Permissive proteolytic activity for visual cortical plasticity. - Wikidata - wikimedia - TriplyDB

Permissive proteolytic activity for visual cortical plasticity.

Permissive proteolytic activity for visual cortical plasticity.

http://www.wikidata.org/entity/Q34065299

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Methodological factors influencing measurement and processing of plasma reelin in humans Activity-regulated genes as mediators of neural circuit plasticity Removing brakes on adult brain plasticity: from molecular to behavioral interventions Physiological and pathological roles of tissue plasminogen activator and its inhibitor neuroserpin in the nervous system Microglial interactions with synapses are modulated by visual experience Microglial P2Y12 is necessary for synaptic plasticity in mouse visual cortex.Key role of tissue plasminogen activator in neurovascular coupling Brain plasticity and disease: a matter of inhibition.Motility of dendritic spines in visual cortex in vivo: changes during the critical period and effects of visual deprivation.gamma-Aminbuturic acid A receptor mitigates homocysteine-induced endothelial cell permeability.GABAergic inhibition in visual cortical plasticity The microRNA bantam regulates a developmental transition in epithelial cells that restricts sensory dendrite growth.Molecular insights and therapeutic targets for blood-brain barrier disruption in ischemic stroke: critical role of matrix metalloproteinases and tissue-type plasminogen activator Demonstration of functional coupling between gamma -aminobutyric acid (GABA) synthesis and vesicular GABA transport into synaptic vesicles Neuroserpin protects neurons from ischemia-induced plasmin-mediated cell death independently of tissue-type plasminogen activator inhibition.Postnatal expression profile of OBCAM implies its involvement in visual cortex development and plasticity.Separable features of visual cortical plasticity revealed by N-methyl-D-aspartate receptor 2A signaling.Endogenous tissue plasminogen activator mediates bone marrow stromal cell-induced neurite remodeling after stroke in mice.AAV-mediated overexpression of neuroserpin in the hippocampus decreases PSD-95 expression but does not affect hippocampal-dependent learning and memory.Is reelin the answer to synapse elimination at the neuromuscular junction?Neurobiology of schizophrenia onset.Perineuronal nets and schizophrenia: the importance of neuronal coatings.Inflammatory Animal Model for Parkinson's Disease: The Intranigral Injection of LPS Induced the Inflammatory Process along with the Selective Degeneration of Nigrostriatal Dopaminergic Neurons.Tissue-type plasminogen activator-mediated shedding of astrocytic low-density lipoprotein receptor-related protein increases the permeability of the neurovascular unit.Progressive maturation of silent synapses governs the duration of a critical period.Treatment of TBI with collagen scaffolds and human marrow stromal cells increases the expression of tissue plasminogen activator.Molecular genetics of activity-dependent structural changes at the synapse.Unmasking Proteolytic Activity for Adult Visual Cortex Plasticity by the Removal of Lynx1 Proteolytic regulation of synaptic plasticity in the mouse primary visual cortex: analysis of matrix metalloproteinase 9 deficient mice Extracellular proteolysis in structural and functional plasticity of mossy fiber synapses in hippocampus Early pharmacological treatment of autism: a rationale for developmental treatment.Tissue-type plasminogen activator induces opening of the blood-brain barrier via the LDL receptor-related protein.Developmental pattern of perineuronal nets in the human prefrontal cortex and their deficit in schizophrenia.Bidirectional synaptic mechanisms of ocular dominance plasticity in visual cortex Molecular mechanisms of experience-dependent plasticity in visual cortex.Extracellular matrix in plasticity and epileptogenesis.Synaptic plasticity-associated proteases and protease inhibitors in the brain linked to the processing of extracellular matrix and cell adhesion molecules.The role of GABAergic inhibition in ocular dominance plasticity Development and plasticity of the primary visual cortex.Extracellular matrix control of dendritic spine and synapse structure and plasticity in adulthood.

P2860

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P2860

Permissive proteolytic activity for visual cortical plasticity.

http://www.wikidata.org/entity/Q34065299

description

2002 nî lūn-bûn

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2002 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2002 թվականի մայիսին հրատարակված գիտական հոդված

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2002年の論文

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年學術文章

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name

Permissive proteolytic activity for visual cortical plasticity.

@ast

Permissive proteolytic activity for visual cortical plasticity.

@en

Permissive proteolytic activity for visual cortical plasticity.

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type

label

Permissive proteolytic activity for visual cortical plasticity.

@ast

Permissive proteolytic activity for visual cortical plasticity.

@en

Permissive proteolytic activity for visual cortical plasticity.

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prefLabel

Permissive proteolytic activity for visual cortical plasticity.

@ast

Permissive proteolytic activity for visual cortical plasticity.

@en

Permissive proteolytic activity for visual cortical plasticity.

@nl

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Permissive proteolytic activity for visual cortical plasticity.

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P2093

Nobuko Mataga

http://www.w3.org/2001/XMLSchema#string

Nobuo Nagai

http://www.w3.org/2001/XMLSchema#string

Takao K Hensch

http://www.w3.org/2001/XMLSchema#string

P2860

Mice lacking the 65 kDa isoform of glutamic acid decarboxylase (GAD65) maintain normal levels of GAD67 and GABA in their brains but are susceptible to seizures

Rapid anatomical plasticity of horizontal connections in the developing visual cortex

The proteolytic activity of tissue-plasminogen activator enhances NMDA receptor-mediated signaling.

Cyclic AMP-dependent protein kinase mediates ocular dominance shifts in cat visual cortex.

Neurotrophins and synaptic plasticity.

Enhanced hippocampal long-term potentiation and learning by increased neuronal expression of tissue-type plasminogen activator in transgenic mice.

Experience-dependent plasticity without long-term depression by type 2 metabotropic glutamate receptors in developing visual cortex.

Long-term but not short-term plasticity at mossy fiber synapses is impaired in neural cell adhesion molecule-deficient mice.

Neuronal migration is retarded in mice lacking the tissue plasminogen activator gene.

Morphology of single geniculocortical afferents and functional recovery of the visual cortex after reverse monocular deprivation in the kitten

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7717-7721

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10.1073/PNAS.102088899

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11

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99

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124331

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