Myocardial smad4 is essential for cardiogenesis in mouse embryos. - Wikidata - wikimedia - TriplyDB

Myocardial smad4 is essential for cardiogenesis in mouse embryos.

Myocardial smad4 is essential for cardiogenesis in mouse embryos.

http://www.wikidata.org/entity/Q34009514

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SM22alpha-targeted deletion of bone morphogenetic protein receptor 1A in mice impairs cardiac and vascular development, and influences organogenesis Abcc6 deficiency causes increased infarct size and apoptosis in a mouse cardiac ischemia-reperfusion model BMP signaling regulates sympathetic nervous system development through Smad4-dependent and -independent pathways Sertad1 encodes a novel transcriptional co-activator of SMAD1 in mouse embryonic hearts Vessel wall BAMBI contributes to hemostasis and thrombus stability.Expression of sumoylation deficient Nkx2.5 mutant in Nkx2.5 haploinsufficient mice leads to congenital heart defects.Deletion of exon 20 of the Familial Dysautonomia gene Ikbkap in mice causes developmental delay, cardiovascular defects, and early embryonic lethality Myocardial deletion of Smad4 using a novel α skeletal muscle actin Cre recombinase transgenic mouse causes misalignment of the cardiac outflow tract Hyaluronan esters drive Smad gene expression and signaling enhancing cardiogenesis in mouse embryonic and human mesenchymal stem cells Cell autonomous requirement of endocardial Smad4 during atrioventricular cushion development in mouse embryos Loss of β2-spectrin prevents cardiomyocyte differentiation and heart development.Redundant and dosage sensitive requirements for Fgf3 and Fgf10 in cardiovascular development Persistent Noggin arrests cardiomyocyte morphogenesis and results in early in utero lethality.Smad4 regulates ureteral smooth muscle cell differentiation during mouse embryogenesis Generation of a tamoxifen inducible Tnnt2MerCreMer knock-in mouse model for cardiac studies Tbx20 regulation of cardiac cell proliferation and lineage specialization during embryonic and fetal development in vivo.Transforming growth factor beta signaling in adult cardiovascular diseases and repair.Transient expression of FRNK reveals stage-specific requirement for focal adhesion kinase activity in cardiac growth Homeobox C9 is not potentially related to congenital heart disease in Chinese patients Vascular smooth muscle cell Smad4 gene is important for mouse vascular development.Myocardial Mycn is essential for mouse ventricular wall morphogenesis.In vivo monitoring of cardiomyocyte proliferation to identify chemical modifiers of heart regeneration.Disruption of Smad4 in neural crest cells leads to mid-gestation death with pharyngeal arch, craniofacial and cardiac defects Requirement of TCTG(G/C) Direct Repeats and Overlapping GATA Site for Maintaining the Cardiac-Specific Expression of Cardiac troponin T in Developing and Adult Mice.Cardiomyocyte-specific inactivation of thyroid hormone in pathologic ventricular hypertrophy: an adaptative response or part of the problem?Histology atlas of the developing mouse heart with emphasis on E11.5 to E18.5.microRNA expression profiling and functional annotation analysis of their targets modulated by oxidative stress during embryonic heart development in diabetic mice.CHD7 interacts with BMP R-SMADs to epigenetically regulate cardiogenesis in mice BMP signaling in congenital heart disease: new developments and future directions.Alcohol Exposure Causes Overexpression of Heart Development-Related Genes by Affecting the Histone H3 Acetylation via BMP Signaling Pathway in Cardiomyoblast Cells.A Functional Variant of SMAD4 Enhances Thoracic Aortic Aneurysm and Dissection Risk through Promoting Smooth Muscle Cell Apoptosis and Proteoglycan Degradation.Expression of Id2 in the second heart field and cardiac defects in Id2 knock-out mice.Myocardial Bmp2 gain causes ectopic EMT and promotes cardiomyocyte proliferation and immaturity.

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P2860

Myocardial smad4 is essential for cardiogenesis in mouse embryos.

http://www.wikidata.org/entity/Q34009514

description

2007 nî lūn-bûn

@nan

2007 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2007 թվականի հունիսին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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name

Myocardial smad4 is essential for cardiogenesis in mouse embryos.

@ast

Myocardial smad4 is essential for cardiogenesis in mouse embryos.

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Myocardial smad4 is essential for cardiogenesis in mouse embryos.

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type

label

Myocardial smad4 is essential for cardiogenesis in mouse embryos.

@ast

Myocardial smad4 is essential for cardiogenesis in mouse embryos.

@en

Myocardial smad4 is essential for cardiogenesis in mouse embryos.

@nl

prefLabel

Myocardial smad4 is essential for cardiogenesis in mouse embryos.

@ast

Myocardial smad4 is essential for cardiogenesis in mouse embryos.

@en

Myocardial smad4 is essential for cardiogenesis in mouse embryos.

@nl

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CIRCRESAHA.107.155630

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Circulation Research

P1476

Myocardial smad4 is essential for cardiogenesis in mouse embryos.

@en

P2093

Chu-xia Deng

http://www.w3.org/2001/XMLSchema#string

Kai Jiao

http://www.w3.org/2001/XMLSchema#string

Lanying Song

http://www.w3.org/2001/XMLSchema#string

Qin Wang

http://www.w3.org/2001/XMLSchema#string

Wensheng Yan

http://www.w3.org/2001/XMLSchema#string

Xinbin Chen

http://www.w3.org/2001/XMLSchema#string

P2860

Direct induction of cyclin D2 by Myc contributes to cell cycle progression and sequestration of p27.

Identification of mZnf8, a mouse Krüppel-like transcriptional repressor, as a novel nuclear interaction partner of Smad1.

Mechanisms of TGF-beta signaling from cell membrane to the nucleus

Hematopoiesis controlled by distinct TIF1gamma and Smad4 branches of the TGFbeta pathway

New insights into TGF-beta-Smad signalling

Smad transcription factors

GPX2, a direct target of p63, inhibits oxidative stress-induced apoptosis in a p53-dependent manner

Cardiomyocyte-specific deletion of the coxsackievirus and adenovirus receptor results in hyperplasia of the embryonic left ventricle and abnormalities of sinuatrial valves

Bmp2 is essential for cardiac cushion epithelial-mesenchymal transition and myocardial patterning

Role of the NF-ATc transcription factor in morphogenesis of cardiac valves and septum

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10.1161/CIRCRESAHA.107.155630

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3

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101

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4123531

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