Nonopsonic binding of type III Group B Streptococci to human neutrophils induces interleukin-8 release mediated by the p38 mitogen-activated protein kinase pathway.
about
Lectin site interaction with capsular polysaccharide mediates nonimmune phagocytosis of type III group B streptococci.Induction and termination of inflammatory signaling in group B streptococcal sepsis.Antibody against surface-bound C5a peptidase is opsonic and initiates macrophage killing of group B streptococci.Streptococcus suis interactions with the murine macrophage cell line J774: adhesion and cytotoxicity.Interaction of neonatal phagocytes with group B streptococcus: recognition and response.Neutrophils Directly Recognize Group B Streptococci and Contribute to Interleukin-1β Production during Infection.Induction of oxidative stress by Taxol® vehicle Cremophor-EL triggers production of interleukin-8 by peripheral blood mononuclear cells through the mechanism not requiring de novo synthesis of mRNA.Critical role of the complement system in group B streptococcus-induced tumor necrosis factor alpha release.Adenovirus type 7 induces interleukin-8 production via activation of extracellular regulated kinase 1/2.Mechanisms of Actinobacillus actinomycetemcomitans-induced expression of interleukin-8 in gingival epithelial cells.An extracellular Staphylococcus epidermidis polysaccharide: relation to Polysaccharide Intercellular Adhesin and its implication in phagocytosisAcquisition of factor H by a novel surface protein on group B Streptococcus promotes complement degradation.NF-kappaB and the MAP kinases/AP-1 pathways are both involved in interleukin-6 and interleukin-8 expression in fibroblast-like synoviocytes stimulated by protein I/II, a modulin from oral streptococci.Interaction of Neisseria meningitidis with human brain microvascular endothelial cells: role of MAP- and tyrosine kinases in invasion and inflammatory cytokine release.Highly purified lipoteichoic acid activates neutrophil granulocytes and delays their spontaneous apoptosis via CD14 and TLR2.Neonatal neutrophils stimulated by group B Streptococcus induce a proinflammatory T-helper cell bias.
P2860
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P2860
Nonopsonic binding of type III Group B Streptococci to human neutrophils induces interleukin-8 release mediated by the p38 mitogen-activated protein kinase pathway.
description
2000 nî lūn-bûn
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2000 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
Nonopsonic binding of type III ...... ivated protein kinase pathway.
@ast
Nonopsonic binding of type III ...... ivated protein kinase pathway.
@en
Nonopsonic binding of type III ...... ivated protein kinase pathway.
@nl
type
label
Nonopsonic binding of type III ...... ivated protein kinase pathway.
@ast
Nonopsonic binding of type III ...... ivated protein kinase pathway.
@en
Nonopsonic binding of type III ...... ivated protein kinase pathway.
@nl
prefLabel
Nonopsonic binding of type III ...... ivated protein kinase pathway.
@ast
Nonopsonic binding of type III ...... ivated protein kinase pathway.
@en
Nonopsonic binding of type III ...... ivated protein kinase pathway.
@nl
P2093
P2860
P1476
Nonopsonic binding of type III ...... ivated protein kinase pathway.
@en
P2093
E A Albanyan
J G Vallejo
M S Edwards
P2860
P304
P356
10.1128/IAI.68.4.2053-2060.2000
P407
P577
2000-04-01T00:00:00Z