RING finger Z protein of lymphocytic choriomeningitis virus (LCMV) inhibits transcription and RNA replication of an LCMV S-segment minigenome.
about
Characterization of the genomic promoter of the prototypic arenavirus lymphocytic choriomeningitis virusArenavirus reverse genetics: new approaches for the investigation of arenavirus biology and development of antiviral strategiesUncovering viral protein-protein interactions and their role in arenavirus life cyclePathogenesis of Lassa feverCrystal structure of the Borna disease virus matrix protein (BDV-M) reveals ssRNA binding propertiesEarly and strong immune responses are associated with control of viral replication and recovery in lassa virus-infected cynomolgus monkeys.Complementarity in the supramolecular design of arenaviruses and retroviruses revealed by electron cryomicroscopy and image analysis.An antiviral disulfide compound blocks interaction between arenavirus Z protein and cellular promyelocytic leukemia proteinDrug discovery technologies and strategies for Machupo virus and other New World arenaviruses.Architecture and regulation of negative-strand viral enzymatic machineryReverse genetics approaches to combat pathogenic arenavirusesMolecular mechanism of arenavirus assembly and budding.Multifunctional nature of the arenavirus RING finger protein Z.ALIX/AIP1 is required for NP incorporation into Mopeia virus Z-induced virus-like particlesRole of the virus nucleoprotein in the regulation of lymphocytic choriomeningitis virus transcription and RNA replication.Altered central nervous system gene expression caused by congenitally acquired persistent infection with lymphocytic choriomeningitis virus.Recombinant lymphocytic choriomeningitis virus expressing vesicular stomatitis virus glycoprotein.Arenavirus Z protein controls viral RNA synthesis by locking a polymerase-promoter complex.Lassa virus.Cells expressing the RING finger Z protein are resistant to arenavirus infection.Tacaribe virus Z protein interacts with the L polymerase protein to inhibit viral RNA synthesis.Minigenomes, transcription and replication competent virus-like particles and beyond: reverse genetics systems for filoviruses and other negative stranded hemorrhagic fever virusesThe small RING finger protein Z drives arenavirus budding: implications for antiviral strategies.Arenavirus Z protein as an antiviral target: virus inactivation and protein oligomerization by zinc finger-reactive compounds.Analysis of Assembly and Budding of Lujo Virus.Arenavirus budding: a common pathway with mechanistic differences.Mapping of the tacaribe arenavirus Z-protein binding sites on the L protein identified both amino acids within the putative polymerase domain and a region at the N terminus of L that are critically involved in binding.The RING domain and the L79 residue of Z protein are involved in both the rescue of nucleocapsids and the incorporation of glycoproteins into infectious chimeric arenavirus-like particlesConstruction of a Sonchus Yellow Net Virus minireplicon: a step toward reverse genetic analysis of plant negative-strand RNA viruses.Activation of the RLR/MAVS Signaling Pathway by the L Protein of Mopeia Virus.Production of CXC and CC chemokines by human antigen-presenting cells in response to Lassa virus or closely related immunogenic viruses, and in cynomolgus monkeys with lassa fever.Rescue of a recombinant Machupo virus from cloned cDNAs and in vivo characterization in interferon (αβ/γ) receptor double knockout miceMyristoylation of the RING finger Z protein is essential for arenavirus buddingLymphocytic choriomeningitis virus (LCMV) infection of macaques: a model for Lassa fever.Inhibition of the type I interferon antiviral response during arenavirus infection.Immune responses and Lassa virus infection.Targeting virulence mechanisms for the prevention and therapy of arenaviral hemorrhagic fever.Human hemorrhagic Fever causing arenaviruses: molecular mechanisms contributing to virus virulence and disease pathogenesis.Absence of an N-Linked Glycosylation Motif in the Glycoprotein of the Live-Attenuated Argentine Hemorrhagic Fever Vaccine, Candid #1, Results in Its Improper Processing, and Reduced Surface Expression.Replicon system for Lassa virus.
P2860
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P2860
RING finger Z protein of lymphocytic choriomeningitis virus (LCMV) inhibits transcription and RNA replication of an LCMV S-segment minigenome.
description
2001 nî lūn-bûn
@nan
2001 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
RING finger Z protein of lymph ...... an LCMV S-segment minigenome.
@ast
RING finger Z protein of lymph ...... an LCMV S-segment minigenome.
@en
RING finger Z protein of lymphocytic choriomeningitis virus
@nl
type
label
RING finger Z protein of lymph ...... an LCMV S-segment minigenome.
@ast
RING finger Z protein of lymph ...... an LCMV S-segment minigenome.
@en
RING finger Z protein of lymphocytic choriomeningitis virus
@nl
prefLabel
RING finger Z protein of lymph ...... an LCMV S-segment minigenome.
@ast
RING finger Z protein of lymph ...... an LCMV S-segment minigenome.
@en
RING finger Z protein of lymphocytic choriomeningitis virus
@nl
P2860
P1433
P1476
RING finger Z protein of lymph ...... an LCMV S-segment minigenome.
@en
P2093
J C de la Torre
P2860
P304
P356
10.1128/JVI.75.19.9415-9426.2001
P407
P577
2001-10-01T00:00:00Z