NADPH Oxidase versus Mitochondria-Derived ROS in Glucose-Induced Apoptosis of Pericytes in Early Diabetic Retinopathy. - Wikidata - wikimedia - TriplyDB

NADPH Oxidase versus Mitochondria-Derived ROS in Glucose-Induced Apoptosis of Pericytes in Early Diabetic Retinopathy.

NADPH Oxidase versus Mitochondria-Derived ROS in Glucose-Induced Apoptosis of Pericytes in Early Diabetic Retinopathy.

http://www.wikidata.org/entity/Q34003012

about

Pericytes, an overlooked player in vascular pathobiology Na+/H+-exchanger-1 inhibition counteracts diabetic cataract formation and retinal oxidative-nitrative stress and apoptosis Protective role of morin, a flavonoid, against high glucose induced oxidative stress mediated apoptosis in primary rat hepatocytes.Angiotensin-converting enzyme 2 priming enhances the function of endothelial progenitor cells and their therapeutic efficacy.STAT1-mediated Bim expression promotes the apoptosis of retinal pericytes under high glucose conditions.Ascorbic acid prevents high glucose-induced apoptosis in human brain pericytes.Aldose reductase inhibition suppresses oxidative stress-induced inflammatory disorders.Role of apoptosis-inducing factor, proline dehydrogenase, and NADPH oxidase in apoptosis and oxidative stress Ameliorative Effect of Zinc Oxide Nanoparticles on Antioxidants and Sperm Characteristics in Streptozotocin-Induced Diabetic Rat Testes.Role of N-ε- carboxy methyl lysine, advanced glycation end products and reactive oxygen species for the development of nonproliferative and proliferative retinopathy in type 2 diabetes mellitus.Which NADPH oxidase isoform is relevant for ischemic stroke? The case for nox 2.SENP1-Mediated Desumoylation of DBC1 Inhibits Apoptosis Induced by High Glucose in Bovine Retinal Pericytes Protective Role of Glucagon-Like Peptide-1 Against High-Glucose-Induced Endothelial Oxidative Damage Supplementation with Phycocyanobilin, Citrulline, Taurine, and Supranutritional Doses of Folic Acid and Biotin-Potential for Preventing or Slowing the Progression of Diabetic Complications.Novel morphological features of developing white matter pericytes and rapid scavenging of reactive oxygen species in the neighbouring endothelia.Glucose Homeostasis and Cardiovascular Alterations in Diabetes.Ascorbic acid repletion: A possible therapy for diabetic macular edema?Mechanisms of the antihypertensive effects of Nigella sativa oil in L-NAME-induced hypertensive rats.Advanced glycation end product Nε-carboxymethyllysine induces endothelial cell injury: the involvement of SHP-1-regulated VEGFR-2 dephosphorylation.MicroRNA-125a-5p alleviates the deleterious effects of ox-LDL on multiple functions of human brain microvessel endothelial cells.Destruxins: fungal-derived cyclohexadepsipeptides with multifaceted anticancer and antiangiogenic activities.

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P2860

NADPH Oxidase versus Mitochondria-Derived ROS in Glucose-Induced Apoptosis of Pericytes in Early Diabetic Retinopathy.

http://www.wikidata.org/entity/Q34003012

description

2010 nî lūn-bûn

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2010 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2010 թվականի հունիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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NADPH Oxidase versus Mitochond ...... in Early Diabetic Retinopathy.

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NADPH Oxidase versus Mitochond ...... in Early Diabetic Retinopathy.

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Eva M Kohner

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Joanna M Tarr

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Nik M Mustapha

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Rakesh Chibber

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P2860

Intensive blood-glucose control with sulphonylureas or insulin compared with conventional treatment and risk of complications in patients with type 2 diabetes (UKPDS 33). UK Prospective Diabetes Study (UKPDS) Group

The effect of intensive treatment of diabetes on the development and progression of long-term complications in insulin-dependent diabetes mellitus. The Diabetes Control and Complications Trial Research Group

Homologs of gp91phox: cloning and tissue expression of Nox3, Nox4, and Nox5

Molecular characterization and localization of the NAD(P)H oxidase components gp91-phox and p22-phox in endothelial cells

Normalizing mitochondrial superoxide production blocks three pathways of hyperglycaemic damage

The release of cytochrome c from mitochondria: a primary site for Bcl-2 regulation of apoptosis

Differential accumulation of advanced glycation end products in the course of diabetic retinopathy.

Role of NADPH oxidase and Stat3 in statin-mediated protection against diabetic retinopathy.

Detection and characterization of the product of hydroethidine and intracellular superoxide by HPLC and limitations of fluorescence

Mechanism of autoxidative glycosylation: identification of glyoxal and arabinose as intermediates in the autoxidative modification of proteins by glucose.

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2010

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