Alpha2 nicotine receptors function as a molecular switch to continuously excite a subset of interneurons in rat hippocampal circuits.
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Regulation of hippocampal inhibitory circuits by nicotinic acetylcholine receptorsQuantitative analysis of the heteromeric neuronal nicotinic receptors in the rat hippocampusCellular, molecular, and genetic substrates underlying the impact of nicotine on learning.Nicotine facilitates long-term potentiation induction in oriens-lacunosum moleculare cells via Ca2+ entry through non-alpha7 nicotinic acetylcholine receptorsSystemic Nicotine Increases Gain and Narrows Receptive Fields in A1 via Integrated Cortical and Subcortical ActionsEndogenous ACh suppresses LTD induction and nicotine relieves the suppression via different nicotinic ACh receptor subtypes in the mouse hippocampusOLM interneurons differentially modulate CA3 and entorhinal inputs to hippocampal CA1 neurons.Targeted deletion of the mouse α2 nicotinic acetylcholine receptor subunit gene (Chrna2) potentiates nicotine-modulated behaviors.Nicotine uses neuron-glia communication to enhance hippocampal synaptic transmission and long-term memory.α2* Nicotinic acetylcholine receptors influence hippocampus-dependent learning and memory in adolescent mice.Nicotinic excitatory postsynaptic potentials in hippocampal CA1 interneurons are predominantly mediated by nicotinic receptors that contain α4 and β2 subunitsAcetylcholine release in mouse hippocampal CA1 preferentially activates inhibitory-selective interneurons via α4β2* nicotinic receptor activation.Nicotine persistently activates ventral tegmental area dopaminergic neurons via nicotinic acetylcholine receptors containing α4 and α6 subunits.Endogenously released ACh and exogenous nicotine differentially facilitate long-term potentiation induction in the hippocampal CA1 region of mice.Early postnatal nicotine exposure disrupts the α2* nicotinic acetylcholine receptor-mediated control of oriens-lacunosum moleculare cells during adolescence in rats.Acetylcholine release and inhibitory interneuron activity in hippocampal CA1.Olfactory Bulb Deep Short-Axon Cells Mediate Widespread Inhibition of Tufted Cell Apical Dendrites.Impaired function of α2-containing nicotinic acetylcholine receptors on oriens-lacunosum moleculare cells causes hippocampus-dependent memory impairments.
P2860
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P2860
Alpha2 nicotine receptors function as a molecular switch to continuously excite a subset of interneurons in rat hippocampal circuits.
description
2009 nî lūn-bûn
@nan
2009 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Alpha2 nicotine receptors func ...... s in rat hippocampal circuits.
@ast
Alpha2 nicotine receptors func ...... s in rat hippocampal circuits.
@en
Alpha2 nicotine receptors func ...... s in rat hippocampal circuits.
@nl
type
label
Alpha2 nicotine receptors func ...... s in rat hippocampal circuits.
@ast
Alpha2 nicotine receptors func ...... s in rat hippocampal circuits.
@en
Alpha2 nicotine receptors func ...... s in rat hippocampal circuits.
@nl
prefLabel
Alpha2 nicotine receptors func ...... s in rat hippocampal circuits.
@ast
Alpha2 nicotine receptors func ...... s in rat hippocampal circuits.
@en
Alpha2 nicotine receptors func ...... s in rat hippocampal circuits.
@nl
P2093
P2860
P1476
Alpha2 nicotine receptors func ...... s in rat hippocampal circuits.
@en
P2093
Katumi Sumikawa
Sakura Nakauchi
Yoshihiko Yamazaki
Yousheng Jia
P2860
P304
P356
10.1111/J.1460-9568.2009.06706.X
P407
P577
2009-04-01T00:00:00Z