Osteocyte-derived RANKL is a critical mediator of the increased bone resorption caused by dietary calcium deficiency.
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The Role of IL-17 and TH17 Cells in the Bone Catabolic Activity of PTHReproduction and breast cancer riskOsteocytes, not Osteoblasts or Lining Cells, are the Main Source of the RANKL Required for Osteoclast Formation in Remodeling BoneParathyroid hormone: anabolic and catabolic actions on the skeletonTartrate-resistant acid phosphatase (TRAP) co-localizes with receptor activator of NF-KB ligand (RANKL) and osteoprotegerin (OPG) in lysosomal-associated membrane protein 1 (LAMP1)-positive vesicles in rat osteoblasts and osteocytes.T cell-expressed CD40L potentiates the bone anabolic activity of intermittent PTH treatment.IL-17A Is Increased in Humans with Primary Hyperparathyroidism and Mediates PTH-Induced Bone Loss in MiceUnique Distal Enhancers Linked to the Mouse Tnfsf11 Gene Direct Tissue-Specific and Inflammation-Induced Expression of RANKL.T cells, osteoblasts, and osteocytes: interacting lineages key for the bone anabolic and catabolic activities of parathyroid hormone.Parathyroid hormone 1-34 and skeletal anabolic action: The use of parathyroid hormone in bone formation.A DNA segment spanning the mouse Tnfsf11 transcription unit and its upstream regulatory domain rescues the pleiotropic biologic phenotype of the RANKL null mouse.The role of the wnt/β-catenin signaling pathway in formation and maintenance of bone and teeth.How rare bone diseases have informed our knowledge of complex diseases.Parathyroid Diseases and T Cells.Cardiovascular risk assessment in osteoporotic patients using osteoprotegerin as a reliable predictive biochemical marker.Neural crest-mediated bone resorption is a determinant of species-specific jaw lengthCortical bone loss caused by glucocorticoid excess requires RANKL production by osteocytes and is associated with reduced OPG expression in mice.Control of Bone Anabolism in Response to Mechanical Loading and PTH by Distinct Mechanisms Downstream of the PTH Receptor.Diet and Exercise: a Match Made in Bone.Mean-field approach to evolving spatial networks, with an application to osteocyte network formation.RANKL (Receptor Activator of NFκB Ligand) Produced by Osteocytes Is Required for the Increase in B Cells and Bone Loss Caused by Estrogen Deficiency in Mice.Immunoporosis: Immunology of Osteoporosis-Role of T Cells.MMP14 is a novel target of PTH signaling in osteocytes that controls resorption by regulating soluble RANKL production.TNF‑α and RANKL promote osteoclastogenesis by upregulating RANK via the NF‑κB pathway.Parathyroid Hormone Signaling in Osteocytes
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P2860
Osteocyte-derived RANKL is a critical mediator of the increased bone resorption caused by dietary calcium deficiency.
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2014 nî lūn-bûn
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2014 թուականի Յունիսին հրատարակուած գիտական յօդուած
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2014 թվականի հունիսին հրատարակված գիտական հոդված
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2014年の論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年论文
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Osteocyte-derived RANKL is a c ...... by dietary calcium deficiency.
@ast
Osteocyte-derived RANKL is a c ...... by dietary calcium deficiency.
@en
Osteocyte-derived RANKL is a c ...... by dietary calcium deficiency.
@nl
type
label
Osteocyte-derived RANKL is a c ...... by dietary calcium deficiency.
@ast
Osteocyte-derived RANKL is a c ...... by dietary calcium deficiency.
@en
Osteocyte-derived RANKL is a c ...... by dietary calcium deficiency.
@nl
prefLabel
Osteocyte-derived RANKL is a c ...... by dietary calcium deficiency.
@ast
Osteocyte-derived RANKL is a c ...... by dietary calcium deficiency.
@en
Osteocyte-derived RANKL is a c ...... by dietary calcium deficiency.
@nl
P2093
P2860
P1433
P1476
Osteocyte-derived RANKL is a c ...... by dietary calcium deficiency.
@en
P2093
Charles A O'Brien
Jeff D Thostenson
Jinhu Xiong
Marilina Piemontese
Robert S Weinstein
Stavros C Manolagas
P2860
P304
P356
10.1016/J.BONE.2014.06.006
P577
2014-06-14T00:00:00Z