Bone marrow transplantation restores epidermal basement membrane protein expression and rescues epidermolysis bullosa model mice. - Wikidata - wikimedia - TriplyDB

Bone marrow transplantation restores epidermal basement membrane protein expression and rescues epidermolysis bullosa model mice.

Bone marrow transplantation restores epidermal basement membrane protein expression and rescues epidermolysis bullosa model mice.

http://www.wikidata.org/entity/Q34069632

about

Molecular therapeutics for heritable skin diseases Advances in understanding and treating dystrophic epidermolysis bullosa One goal, different strategies--molecular and cellular approaches for the treatment of inherited skin fragility disorders Cell therapy in dermatology.Pharmacological mobilization of endogenous stem cells significantly promotes skin regeneration after full-thickness excision: the synergistic activity of AMD3100 and tacrolimus.Induced pluripotent stem cells in dermatology: potentials, advances, and limitations PDGFRalpha-positive cells in bone marrow are mobilized by high mobility group box 1 (HMGB1) to regenerate injured epithelia.Placenta-based therapies for the treatment of epidermolysis bullosa Novel molecular therapies for heritable skin disorders From marrow to matrix: novel gene and cell therapies for epidermolysis bullosa.Concise review: Transplantation of human hematopoietic cells for extracellular matrix protein deficiency in epidermolysis bullosa.Cell-type-specific differentiation and molecular profiles in skin transplantation: implication of medical approach for genetic skin diseases.Bone marrow transplantation in epidermolysis bullosa.The long and winding road that leads to a cure for epidermolysis bullosa.Next generation human skin constructs as advanced tools for drug development.Revertant mosaicism in genodermatoses.Mesenchymal stem cell therapy in skin: why and what for?Human cord blood-derived unrestricted somatic stem cells promote wound healing and have therapeutic potential for patients with recessive dystrophic epidermolysis bullosa.Current and Future Perspectives of Stem Cell Therapy in Dermatology.CRISPR/Cas9-based genetic correction for recessive dystrophic epidermolysis bullosa.Management of severe epidermolysis bullosa by haematopoietic transplant: principles, perspectives and pitfalls.

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P2860

Bone marrow transplantation restores epidermal basement membrane protein expression and rescues epidermolysis bullosa model mice.

http://www.wikidata.org/entity/Q34069632

description

2010 nî lūn-bûn

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2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2010 թվականի հուլիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Bone marrow transplantation re ...... dermolysis bullosa model mice.

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Bone marrow transplantation re ...... dermolysis bullosa model mice.

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Bone marrow transplantation re ...... dermolysis bullosa model mice.

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Proceedings of the National Academy of Sciences of the United States of America

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Bone marrow transplantation re ...... dermolysis bullosa model mice.

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P2093

Daichi Hoshina

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Daisuke Inokuma

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Daisuke Sawamura

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Hideki Nakamura

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Hiroshi Shimizu

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James R McMillan

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Ken Natsuga

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Masashi Akiyama

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Mikako Sasaki

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Riichiro Abe

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P2860

NOD/SCID/gamma(c)(null) mouse: an excellent recipient mouse model for engraftment of human cells

Bone marrow cells regenerate infarcted myocardium

Mesoangioblast stem cells ameliorate muscle function in dystrophic dogs

The classification of inherited epidermolysis bullosa (EB): Report of the Third International Consensus Meeting on Diagnosis and Classification of EB

Pluripotency of mesenchymal stem cells derived from adult marrow

Neovascularization of ischemic myocardium by human bone-marrow-derived angioblasts prevents cardiomyocyte apoptosis, reduces remodeling and improves cardiac function.

Type XVII collagen gene mutations in junctional epidermolysis bullosa and prospects for gene therapy.

Bone marrow stem cells contribute to repair of the ischemically injured renal tubule

In vivo restoration of laminin 5 beta 3 expression and function in junctional epidermolysis bullosa.

A hypomorphic mouse model of dystrophic epidermolysis bullosa reveals mechanisms of disease and response to fibroblast therapy.

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