about
Contribution of anaerobic energy expenditure to whole body thermogenesis.Warburg Effect - a Consequence or the Cause of Carcinogenesis?The role of VDAC in cell death: friend or foe?Modelling acidosis and the cell cycle in multicellular tumour spheroids.A pairwise chemical genetic screen identifies new inhibitors of glucose transport.Acidity generated by the tumor microenvironment drives local invasionTumor acidity as evolutionary spite.Evolution of Tumor Metabolism might Reflect Carcinogenesis as a Reverse Evolution process (Dismantling of Multicellularity).Glycolysis, tumor metabolism, cancer growth and dissemination. A new pH-based etiopathogenic perspective and therapeutic approach to an old cancer questionMetabolic signature identifies novel targets for drug resistance in multiple myeloma.Homeostasis and the glycogen shunt explains aerobic ethanol production in yeast.Hypoxia Strongly Affects Mitochondrial Ribosomal Proteins and Translocases, as Shown by Quantitative Proteomics of HeLa Cells.Prognostic Value of Serum Lactate Dehydrogenase in Renal Cell Carcinoma: A Systematic Review and Meta-Analysis.Mechanisms of appearance of the Pasteur effect in Saccharomyces cerevisiae: inactivation of sugar transport systems.The metabolic switch and its regulation in cancer cells.Regulation of cellular gas exchange, oxygen sensing, and metabolic control.Extracellular acidity, a "reappreciated" trait of tumor environment driving malignancy: perspectives in diagnosis and therapy.Dose painting based on tumor uptake of Cu-ATSM and FDG: a comparative study.Exogenous normal mammary epithelial mitochondria suppress glycolytic metabolism and glucose uptake of human breast cancer cells.Low oxygen tension enhances hepatitis C virus replication.Glucosamine resistance in yeast. I. A preliminary genetic analysis.Press-pulse: a novel therapeutic strategy for the metabolic management of cancer.Short-term starvation is a strategy to unravel the cellular capacity of oxidizing specific exogenous/endogenous substrates in mitochondria.The phosphatidylinositol 3,5-bisphosphate (PI(3,5)P2)-dependent Tup1 conversion (PIPTC) regulates metabolic reprogramming from glycolysis to gluconeogenesis.Did we get pasteur, warburg, and crabtree on a right note?Lactate metabolism: historical context, prior misinterpretations, and current understanding.In Silico Evaluation of Variable pH on the Binding of Epidermal Growth Factor Receptor Ectodomain to its Ligand Through Molecular Dynamics Simulation in Tumors.
P2860
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P2860
description
1974 nî lūn-bûn
@nan
1974 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
1974 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
1974年の論文
@ja
1974年論文
@yue
1974年論文
@zh-hant
1974年論文
@zh-hk
1974年論文
@zh-mo
1974年論文
@zh-tw
1974年论文
@wuu
name
History of the Pasteur effect and its pathobiology.
@ast
History of the Pasteur effect and its pathobiology.
@en
History of the Pasteur effect and its pathobiology.
@nl
type
label
History of the Pasteur effect and its pathobiology.
@ast
History of the Pasteur effect and its pathobiology.
@en
History of the Pasteur effect and its pathobiology.
@nl
prefLabel
History of the Pasteur effect and its pathobiology.
@ast
History of the Pasteur effect and its pathobiology.
@en
History of the Pasteur effect and its pathobiology.
@nl
P356
P1476
History of the Pasteur effect and its pathobiology.
@en
P2093
P2888
P356
10.1007/BF01874168
P577
1974-11-01T00:00:00Z
P6179
1024589490