BET Bromodomain Inhibition Promotes Anti-tumor Immunity by Suppressing PD-L1 Expression.
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Epigenetic Strategies to Boost Cancer Immunotherapies.BET-Bromodomain Inhibitors Engage the Host Immune System and Regulate Expression of the Immune Checkpoint Ligand PD-L1BET inhibitors: a novel epigenetic approach.Amplification of the NSD3-BRD4-CHD8 pathway in pelvic high-grade serous carcinomas of tubo-ovarian and endometrial origin.Targeting the PD-L1/DNMT1 axis in acquired resistance to sorafenib in human hepatocellular carcinomaThe cancer epigenome: Concepts, challenges, and therapeutic opportunities.Marked for death: targeting epigenetic changes in cancer.BET Bromodomain Proteins as Cancer Therapeutic Targets.MYC: Master Regulator of Immune Privilege.Natural Killer Cell Response to Chemotherapy-Stressed Cancer Cells: Role in Tumor Immunosurveillance.BET inhibitors as novel therapeutic agents in breast cancer.BETting on combination to overcome PARPi resistance.Ovarian cancer: how can resistance to chemotherapy be tackled?Chromatin dependencies in cancer and inflammation.Improvement of Antitumor Therapies Based on Vaccines and Immune-Checkpoint Inhibitors by Counteracting Tumor-Immunostimulation.A cancer vaccine-mediated postoperative immunotherapy for recurrent and metastatic tumors.Modulating PD-L1 expression in multiple myeloma: an alternative strategy to target the PD-1/PD-L1 pathway.BET proteins in abnormal metabolism, inflammation, and the breast cancer microenvironment.Bromodomain inhibition exerts its therapeutic potential in malignant pleural mesothelioma by promoting immunogenic cell death and changing the tumor immune-environment.The MYC oncogene is a global regulator of the immune response.Immunotherapy With Human Gamma Delta T Cells-Synergistic Potential of Epigenetic Drugs?Epigenetic control of macrophage polarization: implications for targeting tumor-associated macrophages.Modeling combination therapy for breast cancer with BET and immune checkpoint inhibitorsIL-6 augments IL-4-induced polarization of primary human macrophages through synergy of STAT3, STAT6 and BATF transcription factorsEmerging role of precision medicine in biliary tract cancersGreen Tea Catechin Is an Alternative Immune Checkpoint Inhibitor that Inhibits PD-L1 Expression and Lung Tumor GrowthInterplay between interferon regulatory factor 1 and BRD4 in the regulation of PD-L1 in pancreatic stellate cellsTargeting the Microenvironment in High Grade Serous Ovarian Cancer
P2860
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P2860
BET Bromodomain Inhibition Promotes Anti-tumor Immunity by Suppressing PD-L1 Expression.
description
2016 nî lūn-bûn
@nan
2016 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2016 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
name
BET Bromodomain Inhibition Promotes Anti-tumor Immunity by Suppressing PD-L1 Expression.
@ast
BET Bromodomain Inhibition Promotes Anti-tumor Immunity by Suppressing PD-L1 Expression.
@en
BET Bromodomain Inhibition Promotes Anti-tumor Immunity by Suppressing PD-L1 Expression.
@nl
type
label
BET Bromodomain Inhibition Promotes Anti-tumor Immunity by Suppressing PD-L1 Expression.
@ast
BET Bromodomain Inhibition Promotes Anti-tumor Immunity by Suppressing PD-L1 Expression.
@en
BET Bromodomain Inhibition Promotes Anti-tumor Immunity by Suppressing PD-L1 Expression.
@nl
prefLabel
BET Bromodomain Inhibition Promotes Anti-tumor Immunity by Suppressing PD-L1 Expression.
@ast
BET Bromodomain Inhibition Promotes Anti-tumor Immunity by Suppressing PD-L1 Expression.
@en
BET Bromodomain Inhibition Promotes Anti-tumor Immunity by Suppressing PD-L1 Expression.
@nl
P2093
P2860
P1433
P1476
BET Bromodomain Inhibition Promotes Anti-tumor Immunity by Suppressing PD-L1 Expression
@en
P2093
Andrew V Kossenkov
Fee Bengsch
Hengrui Zhu
James E Bradner
Jose R Conejo-Garcia
Melanie R Rutkowski
Michael J Allegrezza
Nikolaos Svoronos
Rugang Zhang
Yuhki Yokoyama
P2860
P304
P356
10.1016/J.CELREP.2016.08.032
P577
2016-09-01T00:00:00Z