Systems modeling of the role of interleukin-21 in the maintenance of effector CD4+ T cell responses during chronic Helicobacter pylori infection.
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Mucosal Interactions between Genetics, Diet, and Microbiome in Inflammatory Bowel DiseaseModeling-Enabled Systems Nutritional ImmunologyGoals in Nutrition Science 2015-2020Interleukin-23A is associated with tumor growth in Helicobacter-pylori-related human gastric cancer.Modeling the Regulatory Mechanisms by Which NLRX1 Modulates Innate Immune Responses to Helicobacter pylori InfectionSensitivity Analysis of an ENteric Immunity SImulator (ENISI)-Based Model of Immune Responses to Helicobacter pylori Infection.Multiscale modeling of mucosal immune responsesIL-17a and IL-22 Induce Expression of Antimicrobials in Gastrointestinal Epithelial Cells and May Contribute to Epithelial Cell Defense against Helicobacter pyloriSystems-wide analyses of mucosal immune responses to Helicobacter pylori at the interface between pathogenicity and symbiosisModeling the Role of Lanthionine Synthetase C-Like 2 (LANCL2) in the Modulation of Immune Responses to Helicobacter pylori Infection.Decreased expression of Calpain-9 predicts unfavorable prognosis in patients with gastric cancer.Characterisation of inflammatory processes in Helicobacter pylori-induced gastric lymphomagenesis in a mouse model.Demystifying the cytokine network: Mathematical models point the way.Citrobacter rodentium: a model enteropathogen for understanding the interplay of innate and adaptive components of type 3 immunity.Cooperation of Gastric Mononuclear Phagocytes with Helicobacter pylori during Colonization.Supervised learning methods in modeling of CD4+ T cell heterogeneity.Alterations in Th17 and the Respective Cytokine Levels in Helicobacter pylori-Induced Stomach Diseases.Modulation of Immune Signaling and Metabolism Highlights Host and Fungal Transcriptional Responses in Mouse Models of Invasive Pulmonary Aspergillosis.Multiscale positive feedbacks contribute to unidirectional gastric disease progression induced by helicobacter pylori infection.Informatics for Nutritional Genetics and Genomics.Simulation of Stimulation: Cytokine Dosage and Cell Cycle Crosstalk Driving Timing-Dependent T Cell DifferentiationA Mechanistic Computational Model Reveals That Plasticity of CD4 T Cell Differentiation Is a Function of Cytokine Composition and Dosage
P2860
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P2860
Systems modeling of the role of interleukin-21 in the maintenance of effector CD4+ T cell responses during chronic Helicobacter pylori infection.
description
2014 nî lūn-bûn
@nan
2014 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Systems modeling of the role o ...... Helicobacter pylori infection.
@ast
Systems modeling of the role o ...... Helicobacter pylori infection.
@en
type
label
Systems modeling of the role o ...... Helicobacter pylori infection.
@ast
Systems modeling of the role o ...... Helicobacter pylori infection.
@en
prefLabel
Systems modeling of the role o ...... Helicobacter pylori infection.
@ast
Systems modeling of the role o ...... Helicobacter pylori infection.
@en
P2093
P2860
P50
P356
P1433
P1476
Systems modeling of the role o ...... Helicobacter pylori infection.
@en
P2093
Alberto Delgado
M Blanca Piazuelo
M Kay Washington
Rupesh Chaturvedi
P2860
P304
P356
10.1128/MBIO.01243-14
P50
P577
2014-07-22T00:00:00Z