Protection of cardiac mitochondria by diazoxide and protein kinase C: implications for ischemic preconditioning. - Wikidata - wikimedia - TriplyDB

Protection of cardiac mitochondria by diazoxide and protein kinase C: implications for ischemic preconditioning.

Protection of cardiac mitochondria by diazoxide and protein kinase C: implications for ischemic preconditioning.

http://www.wikidata.org/entity/Q34017639

about

Protein kinase Cepsilon interacts with and inhibits the permeability transition pore in cardiac mitochondria Quinones and aromatic chemical compounds in particulate matter induce mitochondrial dysfunction: implications for ultrafine particle toxicity The ubiquitin-proteasome system and cardiovascular disease Past and present course of cardioprotection against ischemia-reperfusion injury The ubiquitin proteasome system and myocardial ischemia The ubiquitin-proteasome system in myocardial ischaemia and preconditioning.Glycolytic oscillations in single ischemic cardiomyocytes at near anoxia.Preconditioning involves selective mitophagy mediated by Parkin and p62/SQSTM1.Analysis of mitochondrial 3D-deformation in cardiomyocytes during active contraction reveals passive structural anisotropy of orthogonal short axes.Glycogen synthase kinase-3beta mediates convergence of protection signaling to inhibit the mitochondrial permeability transition pore Inhibit GSK-3beta or there's heartbreak dead ahead.Mitochondrial ion channels: gatekeepers of life and death.Proteasome functional insufficiency in cardiac pathogenesis.Evidence for mitochondrial K+ channels and their role in cardioprotection.Protective Effects of Kaempferol against Myocardial Ischemia/Reperfusion Injury in Isolated Rat Heart via Antioxidant Activity and Inhibition of Glycogen Synthase Kinase-3β.Altered expression of mitochondrial electron transport chain proteins and improved myocardial energetic state during late ischemic preconditioning.Role of the anion nitrite in ischemia-reperfusion cytoprotection and therapeutics.Mitochondrial K(ATP) channels in cell survival and death.Mitochondria and ischemia/reperfusion injury.Diacylglycerols activate mitochondrial cationic channel(s) and release sequestered Ca(2+).Prenatal cocaine exposure abolished ischemic preconditioning-induced protection in adult male rat hearts: role of PKCepsilon.Dual effects of neuroprotection and neurotoxicity by general anesthetics: role of intracellular calcium homeostasis.Diazoxide Attenuates Postresuscitation Brain Injury in a Rat Model of Asphyxial Cardiac Arrest by Opening Mitochondrial ATP-Sensitive Potassium Channels.Mechanisms of load dependency of myocardial ischemia reperfusion injury.Anesthetic-induced preconditioning delays opening of mitochondrial permeability transition pore via protein Kinase C-epsilon-mediated pathway.Cardioprotective signaling to mitochondria Early long-term L-T3 replacement rescues mitochondria and prevents ischemic cardiac remodelling in rats.Suppression of Excessive Histone Deacetylases Activity in Diabetic Hearts Attenuates Myocardial Ischemia/Reperfusion Injury via Mitochondria Apoptosis Pathway.Inhibition of mitochondrial membrane permeability as a putative pharmacological target for cardioprotection.Interaction of mitochondrial potassium channels with the permeability transition pore.Nitrite as a mediator of ischemic preconditioning and cytoprotection.Cellular and molecular events in ischemic preconditioning: potential therapeutic applications in cardioprotection.Characterization of human cardiac mitochondrial ATP-sensitive potassium channel and its regulation by phorbol ester in vitro.Diesel exhaust particle extracts and associated polycyclic aromatic hydrocarbons inhibit Cox-2-dependent prostaglandin synthesis in murine macrophages and fibroblasts.Role of arachidonic acid, lipoxygenase, and mitochondrial depolarization in reperfusion arrhythmias.Protective effect of exogenous nitrite in postoperative ileus.Ca2+ dysregulation induces mitochondrial depolarization and apoptosis: role of Na+/Ca2+ exchanger and AKT.Targeting nucleotide-requiring enzymes: implications for diazoxide-induced cardioprotection.Effects of fatty acids in isolated mitochondria: implications for ischemic injury and cardioprotection.Sevoflurane depolarizes pre-synaptic mitochondria in the central nervous system.

P2860

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P2860

Protection of cardiac mitochondria by diazoxide and protein kinase C: implications for ischemic preconditioning.

http://www.wikidata.org/entity/Q34017639

description

2002 nî lūn-bûn

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2002 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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Protection of cardiac mitochon ...... for ischemic preconditioning.

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Protection of cardiac mitochon ...... for ischemic preconditioning.

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Protection of cardiac mitochon ...... for ischemic preconditioning.

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Protection of cardiac mitochon ...... for ischemic preconditioning.

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Protection of cardiac mitochon ...... for ischemic preconditioning.

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Protection of cardiac mitochon ...... for ischemic preconditioning.

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Protection of cardiac mitochon ...... for ischemic preconditioning.

@en

P2093

Henry M Honda

http://www.w3.org/2001/XMLSchema#string

James N Weiss

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Paavo Korge

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P2860

Bioenergetic consequences of opening the ATP-sensitive K(+) channel of heart mitochondria.

Diazoxide protects mitochondria from anoxic injury: implications for myopreservation.

Mitochondria: a new target for K channel openers?

Sarcolemmal versus mitochondrial ATP-sensitive K+ channels and myocardial preconditioning.

Mitochondrial ATP-dependent potassium channels. Viable candidate effectors of ischemic preconditioning.

ATP-Sensitive potassium channels: a review of their cardioprotective pharmacology.

Molecular basis of protein kinase C-induced activation of ATP-sensitive potassium channels.

Regulation of cloned ATP-sensitive K channels by phosphorylation, MgADP, and phosphatidylinositol bisphosphate (PIP(2)): a study of channel rundown and reactivation

Elucidating the molecular mechanism of the permeability transition pore and its role in reperfusion injury of the heart.

The mitochondrial permeability transition.

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3312-3317

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10.1073/PNAS.052713199

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99

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11867760

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