Activation of phosphotyrosine phosphatase activity attenuates mitogen-activated protein kinase signaling and inhibits c-FOS and nitric oxide synthase expression in macrophages infected with Leishmania donovani

Activation of phosphotyrosine phosphatase activity attenuates mitogen-activated protein kinase signaling and inhibits c-FOS and nitric oxide synthase expression in macrophages infected with Leishmania donovani

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http://www.wikidata.org/entity/Q34001456

Q34001456

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Proteomic analysis of the secretome of Leishmania donovani A novel Leishmania infantum nuclear phosphoprotein Lepp12 which stimulates IL1-beta synthesis in THP-1 transfectants Mechanisms of cellular invasion by intracellular parasites Impact of Leishmania metalloprotease GP63 on macrophage signaling Interference of antibacterial agents with phagocyte functions: immunomodulation or "immuno-fairy tales"?Bacteria fighting back: how pathogens target and subvert the host innate immune system.Leishmania-induced inactivation of the macrophage transcription factor AP-1 is mediated by the parasite metalloprotease GP63.Subversion mechanisms by which Leishmania parasites can escape the host immune response: a signaling point of view.Leishmania interferes with host cell signaling to devise a survival strategy.The curative effect of fucoidan on visceral leishmaniasis is mediated by activation of MAP kinases through specific protein kinase C isoforms Immune regulation during chronic visceral leishmaniasis.Clinical and experimental advances in treatment of visceral leishmaniasis.Induction of inducible nitric oxide synthase-NO* by lipoarabinomannan of Mycobacterium tuberculosis is mediated by MEK1-ERK, MKK7-JNK, and NF-kappaB signaling pathways.Leishmania donovani amastigotes impair gamma interferon-induced STAT1alpha nuclear translocation by blocking the interaction between STAT1alpha and importin-alpha5 Activation of p38 mitogen-activated protein kinase attenuates Leishmania donovani infection in macrophages.Leishmania donovani suppresses activated protein 1 and NF-kappaB activation in host macrophages via ceramide generation: involvement of extracellular signal-regulated kinase.Immunosuppression in hamsters with progressive visceral leishmaniasis is associated with an impairment of protein kinase C activity in their lymphocytes that can be partially reversed by okadaic acid or anti-transforming growth factor beta antibody Small RNAs derived from tRNAs and rRNAs are highly enriched in exosomes from both old and new world Leishmania providing evidence for conserved exosomal RNA Packaging.Leishmania donovani Utilize Sialic Acids for Binding and Phagocytosis in the Macrophages through Selective Utilization of Siglecs and Impair the Innate Immune Arm.The early interaction of Leishmania with macrophages and dendritic cells and its influence on the host immune response.Recent developments in the interactions between caveolin and pathogens.Mechanisms of immune evasion in leishmaniasis Novel program of macrophage gene expression induced by phagocytosis of Leishmania chagasi.Dual Transcriptome Profiling of Leishmania-Infected Human Macrophages Reveals Distinct Reprogramming Signatures.Eosinophils and mast cells in leishmaniasis.Leishmania major amastigotes induce p50/c-Rel NF-kappa B transcription factor in human macrophages: involvement in cytokine synthesis.SHP-1 as a critical regulator of Mycoplasma pneumoniae-induced inflammation in human asthmatic airway epithelial cells.Complement receptor 3 deficiency influences lesion progression during Leishmania major infection in BALB/c mice Liposomal cholesterol delivery activates the macrophage innate immune arm to facilitate intracellular Leishmania donovani killing.Chlamydia pneumoniae activates epithelial cell proliferation via NF-kappaB and the glucocorticoid receptor.Leishmania exosomes deliver preemptive strikes to create an environment permissive for early infection.Immunopathogenesis of non-healing American cutaneous leishmaniasis and progressive visceral leishmaniasis.Characterization of the Protein Tyrosine Phosphatase LmPRL-1 Secreted by Leishmania major via the Exosome Pathway.Evasion of Host Defence by Leishmania donovani: Subversion of Signaling Pathways.Host cell signalling and leishmania mechanisms of evasion.Comparative study of the ability of Leishmania mexicana promastigotes and amastigotes to alter macrophage signaling and functions.Role of host protein tyrosine phosphatase SHP-1 in Leishmania donovani-induced inhibition of nitric oxide production.Functional Impairment of Murine Dendritic Cell Subsets following Infection with Infective Larval Stage 3 of Brugia malayi The chemotherapeutic drug oxaliplatin differentially affects blood DC function dependent on environmental cues.Sterile-α- and armadillo motif-containing protein inhibits the TRIF-dependent downregulation of signal regulatory protein α to interfere with intracellular bacterial elimination in Burkholderia pseudomallei-infected mouse macrophages.

P2860

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P2860

Activation of phosphotyrosine phosphatase activity attenuates mitogen-activated protein kinase signaling and inhibits c-FOS and nitric oxide synthase expression in macrophages infected with Leishmania donovani

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http://www.wikidata.org/entity/Q34001456

description

1999 nî lūn-bûn

@nan

1999 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

1999 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

1999年の論文

@ja

1999年論文

@yue

1999年論文

@zh-hant

1999年論文

@zh-hk

1999年論文

@zh-mo

1999年論文

@zh-tw

1999年论文

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