Early phagocytosis of glucose-6-phosphate dehydrogenase (G6PD)-deficient erythrocytes parasitized by Plasmodium falciparum may explain malaria protection in G6PD deficiency.
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Biochemical and immunological mechanisms by which sickle cell trait protects against malariaMultiplicity of Plasmodium falciparum infection in asymptomatic children in Senegal: relation to transmission, age and erythrocyte variantsSickle cell anaemia and malariaThe impact of malaria parasitism: from corpuscles to communitiesData mining of the transcriptome of Plasmodium falciparum: the pentose phosphate pathway and ancillary processes.Major Histocompatibility Complex and Malaria: Focus on Plasmodium vivax InfectionThe influence of host genetics on erythrocytes and malaria infection: is there therapeutic potential?Role of the lipoperoxidation product 4-hydroxynonenal in the pathogenesis of severe malaria anemia and malaria immunodepressionTriggers, inhibitors, mechanisms, and significance of eryptosis: the suicidal erythrocyte deathDiscovery of a Plasmodium falciparum glucose-6-phosphate dehydrogenase 6-phosphogluconolactonase inhibitor (R,Z)-N-((1-ethylpyrrolidin-2-yl)methyl)-2-(2-fluorobenzylidene)-3-oxo-3,4-dihydro-2H-benzo[b][1,4]thiazine-6-carboxamide (ML276) that reducesRedox regulation of mitochondrial functionInherited glutathione reductase deficiency and Plasmodium falciparum malaria--a case studyA simplified, sensitive phagocytic assay for malaria cultures facilitated by flow cytometry of differentially-stained cell populationsX-linked G6PD deficiency protects hemizygous males but not heterozygous females against severe malariaMolecular Analysis of Glucose-6-Phosphate Dehydrogenase Gene Mutations in Bangladeshi Individuals.Glucose-6-phosphate dehydrogenase deficiency and the risk of malaria and other diseases in children in Kenya: a case-control and a cohort study.Oxidative risk for atherothrombotic cardiovascular disease.In vitro induction of erythrocyte phosphatidylserine translocation by the natural naphthoquinone shikonin.Irreversible AE1 tyrosine phosphorylation leads to membrane vesiculation in G6PD deficient red cellsPrevalence of glucose-6-phosphate dehydrogenase deficiency and its association with Plasmodium falciparum infection among children in Iganga distric in Uganda.Beneficial effect of aurothiomalate on murine malaria.16alpha-bromoepiandrosterone, an antimalarial analogue of the hormone dehydroepiandrosterone, enhances phagocytosis of ring stage parasitized erythrocytes: a novel mechanism for antimalarial activity.Allelic heterogeneity of G6PD deficiency in West Africa and severe malaria susceptibility.ABO blood groups influence macrophage-mediated phagocytosis of Plasmodium falciparum-infected erythrocytesTriggering of programmed erythrocyte death by alantolactone.Hematological predictors of increased severe anemia in Kenyan children coinfected with Plasmodium falciparum and HIV-1.Eosin B as a novel antimalarial agent for drug-resistant Plasmodium falciparum.Anti-malarial effect of gum arabic.African glucose-6-phosphate dehydrogenase alleles associated with protection from severe malaria in heterozygous females in TanzaniaAntimalarial NADPH-Consuming Redox-Cyclers As Superior Glucose-6-Phosphate Dehydrogenase Deficiency Copycats.Effect of red blood cell variants on childhood malaria in Mali: a prospective cohort study.Plasmodium vivax trophozoite-stage proteomesEnhanced eryptosis following gramicidin exposure.Induction of suicidal erythrocyte death by nelfinavirThe hydration state of human red blood cells and their susceptibility to invasion by Plasmodium falciparumTriggering of Erythrocyte Death by Triparanol.The Redox Cycler Plasmodione Is a Fast-Acting Antimalarial Lead Compound with Pronounced Activity against Sexual and Early Asexual Blood-Stage Parasites.New antimalarial indolone-N-oxides, generating radical species, destabilize the host cell membrane at early stages of Plasmodium falciparum growth: role of band 3 tyrosine phosphorylation.Synthesis and biological evaluation of 1,4-naphthoquinones and quinoline-5,8-diones as antimalarial and schistosomicidal agents.Heterogeneous alleles comprising G6PD deficiency trait in West Africa exert contrasting effects on two major clinical presentations of severe malaria.
P2860
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P2860
Early phagocytosis of glucose-6-phosphate dehydrogenase (G6PD)-deficient erythrocytes parasitized by Plasmodium falciparum may explain malaria protection in G6PD deficiency.
description
1998 nî lūn-bûn
@nan
1998 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
Early phagocytosis of glucose- ...... protection in G6PD deficiency.
@ast
Early phagocytosis of glucose- ...... protection in G6PD deficiency.
@en
Early phagocytosis of glucose-6-phosphate dehydrogenase
@nl
type
label
Early phagocytosis of glucose- ...... protection in G6PD deficiency.
@ast
Early phagocytosis of glucose- ...... protection in G6PD deficiency.
@en
Early phagocytosis of glucose-6-phosphate dehydrogenase
@nl
prefLabel
Early phagocytosis of glucose- ...... protection in G6PD deficiency.
@ast
Early phagocytosis of glucose- ...... protection in G6PD deficiency.
@en
Early phagocytosis of glucose-6-phosphate dehydrogenase
@nl
P2093
P921
P1433
P1476
Early phagocytosis of glucose- ...... protection in G6PD deficiency.
@en
P2093
Cappadoro M
Giribaldi G
Luzzatto L
P304
P407
P577
1998-10-01T00:00:00Z