Gain of 1q and loss of 22 are the most common changes detected by comparative genomic hybridisation in paediatric ependymoma.
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Investigation of chromosome 1q reveals differential expression of members of the S100 family in clinical subgroups of intracranial paediatric ependymomaBiological background of pediatric medulloblastoma and ependymoma: a review from a translational research perspectiveMicroarray analysis of pediatric ependymoma identifies a cluster of 112 candidate genes including four transcripts at 22q12.1-q13.3The Similarities and Differences between Intracranial and Spinal Ependymomas : A Review from a Genetic Research PerspectiveMolecular genetics of ependymomaCortical anaplastic ependymoma with significant desmoplasia: a case report and literature review.Survey of MicroRNA expression in pediatric brain tumors.Molecular characterization of the pediatric preclinical testing panelCurrent management and prognostic factors for adult ependymoma.Genomic imbalances in pediatric intracranial ependymomas define clinically relevant groups.Comparative genomic hybridization in central and peripheral nervous system tumors of childhood and adolescence.Prognosis-related molecular markers in pediatric central nervous system tumors.Advances in the treatment of pediatric brain tumors.Genetic differences on intracranial versus spinal cord ependymal tumors: a meta-analysis of genetic researches.Genetic abnormalities detected in ependymomas by comparative genomic hybridisation.Pediatric CNS tumors: current treatment and future directions.Chromosome 1q gain and tenascin-C expression are candidate markers to define different risk groups in pediatric posterior fossa ependymomaEpendymoma: an update.Chromosomal anomalies and prognostic markers for intracranial and spinal ependymomasUnderstanding Ependymoma Oncogenesis: an Update on Recent Molecular Advances and Current Perspectives.Evaluation of chromosome 1q gain in intracranial ependymomas.Ependymomas: Prognostic Factors and Outcome Analysis in a Retrospective Series of 33 Patients.9p21 and 13q14 dosages in ependymomas. A clinicopathologic study of 101 cases.Structural genomic abnormalities of chromosomes 9 and 18 in myxopapillary ependymomas.
P2860
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P2860
Gain of 1q and loss of 22 are the most common changes detected by comparative genomic hybridisation in paediatric ependymoma.
description
2001 nî lūn-bûn
@nan
2001 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
name
Gain of 1q and loss of 22 are ...... tion in paediatric ependymoma.
@ast
Gain of 1q and loss of 22 are ...... tion in paediatric ependymoma.
@en
Gain of 1q and loss of 22 are ...... tion in paediatric ependymoma.
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type
label
Gain of 1q and loss of 22 are ...... tion in paediatric ependymoma.
@ast
Gain of 1q and loss of 22 are ...... tion in paediatric ependymoma.
@en
Gain of 1q and loss of 22 are ...... tion in paediatric ependymoma.
@nl
prefLabel
Gain of 1q and loss of 22 are ...... tion in paediatric ependymoma.
@ast
Gain of 1q and loss of 22 are ...... tion in paediatric ependymoma.
@en
Gain of 1q and loss of 22 are ...... tion in paediatric ependymoma.
@nl
P2093
P2860
P356
P1476
Gain of 1q and loss of 22 are ...... tion in paediatric ependymoma.
@en
P2093
Darling JL
Harkness W
P2860
P356
10.1002/GCC.1167
P577
2001-09-01T00:00:00Z