Bioluminescent imaging reveals divergent viral pathogenesis in two strains of Stat1-deficient mice, and in αßγ interferon receptor-deficient mice.
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Functional genomics reveals an essential and specific role for Stat1 in protection of the central nervous system following herpes simplex virus corneal infectionNeuronal Interferon Signaling Is Required for Protection against Herpes Simplex Virus Replication and Pathogenesis.Corneal replication is an interferon response-independent bottleneck for virulence of herpes simplex virus 1 in the absence of virion host shutoff.Role of the DNA Sensor STING in Protection from Lethal Infection following Corneal and Intracerebral Challenge with Herpes Simplex Virus 1.Rabbit and mouse models of HSV-1 latency, reactivation, and recurrent eye diseasesExpression of interferon-induced antiviral genes is delayed in a STAT1 knockout mouse model of Crimean-Congo hemorrhagic fever.p53 Is a Host Cell Regulator during Herpes Simplex Encephalitis.Synergistic control of herpes simplex virus pathogenesis by IRF-3, and IRF-7 revealed through non-invasive bioluminescence imaging.Immune- and Nonimmune-Compartment-Specific Interferon Responses Are Critical Determinants of Herpes Simplex Virus-Induced Generalized Infections and Acute Liver Failure.Herpesvirus transport to the nervous system and back again.The differential interferon responses of two strains of Stat1-deficient mice do not alter susceptibility to HSV-1 and VSV in vivo.Cellular Protein WDR11 Interacts with Specific Herpes Simplex Virus Proteins at the trans-Golgi Network To Promote Virus Replication.Innate defense mechanisms against HSV-1 infection in the target tissues, skin and brain.A bright future for bioluminescent imaging in viral researchInnate Immune Mechanisms and Herpes Simplex Virus Infection and Disease.Neuronal IFN signaling is dispensable for the establishment of HSV-1 latency.Herpesvirus-Associated Lymphadenitis Distorts Fibroblastic Reticular Cell Microarchitecture and Attenuates CD8 T Cell Responses to Neurotropic Infection in Mice Lacking the STING-IFNα/β Defense Pathways.Modulation of antitumour immune responses by intratumoural Stat1 expression.Insights into the pathogenesis of herpes simplex encephalitis from mouse modelsA Syngeneic Mouse Model of Epithelial Ovarian Cancer Port Site Metastases
P2860
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P2860
Bioluminescent imaging reveals divergent viral pathogenesis in two strains of Stat1-deficient mice, and in αßγ interferon receptor-deficient mice.
description
2011 nî lūn-bûn
@nan
2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Bioluminescent imaging reveals ...... feron receptor-deficient mice.
@ast
Bioluminescent imaging reveals ...... feron receptor-deficient mice.
@en
Bioluminescent imaging reveals ...... feron receptor-deficient mice.
@nl
type
label
Bioluminescent imaging reveals ...... feron receptor-deficient mice.
@ast
Bioluminescent imaging reveals ...... feron receptor-deficient mice.
@en
Bioluminescent imaging reveals ...... feron receptor-deficient mice.
@nl
prefLabel
Bioluminescent imaging reveals ...... feron receptor-deficient mice.
@ast
Bioluminescent imaging reveals ...... feron receptor-deficient mice.
@en
Bioluminescent imaging reveals ...... feron receptor-deficient mice.
@nl
P2093
P2860
P1433
P1476
Bioluminescent imaging reveals ...... rferon receptor-deficient mice
@en
P2093
Brent L Berwin
David A Leib
David R Piwnica-Worms
Lynne Collins
Tracy Jo Pasieka
Yufei Chen
Zachary M Parker
P2860
P304
P356
10.1371/JOURNAL.PONE.0024018
P407
P577
2011-09-07T00:00:00Z