Inducible response required for repair of low-dose radiation damage in human fibroblasts. - Wikidata - wikimedia - TriplyDB

Inducible response required for repair of low-dose radiation damage in human fibroblasts.

Inducible response required for repair of low-dose radiation damage in human fibroblasts.

http://www.wikidata.org/entity/Q34069451

about

Physiologic brain activity causes DNA double-strand breaks in neurons, with exacerbation by amyloid-β Opposite roles for p38MAPK-driven responses and reactive oxygen species in the persistence and resolution of radiation-induced genomic instability.Recruitment of HRDC domain of WRN and BLM to the sites of DNA damage induced by mitomycin C and methyl methanesulfonate.Increased apoptosis and DNA double-strand breaks in the embryonic mouse brain in response to very low-dose X-rays but not 50 Hz magnetic fields.DNA double-strand break induction in Ku80-deficient CHO cells following boron neutron capture reaction Nonlinear ionizing radiation-induced changes in eye lens cell proliferation, cyclin D1 expression and lens shape.Modeling DNA double-strand break repair kinetics as an epiregulated cell-community-wide (epicellcom) response to radiation stress Cell to Cell Variability of Radiation-Induced Foci: Relation between Observed Damage and Energy Deposition Role of the translationally controlled tumor protein in DNA damage sensing and repair H2AX phosphorylation at the sites of DNA double-strand breaks in cultivated mammalian cells and tissues.RNA-Binding Protein FXR1 Regulates p21 and TERC RNA to Bypass p53-Mediated Cellular Senescence in OSCC.Direct measurement of the 3-dimensional DNA lesion distribution induced by energetic charged particles in a mouse model tissue.Activation of homologous recombination DNA repair in human skin fibroblasts continuously exposed to X-ray radiation Low doses of X-rays induce prolonged and ATM-independent persistence of γH2AX foci in human gingival mesenchymal stem cells.Effects of Chronic Low-Dose Radiation on Human Neural Progenitor Cells.Chemoprevention by N-acetylcysteine of low-dose CT-induced murine lung tumorigenesis.The shape of the radiation dose response for DNA double-strand break induction and repair.The role of dose rate in radiation cancer risk: evaluating the effect of dose rate at the molecular, cellular and tissue levels using key events in critical pathways following exposure to low LET radiation Distinct biological effects of low-dose radiation on normal and cancerous human lung cells are mediated by ATM signaling.DNA damage-associated biomarkers in studying individual sensitivity to low-dose radiation from cardiovascular imaging.Does the number of irradiated cells influence the spatial distribution of bystander effects?Impact of dose-rate on the low-dose hyper-radiosensitivity and induced radioresistance (HRS/IRR) response.Sister chromatid exchanges occur in G2-irradiated cells.Estimation of low-dose radiation-responsive proteins in the absence of genomic instability in normal human fibroblast cells.Dose-rate effects in radiation biology and radiation protection.Nek1 Regulates Rad54 to Orchestrate Homologous Recombination and Replication Fork Stability.Prostate cancer treated with brachytherapy; an exploratory study of dose-dependent biomarkers and quality of life.Changes in the number of double-strand DNA breaks in Chinese hamster V79 cells exposed to γ-radiation with different dose rates Gamma-irradiated quiescent cells repair directly induced double-strand breaks but accumulate persistent double-strand breaks during subsequent DNA replication.Differential Molecular Stress Responses to Low Compared to High Doses of Ionizing Radiation in Normal Human Fibroblasts.Residual γH2AX foci induced by low dose x-ray radiation in bone marrow mesenchymal stem cells do not cause accelerated senescence in the progeny of irradiated cells.Global transcriptome profile reveals abundance of DNA damage response and repair genes in individuals from high level natural radiation areas of Kerala coast.Dose and dose-rate effects of ionizing radiation: a discussion in the light of radiological protection.Genomic Destabilization Triggered by Replication Stress during Senescence.Induction of somatic mutations by low-dose X-rays: the challenge in recognizing radiation-induced events.Radiation-induced conformational changes in chromatin structure in resting human peripheral blood mononuclear cells.Estimation of the effects of medical diagnostic radiation exposure based on DNA damage.

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Inducible response required for repair of low-dose radiation damage in human fibroblasts.

http://www.wikidata.org/entity/Q34069451

description

2010 nî lūn-bûn

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2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2010 թվականի հուլիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年论文

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Inducible response required for repair of low-dose radiation damage in human fibroblasts.

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Inducible response required for repair of low-dose radiation damage in human fibroblasts.

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Inducible response required for repair of low-dose radiation damage in human fibroblasts.

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Inducible response required for repair of low-dose radiation damage in human fibroblasts.

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Inducible response required for repair of low-dose radiation damage in human fibroblasts.

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Inducible response required for repair of low-dose radiation damage in human fibroblasts.

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Inducible response required for repair of low-dose radiation damage in human fibroblasts.

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Inducible response required for repair of low-dose radiation damage in human fibroblasts.

@en

Inducible response required for repair of low-dose radiation damage in human fibroblasts.

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PNAS.1002213107

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Inducible response required for repair of low-dose radiation damage in human fibroblasts.

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P2093

Antonia Raths

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Claudia E Rübe

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Markus Löbrich

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Sandro Conrad

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Saskia Grudzenski

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P2860

Computed Tomography — An Increasing Source of Radiation Exposure

NIMA-related protein kinase 1 is involved early in the ionizing radiation-induced DNA damage response

In vivo formation and repair of DNA double-strand breaks after computed tomography examinations

Cancer risks attributable to low doses of ionizing radiation: assessing what we really know

ATM and Artemis promote homologous recombination of radiation-induced DNA double-strand breaks in G2

Evidence for a lack of DNA double-strand break repair in human cells exposed to very low x-ray doses

Megabase chromatin domains involved in DNA double-strand breaks in vivo

Chromosome breakage after G2 checkpoint release

A pathway of double-strand break rejoining dependent upon ATM, Artemis, and proteins locating to gamma-H2AX foci.

Chromosomal stability and the DNA double-stranded break connection

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scholarly article

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