodegeneration in a Drosophila Model of Leigh Syndrome.

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Glia are critical for the neuropathology of complex I deficiency in Drosophila

http://www.wikidata.org/entity/Q33991794

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2014 nî lūn-bûn

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2014 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2014 թվականի ապրիլին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Glia are critical for the neuropathology of complex I deficiency in Drosophila

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Glia are critical for the neuropathology of complex I deficiency in Drosophila

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type

label

Glia are critical for the neuropathology of complex I deficiency in Drosophila

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Glia are critical for the neuropathology of complex I deficiency in Drosophila

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prefLabel

Glia are critical for the neuropathology of complex I deficiency in Drosophila

@ast

Glia are critical for the neuropathology of complex I deficiency in Drosophila

@en

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Human Molecular Genetics

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Glia are critical for the neuropathology of complex I deficiency in Drosophila

@en

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Mel B Feany

http://www.w3.org/2001/XMLSchema#string

Rutger Vogel

http://www.w3.org/2001/XMLSchema#string

Vijay R Hegde

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P2860

Equal numbers of neuronal and nonneuronal cells make the human brain an isometrically scaled-up primate brain

Leigh syndrome associated with mitochondrial complex I deficiency due to a novel mutation in the NDUFS1 gene

Large-scale deletion and point mutations of the nuclear NDUFV1 and NDUFS1 genes in mitochondrial complex I deficiency

Glial cells as intrinsic components of non-cell-autonomous neurodegenerative disease

Targeted gene expression as a means of altering cell fates and generating dominant phenotypes

Isolated complex I deficiency in children: clinical, biochemical and genetic aspects

Mitochondrial diseases in man and mouse

Complex I deficiency due to loss of Ndufs4 in the brain results in progressive encephalopathy resembling Leigh syndrome.

Neuron-glia metabolic coupling and plasticity.

Energy converting NADH:quinone oxidoreductase (complex I).

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4686-4692

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scholarly article

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10.1093/HMG/DDU188

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17

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23

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2014-04-23T00:00:00Z

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261840355

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24760769

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4119418

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