Increased tumors but uncompromised fertility in the female descendants of mice exposed developmentally to diethylstilbestrol. - Wikidata - wikimedia - TriplyDB

Increased tumors but uncompromised fertility in the female descendants of mice exposed developmentally to diethylstilbestrol.

Increased tumors but uncompromised fertility in the female descendants of mice exposed developmentally to diethylstilbestrol.

http://www.wikidata.org/entity/Q34067657

about

Long-term effects of environmental endocrine disruptors on reproductive physiology and behavior Endocrine-disrupting chemicals: an Endocrine Society scientific statement Evaluating the effects of endocrine disruptors on endocrine function during development Exposure assessment for endocrine disruptors: some considerations in the design of studies.Reproductive consequences of developmental phytoestrogen exposure Proceedings of the Summit on Environmental Challenges to Reproductive Health and Fertility: executive summary Epigenetic effects of endocrine-disrupting chemicals on female reproduction: an ovarian perspective Effects of perinatal polychlorinated biphenyls on adult female rat reproduction: development, reproductive physiology, and second generational effects Birth outcomes and background exposures to select elements, the Longitudinal Investigation of Fertility and the Environment (LIFE)The bisphenol A experience: a primer for the analysis of environmental effects on mammalian reproduction Epigenetic mechanisms in the actions of endocrine-disrupting chemicals: gonadal effects and role in female reproduction Transgenerational epigenetic programming of the brain transcriptome and anxiety behavior Environmental exposures and gene regulation in disease etiology Minireview: transgenerational epigenetic inheritance: focus on endocrine disrupting compounds Assessment and molecular actions of endocrine-disrupting chemicals that interfere with estrogen receptor pathways.Expression of estrogen receptor beta is developmentally regulated in reproductive tissues of male and female mice.Viewpoint: Policy requirements for protecting wildlife from endocrine disruptors.Birth defects in the sons and daughters of women who were exposed in utero to diethylstilbestrol (DES).Molecular and environmental causes of cancer.Proliferative lesions and reproductive tract tumors in male descendants of mice exposed developmentally to diethylstilbestrol.Critical windows of exposure for children's health: cancer in human epidemiological studies and neoplasms in experimental animal models Hypospadias: a transgenerational effect of diethylstilbestrol?Exposure to diethylstilbestrol during sensitive life stages: a legacy of heritable health effects.Menstrual and reproductive characteristics of women whose mothers were exposed in utero to diethylstilbestrol (DES).Endocrine disruptor vinclozolin induced epigenetic transgenerational adult-onset disease.Epigenetic transgenerational actions of vinclozolin on the development of disease and cancer.Differences between children and adults: implications for risk assessment at California EPA.Polychlorinated biphenyl (PCB) exposure in mothers and time to pregnancy in daughters.Developmental exposure to environmental endocrine disruptors: consequences within the ovary and on female reproductive function Hsp90 and environmental impacts on epigenetic states: a model for the trans-generational effects of diethylstibesterol on uterine development and cancer.Transgenerational epigenetic programming of the embryonic testis transcriptome Intersection of nuclear receptors and the proteasome on the epigenetic landscape.Epigenetic transgenerational toxicology and germ cell disease.Epigenetic perspective on the developmental effects of bisphenol A.Key developments in endocrine disrupter research and human health.Oxidative stress and antioxidants: exposure and impact on female fertility Transgenerational neuroendocrine disruption of reproduction.Environmental signaling: from environmental estrogens to endocrine-disrupting chemicals and beyond.Neonatal exposure to p-tert-octylphenol causes abnormal expression of estrogen receptor alpha and subsequent alteration of cell proliferating activity in the developing Donryu rat uterus.Effects of neonatal exposure to diethylstilbestrol, tamoxifen, and toremifene on the BALB/c mouse mammary gland.

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P2860

Increased tumors but uncompromised fertility in the female descendants of mice exposed developmentally to diethylstilbestrol.

http://www.wikidata.org/entity/Q34067657

description

1998 nî lūn-bûn

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1998 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

1998 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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1998年の論文

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1998年論文

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1998年論文

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1998年論文

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1998年論文

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1998年論文

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1998年论文

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name

Increased tumors but uncomprom ...... entally to diethylstilbestrol.

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Increased tumors but uncomprom ...... entally to diethylstilbestrol.

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Increased tumors but uncomprom ...... entally to diethylstilbestrol.

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type

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Increased tumors but uncomprom ...... entally to diethylstilbestrol.

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Increased tumors but uncomprom ...... entally to diethylstilbestrol.

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Increased tumors but uncomprom ...... entally to diethylstilbestrol.

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Increased tumors but uncomprom ...... entally to diethylstilbestrol.

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Increased tumors but uncomprom ...... entally to diethylstilbestrol.

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Increased tumors but uncomprom ...... entally to diethylstilbestrol.

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Bullock BC

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