Beta-adrenergic stimulation reverses the I Kr-I Ks dominant pattern during cardiac action potential - Wikidata - wikimedia - TriplyDB

Beta-adrenergic stimulation reverses the I Kr-I Ks dominant pattern during cardiac action potential

Beta-adrenergic stimulation reverses the I Kr-I Ks dominant pattern during cardiac action potential

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Quantitative analysis of the Ca2+ -dependent regulation of delayed rectifier K+ current IKs in rabbit ventricular myocytes.Potassium currents in the heart: functional roles in repolarization, arrhythmia and therapeutics.Potassium channels in the heart: structure, function and regulation.β-adrenergic stimulation augments transmural dispersion of repolarization via modulation of delayed rectifier currents IKs and IKr in the human ventricle.Differential roles of two delayed rectifier potassium currents in regulation of ventricular action potential duration and arrhythmia susceptibility.Altered Repolarization Reserve in Failing Rabbit Ventricular Myocytes: Calcium and β-Adrenergic Effects on Delayed- and Inward-Rectifier Potassium Currents.

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Beta-adrenergic stimulation reverses the I Kr-I Ks dominant pattern during cardiac action potential

http://www.wikidata.org/entity/Q34067125

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2014 nî lūn-bûn

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2014 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2014 թվականի փետրվարին հրատարակված գիտական հոդված

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2014年の論文

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2014年学术文章

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2014年学术文章

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Beta-adrenergic stimulation re ...... uring cardiac action potential

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Beta-adrenergic stimulation re ...... uring cardiac action potential

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Beta-adrenergic stimulation re ...... uring cardiac action potential

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Beta-adrenergic stimulation re ...... uring cardiac action potential

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Pfluegers Archiv

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Beta-adrenergic stimulation re ...... uring cardiac action potential

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Balazs Horvath

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Leighton T Izu

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Shaden Khabbaz

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Tamas Banyasz

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Ye Chen-Izu

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Zhong Jian

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P2860

Requirement of a macromolecular signaling complex for beta adrenergic receptor modulation of the KCNQ1-KCNE1 potassium channel

Rate dependency of delayed rectifier currents during the guinea-pig ventricular action potential

Pharmacological block of the slow component of the outward delayed rectifier current (I(Ks)) fails to lengthen rabbit ventricular muscle QT(c) and action potential duration

A novel action of isoproterenol to inactivate a cardiac K+ current is not blocked by beta and alpha adrenergic blockers.

Sequential dissection of multiple ionic currents in single cardiac myocytes under action potential-clamp

Adrenergic regulation of the rapid component of the cardiac delayed rectifier potassium current, I(Kr), and the underlying hERG ion channel.

Calcium/calmodulin-dependent kinase II regulation of cardiac ion channels.

Receptor stimulation causes slow inhibition of IRK1 inwardly rectifying K+ channels by direct protein kinase A-mediated phosphorylation.

Beta-adrenergic receptor signaling in the heart: role of CaMKII.

Effects of β-adrenoceptor stimulation on delayed rectifier K(+) currents in canine ventricular cardiomyocytes

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2067-2076

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10.1007/S00424-014-1465-7

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11

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466

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1031118506

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