Accumulation of oxidative DNA damage in brain mitochondria in mouse model of hereditary ferritinopathy.
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Neurodegeneration with brain iron accumulation: update on pathogenic mechanismsIron chelation and multiple sclerosisNeuroferritinopathy: From ferritin structure modification to pathogenetic mechanismThe role of iron in brain ageing and neurodegenerative disorders.Pathogenic implications of iron accumulation in multiple sclerosis.Brain, blood, and iron: perspectives on the roles of erythrocytes and iron in neurodegeneration.Abnormal iron metabolism in fibroblasts from a patient with the neurodegenerative disease hereditary ferritinopathy.Iron loading-induced aggregation and reduction of iron incorporation in heteropolymeric ferritin containing a mutant light chain that causes neurodegeneration.Effect of Systemic Iron Overload and a Chelation Therapy in a Mouse Model of the Neurodegenerative Disease Hereditary FerritinopathyA novel neuroferritinopathy mouse model (FTL 498InsTC) shows progressive brain iron dysregulation, morphological signs of early neurodegeneration and motor coordination deficits.On the complexity of clinical and molecular bases of neurodegeneration with brain iron accumulation.Review: Insights into molecular mechanisms of disease in neurodegeneration with brain iron accumulation: unifying theories.Abnormal iron homeostasis and neurodegeneration.Brain iron homeostasis: from molecular mechanisms to clinical significance and therapeutic opportunitiesOxidative genome damage and its repair in neurodegenerative diseases: function of transition metals as a double-edged sword.Milestones in atypical and secondary Parkinsonisms.Syndromes of neurodegeneration with brain iron accumulation (NBIA): an update on clinical presentations, histological and genetic underpinnings, and treatment considerations.Excess iron harms the brain: the syndromes of neurodegeneration with brain iron accumulation (NBIA).Genetics and Pathophysiology of Neurodegeneration with Brain Iron Accumulation (NBIA).Differential effect of nimodipine in attenuating iron-induced toxicity in brain- and blood-brain barrier-associated cell types.Blocking of L-type calcium channels protects hippocampal and nigral neurons against iron neurotoxicity. The role of L-type calcium channels in iron-induced neurotoxicity.Quantitative Susceptibility Mapping of the Thalamus: Relationships with Thalamic Volume, Total Gray Matter Volume, and T2 Lesion Burden.
P2860
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P2860
Accumulation of oxidative DNA damage in brain mitochondria in mouse model of hereditary ferritinopathy.
description
2010 nî lūn-bûn
@nan
2010 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Accumulation of oxidative DNA ...... of hereditary ferritinopathy.
@ast
Accumulation of oxidative DNA ...... of hereditary ferritinopathy.
@en
Accumulation of oxidative DNA ...... of hereditary ferritinopathy.
@nl
type
label
Accumulation of oxidative DNA ...... of hereditary ferritinopathy.
@ast
Accumulation of oxidative DNA ...... of hereditary ferritinopathy.
@en
Accumulation of oxidative DNA ...... of hereditary ferritinopathy.
@nl
prefLabel
Accumulation of oxidative DNA ...... of hereditary ferritinopathy.
@ast
Accumulation of oxidative DNA ...... of hereditary ferritinopathy.
@en
Accumulation of oxidative DNA ...... of hereditary ferritinopathy.
@nl
P2093
P2860
P1433
P1476
Accumulation of oxidative DNA ...... of hereditary ferritinopathy.
@en
P2093
Ella W Englander
Ruben Vidal
Xiaoling Deng
P2860
P356
10.1016/J.NEULET.2010.05.025
P407
P577
2010-05-15T00:00:00Z