Use of genetically manipulated strains of Clostridium perfringens reveals that both alpha-toxin and theta-toxin are required for vascular leukostasis to occur in experimental gas gangrene
about
Synergistic effects of alpha-toxin and perfringolysin O in Clostridium perfringens-mediated gas gangreneMolecular and cellular basis of microvascular perfusion deficits induced by Clostridium perfringens and Clostridium septicumThe Cholesterol-dependent Cytolysin Membrane-binding Interface Discriminates Lipid Environments of Cholesterol to Support β-Barrel Pore Insertion.Membrane assembly of the cholesterol-dependent cytolysin pore complex.NetB, a pore-forming toxin from necrotic enteritis strains of Clostridium perfringens.Pore-forming activity of alpha-toxin is essential for clostridium septicum-mediated myonecrosis.The MACPF/CDC family of pore-forming toxins.Proteome and transcriptome analysis of the virulence genes regulated by the VirR/VirS system in Clostridium perfringensFunctional analysis of the VirSR phosphorelay from Clostridium perfringens.The oncopathic potency of Clostridium perfringens is independent of its alpha-toxin gene.The cysteine protease α-clostripain is not essential for the pathogenesis of Clostridium perfringens-mediated myonecrosis.The VirR response regulator from Clostridium perfringens binds independently to two imperfect direct repeats located upstream of the pfoA promoterRole of catabolite control protein A in the regulation of intermedilysin production by Streptococcus intermedius.The spatial organization of the VirR boxes is critical for VirR-mediated expression of the perfringolysin O gene, pfoA, from Clostridium perfringens.Perfringolysin O expression in Clostridium perfringens is independent of the upstream pfoR gene.Clostridium Perfringens Toxins Involved in Mammalian Veterinary Diseases.The CpAL quorum sensing system regulates production of hemolysins CPA and PFO to build Clostridium perfringens biofilms.Perfringolysin O: The Underrated Clostridium perfringens Toxin?Packing a punch: the mechanism of pore formation by cholesterol dependent cytolysins and membrane attack complex/perforin-like proteins.The NanI and NanJ sialidases of Clostridium perfringens are not essential for virulence.Effects of Clostridium perfringens alpha-toxin (PLC) and perfringolysin O (PFO) on cytotoxicity to macrophages, on escape from the phagosomes of macrophages, and on persistence of C. perfringens in host tissuesAnimal models to study the pathogenesis of human and animal Clostridium perfringens infections.Alpha-toxin of Clostridium perfringens is not an essential virulence factor in necrotic enteritis in chickens.The synergistic necrohemorrhagic action of Clostridium perfringens perfringolysin and alpha toxin in the bovine intestine and against bovine endothelial cells.Cholesterol-dependent cytolysins impair pro-inflammatory macrophage responses.Concurrent Host-Pathogen Transcriptional Responses in a Clostridium perfringens Murine Myonecrosis Infection.
P2860
Q24548619-27D7D669-A67E-4735-BB01-635AFD9F18FCQ27318768-C6FC5F66-867C-4C26-BA9C-61311C6A9BD6Q30152918-0B281D2C-C67F-46DF-AF73-6966D1D351D2Q30155494-DB073162-F064-4145-BD3B-675A2280D5A0Q30156809-9D08D712-AAE2-49C6-B6DF-00F86ABA223EQ30157403-82190F1B-311B-405C-882A-0649598ADF6EQ30157659-223243A2-7E3D-4765-962E-DA3A6290E043Q30834212-F975A42B-BDD3-4917-A895-B8E2EB08E385Q33464950-724F15AC-4258-425D-9F19-E3FBC160960DQ33691013-C60BD844-6377-4C46-90F1-80C57D5AE084Q33987845-E2D97778-DE38-44B9-9860-2B6574AA88C2Q33993392-87445B89-0B0A-424C-8FA9-70C1A246D199Q34119507-05F304E8-8D7D-41AE-B569-80B4D021761FQ34149509-B82B67AF-D266-44D7-A7E6-6C62B6FA383EQ34308193-EACE38C0-BBF6-4F6C-A3B1-12BB7EB35482Q34403323-D5407CAC-13F6-43C1-858A-5911BA1E2CFDQ35609367-18138BFD-EEFC-48A2-846A-C72731B62CCDQ35662926-46B712E5-686E-4271-BEC0-DDA070853046Q36059050-5BC9C3D0-D153-40E8-A492-56347F66C126Q37355868-D2A95B4F-452E-4BAA-9B90-27D10426CEF4Q37521613-FB9BB391-1CB6-4337-8854-D62038444699Q37561946-710DD661-2F5C-4220-92E5-DE6E90073683Q40953202-D8866D0C-C182-438D-8730-282275DA841AQ43076582-3F0908F1-6CB0-4B46-98EC-33393ABF0A77Q54228851-00A13A94-1722-4DA8-98A7-3304314AEE11Q55115047-31135E17-24FA-46FC-8B9C-7EDFFF120314
P2860
Use of genetically manipulated strains of Clostridium perfringens reveals that both alpha-toxin and theta-toxin are required for vascular leukostasis to occur in experimental gas gangrene
description
1999 nî lūn-bûn
@nan
1999 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
Use of genetically manipulated ...... r in experimental gas gangrene
@ast
Use of genetically manipulated ...... r in experimental gas gangrene
@en
Use of genetically manipulated ...... r in experimental gas gangrene
@nl
type
label
Use of genetically manipulated ...... r in experimental gas gangrene
@ast
Use of genetically manipulated ...... r in experimental gas gangrene
@en
Use of genetically manipulated ...... r in experimental gas gangrene
@nl
prefLabel
Use of genetically manipulated ...... r in experimental gas gangrene
@ast
Use of genetically manipulated ...... r in experimental gas gangrene
@en
Use of genetically manipulated ...... r in experimental gas gangrene
@nl
P2093
P2860
P1476
Use of genetically manipulated ...... r in experimental gas gangrene
@en
P2093
P2860
P304
P407
P577
1999-09-01T00:00:00Z