Innate immune factors modulate ethanol interaction with GABAergic transmission in mouse central amygdala. - Wikidata - wikimedia - TriplyDB

Innate immune factors modulate ethanol interaction with GABAergic transmission in mouse central amygdala.

Innate immune factors modulate ethanol interaction with GABAergic transmission in mouse central amygdala.

http://www.wikidata.org/entity/Q34020259

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CRF-amplified neuronal TLR4/MCP-1 signaling regulates alcohol self-administration.Neuroimmune pathways in alcohol consumption: evidence from behavioral and genetic studies in rodents and humans IL-1 interacts with ethanol effects on GABAergic transmission in the mouse central amygdala Neuroimmune mechanisms of alcohol and drug addiction.Persistent adaptation by chronic alcohol is facilitated by neuroimmune activation linked to stress and CRF Alcohol resistance in Drosophila is modulated by the Toll innate immune pathway Gut-liver axis in alcoholic liver disease Targeting the Toll of Drug Abuse: The Translational Potential of Toll-Like Receptor 4.Disentangling the Role of Astrocytes in Alcohol Use Disorder.Glial and Neuroimmune Mechanisms as Critical Modulators of Drug Use and Abuse.Evaluation of TLR4 Inhibitor, T5342126, in Modulation of Ethanol-Drinking Behavior in Alcohol-Dependent Mice.Genetic and Pharmacologic Manipulation of TLR4 Has Minimal Impact on Ethanol Consumption in Rodents.Microglial-specific transcriptome changes following chronic alcohol consumption.Ethanol and Cytokines in the Central Nervous System.Role of the innate immune system in the neuropathological consequences induced by adolescent binge drinking.Sedative and Motor Incoordination Effects of Ethanol in Mice Lacking CD14, TLR2, TLR4, or MyD88.Chronic ethanol consumption: role of TLR3/TRIF-dependent signaling.Increased expression of M1 and M2 phenotypic markers in isolated microglia after four-day binge alcohol exposure in male rats.Role of TLR4 in the Modulation of Central Amygdala GABA Transmission by CRF Following Restraint Stress.

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Innate immune factors modulate ethanol interaction with GABAergic transmission in mouse central amygdala.

http://www.wikidata.org/entity/Q34020259

description

2014 nî lūn-bûn

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2014 թուականի Մարտին հրատարակուած գիտական յօդուած

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2014 թվականի մարտին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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Innate immune factors modulate ...... ion in mouse central amygdala.

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J.BBI.2014.03.007

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Brain, Behavior, and Immunity

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Innate immune factors modulate ...... ion in mouse central amygdala.

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Edward Roberts

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George R Siggins

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Kenner C Rice

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Marisa Roberto

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Michal Bajo

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R Adron Harris

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Samuel G Madamba

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Vasudeva N Sagi

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Yuri A Blednov

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P2860

Binge alcohol drinking is associated with GABAA alpha2-regulated Toll-like receptor 4 (TLR4) expression in the central amygdala

Systemic LPS causes chronic neuroinflammation and progressive neurodegeneration

Neurocircuitry of addiction

The μ-opioid receptor and treatment response to naltrexone

Microglial morphology and dynamic behavior is regulated by ionotropic glutamatergic and GABAergic neurotransmission

Evidence that opioids may have toll-like receptor 4 and MD-2 effects

Involvement of TLR4/type I IL-1 receptor signaling in the induction of inflammatory mediators and cell death induced by ethanol in cultured astrocytes

Glucuronic acid and the ethanol metabolite ethyl-glucuronide cause toll-like receptor 4 activation and enhanced pain

TLR4, ethanol, and lipid rafts: a new mechanism of ethanol action with implications for other receptor-mediated effects

Non-stereoselective reversal of neuropathic pain by naloxone and naltrexone: involvement of toll-like receptor 4 (TLR4)

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191-202

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scholarly article

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10.1016/J.BBI.2014.03.007

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40

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2014-03-24T00:00:00Z

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261184872

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24675033

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