In vivo functional role of the Drosophila hyperkinetic beta subunit in gating and inactivation of Shaker K+ channels.
about
Lithium-Responsive Seizure-Like Hyperexcitability Is Caused by a Mutation in the Drosophila Voltage-Gated Sodium Channel Gene paralytic.Shaker and Shal mediate transient calcium-independent potassium current in a Drosophila flight motoneuron.Effects of social isolation on neuromuscular excitability and aggressive behaviors in Drosophila: altered responses by Hk and gsts1, two mutations implicated in redox regulationKv1 potassium channel complexes in vivo require Kvbeta2 subunits in dorsal spinal neurons.Fast inactivation of Shal (K(v)4) K+ channels is regulated by the novel interactor SKIP3 in Drosophila neuronsReduced K+ channel inactivation, spike broadening, and after-hyperpolarization in Kvbeta1.1-deficient mice with impaired learning.Flight and seizure motor patterns in Drosophila mutants: simultaneous acoustic and electrophysiological recordings of wing beats and flight muscle activityEffects of hyperkinetic, a beta subunit of Shaker voltage-dependent K+ channels, on the oxidation state of presynaptic nerve terminals.Effects of mutant Drosophila K+ channel subunits on habituation of the olfactory jump response.Modulation of the frequency response of Shaker potassium channels by the quiver peptide suggesting a novel extracellular interaction mechanism.Treatment with pentylenetetrazole (PTZ) and 4-aminopyridine (4-AP) differently affects survival, locomotor activity, and biochemical markers in Drosophila melanogaster.A modified minimal hemolymph-like solution, HL3.1, for physiological recordings at the neuromuscular junctions of normal and mutant Drosophila larvae.Interaction of the K channel beta subunit, Hyperkinetic, with eag family members.Morphometric description of the wandering behavior in Drosophila larvae: aberrant locomotion in Na+ and K+ channel mutants revealed by computer-assisted motion analysis.Auxiliary Hyperkinetic beta subunit of K+ channels: regulation of firing properties and K+ currents in Drosophila neurons.Genetic modifications of seizure susceptibility and expression by altered excitability in Drosophila Na(+) and K(+) channel mutants.
P2860
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P2860
In vivo functional role of the Drosophila hyperkinetic beta subunit in gating and inactivation of Shaker K+ channels.
description
1996 nî lūn-bûn
@nan
1996 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1996 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
name
In vivo functional role of the ...... ivation of Shaker K+ channels.
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In vivo functional role of the ...... ivation of Shaker K+ channels.
@en
In vivo functional role of the ...... ivation of Shaker K+ channels.
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In vivo functional role of the ...... ivation of Shaker K+ channels.
@ast
In vivo functional role of the ...... ivation of Shaker K+ channels.
@en
In vivo functional role of the ...... ivation of Shaker K+ channels.
@nl
prefLabel
In vivo functional role of the ...... ivation of Shaker K+ channels.
@ast
In vivo functional role of the ...... ivation of Shaker K+ channels.
@en
In vivo functional role of the ...... ivation of Shaker K+ channels.
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P2860
P1433
P1476
In vivo functional role of the ...... ivation of Shaker K+ channels.
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P2860
P304
P356
10.1016/S0006-3495(96)79510-3
P407
P577
1996-12-01T00:00:00Z