Clonal expansion of normal-appearing human hepatocytes during chronic hepatitis B virus infection. - Wikidata - wikimedia - TriplyDB

Clonal expansion of normal-appearing human hepatocytes during chronic hepatitis B virus infection.

Clonal expansion of normal-appearing human hepatocytes during chronic hepatitis B virus infection.

http://www.wikidata.org/entity/Q34046044

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Re-evaluation of the carcinogenic significance of hepatitis B virus integration in hepatocarcinogenesis Molecular biology of hepatitis B virus infection Finding the lost treasures in exome sequencing data.The Role of cccDNA in HBV Maintenance.Immune Tolerant Chronic Hepatitis B: The Unrecognized Risks Passenger mutations as a marker of clonal cell lineages in emerging neoplasia Recurrent targeted genes of hepatitis B virus in the liver cancer genomes identified by a next-generation sequencing-based approach.Viral carcinogenesis: factors inducing DNA damage and virus integration Superinfection with woodchuck hepatitis virus strain WHVNY of livers chronically infected with strain WHV7 Somatic mutations, viral integration and epigenetic modification in the evolution of hepatitis B virus-induced hepatocellular carcinoma Distinct distribution pattern of hepatitis B virus genotype C and D in liver tissue and serum of dual genotype infected liver cirrhosis and hepatocellular carcinoma patients Clonal expansion of hepatocytes with a selective advantage occurs during all stages of chronic hepatitis B virus infection.Capacity of a natural strain of woodchuck hepatitis virus, WHVNY, to induce acute infection in naive adult woodchucks.The effects of hepatitis B virus integration into the genomes of hepatocellular carcinoma patients.Genomic Methylation Inhibits Expression of Hepatitis B Virus Envelope Protein in Transgenic Mice: A Non-Infectious Mouse Model to Study Silencing of HBV Surface Antigen Genes Accumulation of Deleterious Passenger Mutations Is Associated with the Progression of Hepatocellular Carcinoma Chronic hepatitis B: what should be the goal for new therapies?Medical virology of hepatitis B: how it began and where we are now Microarray analysis on the lncRNA expression profile in male hepatocelluar carcinoma patients with chronic hepatitis B virus infection Why do viruses cause cancer? Highlights of the first century of human tumour virology.Conceptual models for the initiation of hepatitis B virus-associated hepatocellular carcinoma.Wild type HBx and truncated HBx: Pleiotropic regulators driving sequential genetic and epigenetic steps of hepatocarcinogenesis and progression of HBV-associated neoplasms.HBV DNA Integration: Molecular Mechanisms and Clinical Implications Role of aetiology in the progression, regression, and parenchymal remodelling of liver disease: implications for liver biopsy interpretation.HBx triggers either cellular senescence or cell proliferation depending on cellular phenotype.Envelope proteins derived from naturally integrated hepatitis B virus DNA support assembly and release of infectious hepatitis delta virus particles.Smaller reduction of hepatitis B virus DNA in liver tissue than in serum in patients losing HBeAg.Initial sites of hepadnavirus integration into host genome in human hepatocytes and in the woodchuck model of hepatitis B-associated hepatocellular carcinoma.Molecular interactions between hepatitis B virus and delta virus.Detection of Hepatocyte Clones Containing Integrated Hepatitis B Virus DNA Using Inverse Nested PCR.Infection Patterns Induced in Naive Adult Woodchucks by Virions of Woodchuck Hepatitis Virus Collected during either the Acute or Chronic Phase of Infection.RNAi-based treatment of chronically infected patients and chimpanzees reveals that integrated hepatitis B virus DNA is a source of HBsAg.The growth pattern of transplanted normal and nodular hepatocytes.The Role of Infected Cell Proliferation in the Clearance of Acute HBV Infection in Humans.Global strategies are required to cure and eliminate HBV infection.Hepatitis B virus DNA integration occurs early in the viral life cycle in an in vitro infection model via NTCP-dependent uptake of enveloped virus particles.Relative Abundance of Integrant-Derived Viral RNAs in Infected Tissues Harvested from Chronic Hepatitis B Virus Carriers.Sequence analysis of integrated hepatitis B virus DNA during HBeAg-seroconversion

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P2860

Clonal expansion of normal-appearing human hepatocytes during chronic hepatitis B virus infection.

http://www.wikidata.org/entity/Q34046044

description

2010 nî lūn-bûn

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2010 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հունիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Clonal expansion of normal-app ...... c hepatitis B virus infection.

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Clonal expansion of normal-app ...... c hepatitis B virus infection.

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Clonal expansion of normal-app ...... c hepatitis B virus infection.

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type

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Clonal expansion of normal-app ...... c hepatitis B virus infection.

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Clonal expansion of normal-app ...... c hepatitis B virus infection.

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Clonal expansion of normal-app ...... c hepatitis B virus infection.

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Clonal expansion of normal-app ...... c hepatitis B virus infection.

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Clonal expansion of normal-app ...... c hepatitis B virus infection.

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Clonal expansion of normal-app ...... c hepatitis B virus infection.

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Journal of Virology

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Clonal expansion of normal-app ...... c hepatitis B virus infection.

@en

P2093

Carol E Aldrich

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Chen Liu

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Matthew M Yeh

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Samuel Litwin

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William S Mason

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P2860

Histological grading and staging of chronic hepatitis

Double-stranded linear duck hepatitis B virus (DHBV) stably integrates at a higher frequency than wild-type DHBV in LMH chicken hepatoma cells.

Clonal expansion of hepatocytes during chronic woodchuck hepatitis virus infection.

Integration of hepadnavirus DNA in infected liver: evidence for a linear precursor.

Nucleotide sequence of the hepatitis B virus genome (subtype ayw) cloned in E. coli.

The liver of woodchucks chronically infected with the woodchuck hepatitis virus contains foci of virus core antigen-negative hepatocytes with both altered and normal morphology.

Liver regeneration and repair: hepatocytes, progenitor cells, and stem cells.

Hepatocyte turnover during resolution of a transient hepadnaviral infection.

Natural history of chronic hepatitis B virus infection: what we knew in 1981 and what we know in 2005.

The degrees of hepatocyte nuclear but not cytoplasmic expression of hepatitis B core antigen reflect the level of viral replication in chronic hepatitis B virus infection.

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8308-8315

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scholarly article

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10.1128/JVI.00833-10

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16

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84

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2916518

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