Increased catecholamine secretion contributes to hypertension in TRPM4-deficient mice. - Wikidata - wikimedia - TriplyDB

Increased catecholamine secretion contributes to hypertension in TRPM4-deficient mice.

Increased catecholamine secretion contributes to hypertension in TRPM4-deficient mice.

http://www.wikidata.org/entity/Q34090105

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Role of K₂p channels in stimulus-secretion coupling TRPM4 channels in smooth muscle function Trpm4 gene invalidation leads to cardiac hypertrophy and electrophysiological alterations Transient receptor potential channels as therapeutic targets Molecular genetics and functional anomalies in a series of 248 Brugada cases with 11 mutations in the TRPM4 channel Transient receptor potential melastatin 4 channel controls calcium signals and dental follicle stem cell differentiation Mechanical activation of angiotensin II type 1 receptors causes actin remodelling and myogenic responsiveness in skeletal muscle arterioles.Disruption of vascular Ca2+-activated chloride currents lowers blood pressure.Transient receptor potential (TRP) channels: a clinical perspective A novel mechanism of angiotensin II-regulated placental vascular tone in the development of hypertension in preeclampsia.Vascular TRP channels: performing under pressure and going with the flow Role of TRP channels in the cardiovascular system.The Ca(2+)-activated cation channel TRPM4 is a negative regulator of angiotensin II-induced cardiac hypertrophy.Transient receptor potential channels in the vasculature.Transient receptor potential melastatin 4 and cell death.Regulation of cerebral artery smooth muscle membrane potential by Ca²⁺-activated cation channels.Neuropeptide y gates a stress-induced, long-lasting plasticity in the sympathetic nervous system.Ion channel-kinase TRPM7 is required for maintaining cardiac automaticity.Mechanobiology in cardiac physiology and diseases.Glycosylation of TRPM4 and TRPM5 channels: molecular determinants and functional aspects.The Pathophysiologic Roles of TRPM7 Channel.TRPM2 channels protect against cardiac ischemia-reperfusion injury: role of mitochondria.Emerging concepts for the role of TRP channels in the cardiovascular system.Cell signaling of angiotensin II on vascular tone: novel mechanisms.TRPM channels: same ballpark, different players, and different rules in immunogenetics.Disorders of blood pressure regulation-role of catecholamine biosynthesis, release, and metabolism.TRP channels.Transient receptor potential melastatin 4 channel contributes to migration of androgen-insensitive prostate cancer cells TRPM4 in cardiac electrical activity.The role of transient receptor potential channels in hypertension and metabolic vascular damage.Low pHo boosts burst firing and catecholamine release by blocking TASK-1 and BK channels while preserving Cav1 channels in mouse chromaffin cells.Smooth Muscle Ion Channels and Regulation of Vascular Tone in Resistance Arteries and Arterioles.The Role of TRP Proteins in Mast Cells.Timing of myocardial trpm7 deletion during cardiogenesis variably disrupts adult ventricular function, conduction, and repolarization.TRPM4 inhibition promotes angiogenesis after ischemic stroke.Methods for Evaluation of Vascular Endothelial Cell Function with Transient Receptor Potential (TRP) Channel Drugs.Structure of full-length human TRPM4.TRPM4 cation channel mediates axonal and neuronal degeneration in experimental autoimmune encephalomyelitis and multiple sclerosis.Store-Operated Ca2+ Entry as a Prostate Cancer Biomarker — a Riddle with Perspectives.Role of the TRPM4 Channel in Cardiovascular Physiology and Pathophysiology.

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P2860

Increased catecholamine secretion contributes to hypertension in TRPM4-deficient mice.

http://www.wikidata.org/entity/Q34090105

description

2010 nî lūn-bûn

@nan

2010 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Increased catecholamine secretion contributes to hypertension in TRPM4-deficient mice.

@ast

Increased catecholamine secretion contributes to hypertension in TRPM4-deficient mice.

@en

Increased catecholamine secretion contributes to hypertension in TRPM4-deficient mice.

@nl

type

label

Increased catecholamine secretion contributes to hypertension in TRPM4-deficient mice.

@ast

Increased catecholamine secretion contributes to hypertension in TRPM4-deficient mice.

@en

Increased catecholamine secretion contributes to hypertension in TRPM4-deficient mice.

@nl

prefLabel

Increased catecholamine secretion contributes to hypertension in TRPM4-deficient mice.

@ast

Increased catecholamine secretion contributes to hypertension in TRPM4-deficient mice.

@en

Increased catecholamine secretion contributes to hypertension in TRPM4-deficient mice.

@nl

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Journal of Clinical Investigation

P1476

Increased catecholamine secretion contributes to hypertension in TRPM4-deficient mice.

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P2093

An van den Bergh

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Björn Hummel

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Frank Schweda

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Frieder Kees

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Gerry Van der Mieren

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Ilka Mathar

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Juan E Camacho Londoño

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Marc Freichel

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Marcel Meissner

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Sebastian Uhl

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P2860

Intracellular Ca2+ and the phospholipid PIP2 regulate the taste transduction ion channel TRPM5

Impaired endocytosis of the ion channel TRPM4 is associated with human progressive familial heart block type I

Regulation of melastatin, a TRP-related protein, through interaction with a cytoplasmic isoform

Genome-wide atlas of gene expression in the adult mouse brain

The TRP channels, a remarkably functional family

Transient receptor potential cation channels in disease

The TRPM7 ion channel functions in cholinergic synaptic vesicles and affects transmitter release

Critical role for transient receptor potential channel TRPM4 in myogenic constriction of cerebral arteries

Voltage dependence of the Ca2+-activated cation channel TRPM4

Connexin40 is essential for the pressure control of renin synthesis and secretion

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3267-3279

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scholarly article

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10.1172/JCI41348

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9

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120

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2010-08-02T00:00:00Z

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20679729

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2929713

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