PRR1, a homolog of Aspergillus nidulans palF, controls pH-dependent gene expression and filamentation in Candida albicans.
about
Regulation of gene expression by ambient pH in filamentous fungi and yeastspH signaling in human fungal pathogens: a new target for antifungal strategiesThe Peptide-binding Cavity Is Essential for Als3-mediated Adhesion of Candida albicans to Human CellsThe transcription factor Rim101p governs ion tolerance and cell differentiation by direct repression of the regulatory genes NRG1 and SMP1 in Saccharomyces cerevisiae.Mds3 regulates morphogenesis in Candida albicans through the TOR pathwayRole of Ess1 in growth, morphogenetic switching, and RNA polymerase II transcription in Candida albicansRefining the pH response in Aspergillus nidulans: a modulatory triad involving PacX, a novel zinc binuclear cluster proteinGenetic control of extracellular protease synthesis in the yeast Yarrowia lipolytica.ABG1, a novel and essential Candida albicans gene encoding a vacuolar protein involved in cytokinesis and hyphal branching.Candida albicans Als3p is required for wild-type biofilm formation on silicone elastomer surfacesCandida albicans RIM101 pH response pathway is required for host-pathogen interactions.Mutational analysis of Candida albicans SNF7 reveals genetically separable Rim101 and ESCRT functions and demonstrates divergence in bro1-domain protein interactions.Dominant active alleles of RIM101 (PRR2) bypass the pH restriction on filamentation of Candida albicans.Effect of environmental pH on morphological development of Candida albicans is mediated via the PacC-related transcription factor encoded by PRR2.The role of Candida albicans NOT5 in virulence depends upon diverse host factors in vivo.The RIM101 signal transduction pathway regulates Candida albicans virulence during experimental keratomycosis.Transcription profiling of Candida albicans cells undergoing the yeast-to-hyphal transition.Regulatory circuitry governing fungal development, drug resistance, and disease.Deletions of endocytic components VPS28 and VPS32 affect growth at alkaline pH and virulence through both RIM101-dependent and RIM101-independent pathways in Candida albicans.Conserved serine/threonine kinase encoded by CBK1 regulates expression of several hypha-associated transcripts and genes encoding cell wall proteins in Candida albicans.A proposed mechanism for the interaction between the Candida albicans Als3 adhesin and streptococcal cell wall proteinsRegulation of innate immune response to Candida albicans infections by αMβ2-Pra1p interaction.Heterogeneous distribution of Candida albicans cell-surface antigens demonstrated with an Als1-specific monoclonal antibodyAdaptation to pH and role of PacC in the rice blast fungus Magnaporthe oryzaeEvidence for novel pH-dependent regulation of Candida albicans Rim101, a direct transcriptional repressor of the cell wall beta-glycosidase Phr2.Roles of Candida albicans Dfg5p and Dcw1p cell surface proteins in growth and hypha formation.A monoclonal antibody specific for Candida albicans Als4 demonstrates overlapping localization of Als family proteins on the fungal cell surface and highlights differences between Als localization in vitro and in vivo.Environmental sensing and signal transduction pathways regulating morphopathogenic determinants of Candida albicans.The β-arrestin-like protein Rim8 is hyperphosphorylated and complexes with Rim21 and Rim101 to promote adaptation to neutral-alkaline pH.Evaluation of the roles of four Candida albicans genes in virulence by using gene disruption strains that express URA3 from the native locus.Molecular Components of the Neurospora crassa pH Signaling Pathway and Their Regulation by pH and the PAC-3 Transcription FactorIntegrin αXβ₂ is a leukocyte receptor for Candida albicans and is essential for protection against fungal infections.Reduced TOR signaling sustains hyphal development in Candida albicans by lowering Hog1 basal activity.A competitive infection model of hematogenously disseminated candidiasis in mice redefines the role of Candida albicans IRS4 in pathogenesisAnalysis of the Candida albicans Als2p and Als4p adhesins suggests the potential for compensatory function within the Als family.GLN3 encodes a global regulator of nitrogen metabolism and virulence of C. albicans.Monoclonal antibodies specific for Candida albicans Als3 that immunolabel fungal cells in vitro and in vivo and block adhesion to host surfacesDeletion of ALS5, ALS6 or ALS7 increases adhesion of Candida albicans to human vascular endothelial and buccal epithelial cellsRecognition of Candida albicans Als3 by the germ tube-specific monoclonal antibody 3D9.3.Unequal contribution of ALS9 alleles to adhesion between Candida albicans and human vascular endothelial cells.
P2860
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P2860
PRR1, a homolog of Aspergillus nidulans palF, controls pH-dependent gene expression and filamentation in Candida albicans.
description
1999 nî lūn-bûn
@nan
1999 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
PRR1, a homolog of Aspergillus ...... mentation in Candida albicans.
@ast
PRR1, a homolog of Aspergillus ...... mentation in Candida albicans.
@en
type
label
PRR1, a homolog of Aspergillus ...... mentation in Candida albicans.
@ast
PRR1, a homolog of Aspergillus ...... mentation in Candida albicans.
@en
prefLabel
PRR1, a homolog of Aspergillus ...... mentation in Candida albicans.
@ast
PRR1, a homolog of Aspergillus ...... mentation in Candida albicans.
@en
P2093
P2860
P1476
PRR1, a homolog of Aspergillus ...... mentation in Candida albicans.
@en
P2093
P2860
P304
P407
P577
1999-12-01T00:00:00Z