"Drinking in the Dark" (DID): a simple mouse model of binge-like alcohol intake
about
Alcohol alters the activation of ERK1/2, a functional regulator of binge alcohol drinking in adult C57BL/6J mice.NPY signaling inhibits extended amygdala CRF neurons to suppress binge alcohol drinking.Chronic ethanol exposure produces time- and brain region-dependent changes in gene coexpression networksAssessment of the Effects of 6 Standard Rodent Diets on Binge-Like and Voluntary Ethanol Consumption in Male C57BL/6J MiceEvidence that Melanocortin Receptor Agonist Melanotan-II Synergistically Augments the Ability of Naltrexone to Blunt Binge-Like Ethanol Intake in Male C57BL/6J MiceLateral hypothalamic melanocortin receptor signaling modulates binge-like ethanol drinking in C57BL/6J miceRodent models and mechanisms of voluntary binge-like ethanol consumption: Examples, opportunities, and strategies for preclinical research.Lateral Hypothalamus GABAergic Neurons Modulate Consummatory Behaviors Regardless of the Caloric Content or Biological Relevance of the Consumed Stimuli.Concomitant Caffeine Increases Binge Consumption of Ethanol in Adolescent and Adult Mice, But Produces Additive Motor Stimulation Only in Adolescent Animals.Cannabinoid CB1 receptor inhibition blunts adolescent-typical increased binge alcohol and sucrose consumption in male C57BL/6J miceGlutamate plasticity woven through the progression to alcohol use disorder: a multi-circuit perspective.Acquisition, Maintenance and Relapse-Like Alcohol Drinking: Lessons from the UChB Rat Line.High Drinking in the Dark (HDID) mice are sensitive to the effects of some clinically relevant drugs to reduce binge-like drinking.Extended Amygdala to Ventral Tegmental Area Corticotropin-Releasing Factor Circuit Controls Binge Ethanol Intake.Chronic pain causes a persistent anxiety state leading to increased ethanol intake in CD1 mice.Genetic and Pharmacologic Manipulation of TLR4 Has Minimal Impact on Ethanol Consumption in Rodents.Circuit and synaptic mechanisms of repeated stress: Perspectives from differing contexts, duration, and development.Cannabinoid-1 receptor neutral antagonist reduces binge-like alcohol consumption and alcohol-induced accumbal dopaminergic signaling.Orexin/Hypocretin System: Role in Food and Drug Overconsumption.Alcohol exposure disrupts mu opioid receptor-mediated long-term depression at insular cortex inputs to dorsolateral striatum.Genetic Contribution to Initial and Progressive Alcohol Intake Among Recombinant Inbred Strains of MicePaternal Preconception Chronic Variable Stress Confers Attenuated Ethanol Drinking Behavior Selectively to Male Offspring in a Pre-Stress Environment Dependent Manner
P2860
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P2860
"Drinking in the Dark" (DID): a simple mouse model of binge-like alcohol intake
description
2014 nî lūn-bûn
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2014 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հուլիսին հրատարակված գիտական հոդված
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2014年の論文
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2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
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name
"Drinking in the Dark" (DID): a simple mouse model of binge-like alcohol intake
@ast
"Drinking in the Dark" (DID): a simple mouse model of binge-like alcohol intake
@en
"Drinking in the Dark"
@nl
type
label
"Drinking in the Dark" (DID): a simple mouse model of binge-like alcohol intake
@ast
"Drinking in the Dark" (DID): a simple mouse model of binge-like alcohol intake
@en
"Drinking in the Dark"
@nl
prefLabel
"Drinking in the Dark" (DID): a simple mouse model of binge-like alcohol intake
@ast
"Drinking in the Dark" (DID): a simple mouse model of binge-like alcohol intake
@en
"Drinking in the Dark"
@nl
P2093
P2860
P1476
"Drinking in the Dark" (DID): a simple mouse model of binge-like alcohol intake
@en
P2093
John C Crabbe
Stephen L Boehm
Todd E Thiele
P2860
P304
P356
10.1002/0471142301.NS0949S68
P407
P577
2014-07-01T00:00:00Z