Virulence is positively selected by transmission success between mammalian hosts.
about
Evolutionary medicine: update on the relevance to family practiceIncreased bursting glutamatergic neurotransmission in an auditory forebrain area of the zebra finch (Taenopygia guttata) induced by auditory stimulation.Evolution, genomics and epidemiology of Pseudomonas syringae: Challenges in Bacterial Molecular Plant Pathology.Molecular analysis as an aid to assess the public health risk of non-O157 Shiga toxin-producing Escherichia coli strains.Different effects of whole-cell and acellular vaccines on Bordetella transmission.The blessings and curses of intestinal inflammation.A horizontally acquired transcription factor coordinates Salmonella adaptations to host microenvironments.Salmonella phage ST64B encodes a member of the SseK/NleB effector familyA type III effector protease NleC from enteropathogenic Escherichia coli targets NF-κB for degradationPotential origins and horizontal transfer of type III secretion systems and effectors.Within-host evolution decreases virulence in an opportunistic bacterial pathogen.Active Transport of Phosphorylated Carbohydrates Promotes Intestinal Colonization and Transmission of a Bacterial Pathogen.IsaB Inhibits Autophagic Flux to Promote Host Transmission of Methicillin-Resistant Staphylococcus aureus.A type III effector antagonizes death receptor signalling during bacterial gut infection.Targeting of immune signalling networks by bacterial pathogens.Toll-like receptor 4 limits transmission of Bordetella bronchiseptica.Intestinal colonization resistance.Hijacking of death receptor signaling by bacterial pathogen effectors.Bringing down the host: enteropathogenic and enterohaemorrhagic Escherichia coli effector-mediated subversion of host innate immune pathways.The bacterial virulence factor NleA is required for the disruption of intestinal tight junctions by enteropathogenic Escherichia coli.Virulence evolution and the trade-off hypothesis: history, current state of affairs and the future.Pathogen-Mediated Inhibition of Anorexia Promotes Host Survival and Transmission.Probiotic Enterococcus faecalis Symbioflor® down regulates virulence genes of EHEC in vitro and decrease pathogenicity in a Caenorhabditis elegans model.Understanding the host-adapted state of Citrobacter rodentium by transcriptomic analysis.The Serine Protease Autotransporter Pic Modulates Citrobacter rodentium Pathogenesis and Its Innate Recognition by the Host.The evolution of virulence in non-o157 shiga toxin-producing Escherichia coli.The impact of bottlenecks on microbial survival, adaptation and phenotypic switching in host-pathogen interactions.The bacterial virulence factor NleA's involvement in intestinal tight junction disruption during enteropathogenic E. coli infection is independent of its putative PDZ binding domain.Infection biology: Cheats never prosper.Virulence not only costs but also benefits the transmission of a fungal virus.Attaching-and-Effacing Pathogens Exploit Junction Regulatory Activities of N-WASP and SNX9 to Disrupt the Intestinal Barrier.Molecular characterization of Shiga-toxigenic Escherichia coli isolated from diverse sources from India by multi-locus variable number tandem repeat analysis (MLVA).
P2860
Q24644597-752A8A49-38B2-4D03-910F-221E2B04616EQ28727965-0509E6A7-2872-4538-ADF5-3122A81C66FFQ30243898-9EDAEF78-1A88-4520-8633-5FC23D1651A9Q33378528-6B8169D9-0B0A-4DBC-BF18-F3616BB80783Q33684401-9290E9D8-5074-48E5-B6F3-D46E87107CFFQ34052242-C6C95076-8D9D-4D03-A8BC-7D662731B7C8Q34237755-09D6F063-5F43-424F-B3F8-1D29CC2749D9Q34698809-14797D0A-EAA7-4FD1-B52C-73FD1C4DC9BBQ35228516-F9EC24A6-3115-4831-84D3-044084AAB456Q35305175-DC70082F-27AA-41A1-B0B6-9B2E5593A3B5Q35748096-643099FD-F4F3-419A-8D69-57F2DFAC7E59Q35752378-B73BBE63-DB5A-488D-80D3-D690C20BC569Q36269219-62DD5A52-03FF-4028-87AD-4EF6187EAD3CQ37331618-ACEB3104-9B3C-437E-8E64-BF2814FF2558Q37464760-AA53A614-F33A-4F80-B3B0-708A083187E6Q37534202-0BE4ABBB-5BCC-40A3-ADE1-C8533AAFACF6Q38067461-F8B39928-D57F-4876-9280-5B8AEB3F5F95Q38295538-896E3100-AE84-4362-AAFE-5F7526DC4E40Q38318508-1C6D89A6-F5A4-4AF7-AD10-AAC8943841A1Q39807355-166981DC-E72D-4367-A3EB-61EA9256D863Q40004898-72CBCBE1-C024-4661-A55B-FFD815DFBCDEQ40355774-A3F179C4-0162-47FB-94A4-BEBB5430D0BBQ40531822-986AD146-9DC3-46B2-BE35-EC521DDB77C8Q40807932-E1FBFB48-5419-446D-AF43-9B0BB90E59F9Q41304756-F6A8DC98-DE38-4477-A6DD-FFEA76E0D3FEQ41853326-555C78D2-9EFD-4A92-B8AC-D708D8ECE646Q41923111-774A1558-5A2B-4053-822C-8130B52E7041Q42145316-C04D23FA-A177-4180-B0F6-66C78BE21156Q43581710-3888C22D-EDD0-4EC0-B489-CC88AF7CEBAEQ45355932-67411905-B5E4-4B00-BB7A-3F8DA70060D4Q52314672-6DFD3EDE-8EDD-467E-8DBB-03967FE61D05Q54222372-D4A5236D-5817-4B3F-A83E-D4942127D6ED
P2860
Virulence is positively selected by transmission success between mammalian hosts.
description
2007 nî lūn-bûn
@nan
2007 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
name
Virulence is positively selected by transmission success between mammalian hosts.
@ast
Virulence is positively selected by transmission success between mammalian hosts.
@en
Virulence is positively selected by transmission success between mammalian hosts.
@nl
type
label
Virulence is positively selected by transmission success between mammalian hosts.
@ast
Virulence is positively selected by transmission success between mammalian hosts.
@en
Virulence is positively selected by transmission success between mammalian hosts.
@nl
prefLabel
Virulence is positively selected by transmission success between mammalian hosts.
@ast
Virulence is positively selected by transmission success between mammalian hosts.
@en
Virulence is positively selected by transmission success between mammalian hosts.
@nl
P2093
P1433
P1476
Virulence is positively selected by transmission success between mammalian hosts.
@en
P2093
B Brett Finlay
Erin C Boyle
Nat F Brown
P304
P356
10.1016/J.CUB.2007.03.067
P407
P577
2007-04-19T00:00:00Z