Monocyte chemoattractant protein-1-deficiency impairs the expression of IL-6, IL-1β and G-CSF after transient focal ischemia in mice - Wikidata - wikimedia - TriplyDB

Monocyte chemoattractant protein-1-deficiency impairs the expression of IL-6, IL-1β and G-CSF after transient focal ischemia in mice

Monocyte chemoattractant protein-1-deficiency impairs the expression of IL-6, IL-1β and G-CSF after transient focal ischemia in mice

http://www.wikidata.org/entity/Q34059585

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Rational modulation of the innate immune system for neuroprotection in ischemic stroke Blood-brain barrier integrity and glial support: mechanisms that can be targeted for novel therapeutic approaches in stroke Update on Inflammatory Biomarkers and Treatments in Ischemic Stroke Immunological profile of silent brain infarction and lacunar stroke Delayed decompression exacerbates ischemia-reperfusion injury in cervical compressive myelopathy.Effects of wind-dispelling drugs and deficiency-nourishing drugs of Houshiheisan compound prescription on astrocyte activation and inflammatory factor expression in the corpus striatum of cerebral ischemia rats.Monocyte chemotactic protein-1 as a potential biomarker for early anti-thrombotic therapy after ischemic stroke.Intrapulmonary G-CSF rescues neutrophil recruitment to the lung and neutrophil release to blood in Gram-negative bacterial infection in MCP-1-/- mice.Method parameters' impact on mortality and variability in mouse stroke experiments: a meta-analysis.Human umbilical cord blood mesenchymal stem cell transplantation suppresses inflammatory responses and neuronal apoptosis during early stage of focal cerebral ischemia in rabbits.Multiplex immunoassay characterization and species comparison of inflammation in acute and non-acute ischemic infarcts in human and mouse brain tissue.miRNAs in acute myeloid leukemia.Lack of collagen XV is protective after ischemic stroke in mice.Post-ischemic inflammation in the brain.Help-me signaling: Non-cell autonomous mechanisms of neuroprotection and neurorecovery.Propofol Attenuates Lipopolysaccharide-Induced Monocyte Chemoattractant Protein-1 Production Through Enhancing apoM and foxa2 Expression in HepG2 Cells.The pivotal role of astrocytes in an in vitro stroke model of the blood-brain barrier.Ischemia-induced endothelial cell swelling and mitochondrial dysfunction are attenuated by cinnamtannin D1, green tea extract, and resveratrol in vitro.miR-155 is associated with the leukemogenic potential of the class IV granulocyte colony-stimulating factor receptor in CD34⁺ progenitor cells.The intrinsic microglial clock system regulates interleukin-6 expression.Hypothermia Reduces but Hyperthermia Augments T Cell-Derived Release of Interleukin-17 and Granzyme B that Mediate Neuronal Cell Death.Granulocyte-colony-stimulating factor (G-CSF) signaling in spinal microglia drives visceral sensitization following colitis.Upregulation of insulin-like growth factor and interleukin 1β occurs in neurons but not in glial cells in the cochlear nucleus following cochlear ablation.

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Monocyte chemoattractant protein-1-deficiency impairs the expression of IL-6, IL-1β and G-CSF after transient focal ischemia in mice

http://www.wikidata.org/entity/Q34059585

description

2011 nî lūn-bûn

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2011 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2011 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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Monocyte chemoattractant prote ...... ansient focal ischemia in mice

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Monocyte chemoattractant prote ...... ansient focal ischemia in mice

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Monocyte chemoattractant prote ...... ansient focal ischemia in mice

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type

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Monocyte chemoattractant prote ...... ansient focal ischemia in mice

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Monocyte chemoattractant prote ...... ansient focal ischemia in mice

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Monocyte chemoattractant prote ...... ansient focal ischemia in mice

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Monocyte chemoattractant prote ...... ansient focal ischemia in mice

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Monocyte chemoattractant prote ...... ansient focal ischemia in mice

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Monocyte chemoattractant prote ...... ansient focal ischemia in mice

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Burkhard Gess

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E Bernd Ringelstein

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Jan-Kolja Strecker

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Jens Minnerup

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Matthias Schilling

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Wolf-Rüdiger Schäbitz

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P2860

The hematopoietic factor G-CSF is a neuronal ligand that counteracts programmed cell death and drives neurogenesis

A new mathematical model for relative quantification in real-time RT-PCR

Src-dependent STAT-3-mediated expression of monocyte chemoattractant protein-1 is required for 15(S)-hydroxyeicosatetraenoic acid-induced vascular smooth muscle cell migration

Role of JAK/STAT pathway in IL-6-induced activation of vascular smooth muscle cells

Relative expression software tool (REST©) for group-wise comparison and statistical analysis of relative expression results in real-time PCR

G-CSF suppresses edema formation and reduces interleukin-1beta expression after cerebral ischemia in mice.

A reproducible model of middle cerebral artery occlusion in mice: hemodynamic, biochemical, and magnetic resonance imaging.

G-CSF administration is neuroprotective following transient cerebral ischemia even in the absence of a functional NOS-2 gene

The interaction between tumor necrosis factor-like weak inducer of apoptosis and its receptor fibroblast growth factor-inducible 14 promotes the recruitment of neutrophils into the ischemic brain.

Astrocyte activation and reactive gliosis.

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