Role of HLA-G in tumor escape through expansion of myeloid-derived suppressor cells and cytokinic balance in favor of Th2 versus Th1/Th17.
about
Multimeric structures of HLA-G isoforms function through differential binding to LILRB receptorsMesenchymal Stem Cells and Myeloid Derived Suppressor Cells: Common Traits in Immune RegulationThe dual role of HLA-G in cancerRole of non-classical MHC class I molecules in cancer immunosuppressionReciprocal relationship between myeloid-derived suppressor cells and T cellsImmunomodulatory properties of HLA-G in infectious diseasesHLA-dependent tumour development: a role for tumour associate macrophages?Interplay between myeloid-derived suppressor cells (MDSCs) and Th17 cells: foe or friend?The Pathophysiological Impact of HLA Class Ia and HLA-G Expression and Regulatory T Cells in Malignant Melanoma: A ReviewThe dark side of dendritic cells: development and exploitation of tolerogenic activity that favor tumor outgrowth and immune escape.Synthetic HLA-G proteins for therapeutic use in transplantation.HLA-G expression is an independent predictor for improved survival in high grade ovarian carcinomasHigh plasma levels of HLA-G are associated with low birth weight and with an increased risk of malaria in infancyIdentification of a novel HLA-G+ regulatory population in blood: expansion after allogeneic transplantation and de novo HLA-G expression at graft-versus-host disease sitesRescuing lymphocytes from HLA-G immunosuppressive effects mediated by the tumor microenvironment.Antiangiogenic therapy for cancer: an updateHLA-G expression in cancers: roles in immune evasion, metastasis and target for therapyHuman leukocyte antigen-G is frequently expressed in glioblastoma and may be induced in vitro by combined 5-aza-2'-deoxycytidine and interferon-γ treatments: results from a multicentric study.Hypoxia inducible factor-1 mediates the expression of the immune checkpoint HLA-G in glioma cells through hypoxia response element located in exon 2.HLA-G dimers in the prolongation of kidney allograft survival.T-cell tolerance in cancer.New insights into HLA-G mediated tolerance.Novel landscape of HLA-G isoforms expressed in clear cell renal cell carcinoma patients.Importance of the plasma soluble HLA-G levels for prognostic stratification with traditional prognosticators in colorectal cancer.Soluble monomers, dimers and HLA-G-expressing extracellular vesicles: the three dimensions of structural complexity to use HLA-G as a clinical biomarker.The Potential of HLA-G-Bearing Extracellular Vesicles as a Future Element in HLA-G Immune Biology.Trogocytic intercellular membrane exchanges among hematological tumors.Advances and challenges: dendritic cell vaccination strategies for glioblastoma.Circulating and disseminated tumour cells - mechanisms of immune surveillance and escape.Tuning cancer fate: the unremitting role of host immunity.HLA-G expression is associated with metastasis and poor survival in the Balb/c nu/nu murine tumor model with ovarian cancer.NKG2D ligand RAE1ε induces generation and enhances the inhibitor function of myeloid-derived suppressor cells in mice.Co-expression of ILT4/HLA-G in human non-small cell lung cancer correlates with poor prognosis and ILT4-HLA-G interaction activates ERK signaling.Amnion-derived multipotent progenitor cells support allograft tolerance induction.Granulocytic Myeloid-Derived Suppressor Cells Accumulate in Human Placenta and Polarize toward a Th2 Phenotype.Genetic and epigenetic regulation of major histocompatibility complex class I gene expression in bovine trophoblast cells.Human leukocyte antigen-G expression and polymorphisms promote cancer development and guide cancer diagnosis/treatment.T cell infiltration into Ewing sarcomas is associated with local expression of immune-inhibitory HLA-G.HLA-G promotes myeloid-derived suppressor cell accumulation and suppressive activity during human pregnancy through engagement of the receptor ILT4.Sonodynamic Therapy Assisted Immunotherapy: A Novel Modality for Cancer Treatment.
P2860
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P2860
Role of HLA-G in tumor escape through expansion of myeloid-derived suppressor cells and cytokinic balance in favor of Th2 versus Th1/Th17.
description
2011 nî lūn-bûn
@nan
2011 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Role of HLA-G in tumor escape ...... favor of Th2 versus Th1/Th17.
@ast
Role of HLA-G in tumor escape ...... favor of Th2 versus Th1/Th17.
@en
Role of HLA-G in tumor escape ...... favor of Th2 versus Th1/Th17.
@nl
type
label
Role of HLA-G in tumor escape ...... favor of Th2 versus Th1/Th17.
@ast
Role of HLA-G in tumor escape ...... favor of Th2 versus Th1/Th17.
@en
Role of HLA-G in tumor escape ...... favor of Th2 versus Th1/Th17.
@nl
prefLabel
Role of HLA-G in tumor escape ...... favor of Th2 versus Th1/Th17.
@ast
Role of HLA-G in tumor escape ...... favor of Th2 versus Th1/Th17.
@en
Role of HLA-G in tumor escape ...... favor of Th2 versus Th1/Th17.
@nl
P2093
P1433
P1476
Role of HLA-G in tumor escape ...... favor of Th2 versus Th1/Th17.
@en
P2093
Edgardo D Carosella
Nathalie Rouas-Freiss
Sophie Agaugué
P304
P356
10.1182/BLOOD-2010-07-294389
P407
P577
2011-04-11T00:00:00Z