Nucleotide-binding oligomerization domain-2 inhibits toll-like receptor-4 signaling in the intestinal epithelium. - Wikidata - wikimedia - TriplyDB

Nucleotide-binding oligomerization domain-2 inhibits toll-like receptor-4 signaling in the intestinal epithelium.

Nucleotide-binding oligomerization domain-2 inhibits toll-like receptor-4 signaling in the intestinal epithelium.

http://www.wikidata.org/entity/Q34091864

about

Necrotizing enterocolitis: new insights into pathogenesis and mechanisms Discovery and validation of a new class of small molecule Toll-like receptor 4 (TLR4) inhibitors Epithelial cell-specific MyD88 signaling mediates ischemia/reperfusion-induced intestinal injury independent of microbial status.Animal models of gastrointestinal and liver diseases. Animal models of necrotizing enterocolitis: pathophysiology, translational relevance, and challenges.Lactobacillus reuteri DSM 17938 differentially modulates effector memory T cells and Foxp3+ regulatory T cells in a mouse model of necrotizing enterocolitis.Evidence-based feeding strategies before and after the development of necrotizing enterocolitis.Update in pathogenesis and prospective in treatment of necrotizing enterocolitis.Innate immunity in the small intestine.Antibiotics increase gut metabolism and antioxidant proteins and decrease acute phase response and necrotizing enterocolitis in preterm neonates.The microbiota protects against ischemia/reperfusion-induced intestinal injury through nucleotide-binding oligomerization domain-containing protein 2 (NOD2) signaling.Worms, flies and four-legged friends: the applicability of biological models to the understanding of intestinal inflammatory diseases.New insights into the pathogenesis and treatment of necrotizing enterocolitis: Toll-like receptors and beyond NOD2 deficiency results in increased susceptibility to peptidoglycan-induced uveitis in mice Salmonella enterica serovar Typhimurium ΔmsbB triggers exacerbated inflammation in Nod2 deficient mice.Toll-like receptor-4 in human and mouse colonic epithelium is developmentally regulated: a possible role in necrotizing enterocolitis.Intracellular heat shock protein-70 negatively regulates TLR4 signaling in the newborn intestinal epithelium.Breast milk protects against the development of necrotizing enterocolitis through inhibition of Toll-like receptor 4 in the intestinal epithelium via activation of the epidermal growth factor receptor Amniotic fluid inhibits Toll-like receptor 4 signaling in the fetal and neonatal intestinal epithelium.Toll-like receptor 4 is expressed on intestinal stem cells and regulates their proliferation and apoptosis via the p53 up-regulated modulator of apoptosis Intestinal epithelial Toll-like receptor 4 regulates goblet cell development and is required for necrotizing enterocolitis in mice.A critical role for TLR4 induction of autophagy in the regulation of enterocyte migration and the pathogenesis of necrotizing enterocolitis Mechanisms of gut barrier failure in the pathogenesis of necrotizing enterocolitis: Toll-like receptors throw the switch.Mapping the New World of Necrotizing Enterocolitis (NEC): Review and Opinion.Innate immune signaling in the pathogenesis of necrotizing enterocolitis.Guts, germs and glucose: understanding the effects of prematurity on the interaction between bacteria and nutrient absorption across the intestine The immunological landscape in necrotising enterocolitis The Role of Mucosal Immunity in the Pathogenesis of Necrotizing Enterocolitis.Toll-like receptor 4-mediated endoplasmic reticulum stress in intestinal crypts induces necrotizing enterocolitis.The role of innate immune-stimulated epithelial apoptosis during gastrointestinal inflammatory diseases.Innate Immunity in the Small Intestine of the Preterm Infant.Progress in the field of necrotising enterocolitis--year 2012.Mitochondria: sensors and mediators of innate immune receptor signaling.Toll-like receptor regulation of intestinal development and inflammation in the pathogenesis of necrotizing enterocolitis.Intestinal proteomics in pig models of necrotising enterocolitis, short bowel syndrome and intrauterine growth restriction.Pathogenesis of necrotizing enterocolitis: modeling the innate immune response.Inflammation and Apoptosis: Dual Mediator Role for Toll-like Receptor 4 in the Development of Necrotizing Enterocolitis.LPS Induces Hyper-Permeability of Intestinal Epithelial Cells.Critical role of myeloid differentiation factor 88 in necrotizing enterocolitis.Pathogenesis of NEC: Role of the innate and adaptive immune response.Nucleotide-binding oligomerization domain 2 (Nod2) is dispensable for the innate immune responses of macrophages against Yersinia enterocolitica.

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P2860

Nucleotide-binding oligomerization domain-2 inhibits toll-like receptor-4 signaling in the intestinal epithelium.

http://www.wikidata.org/entity/Q34091864

description

2010 nî lūn-bûn

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2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2010 թվականի մայիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Nucleotide-binding oligomeriza ...... in the intestinal epithelium.

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Nucleotide-binding oligomeriza ...... in the intestinal epithelium.

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Nucleotide-binding oligomeriza ...... in the intestinal epithelium.

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Nucleotide-binding oligomeriza ...... in the intestinal epithelium.

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Nucleotide-binding oligomeriza ...... in the intestinal epithelium.

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Nucleotide-binding oligomeriza ...... in the intestinal epithelium.

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Nucleotide-binding oligomeriza ...... in the intestinal epithelium.

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J.GASTRO.2010.05.038

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Gastroenterology

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Nucleotide-binding oligomeriza ...... in the intestinal epithelium.

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Amin Afrazi

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Anthony Russo

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Chhinder P Sodhi

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Congrong Ma

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David J Hackam

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John Ozolek

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Maria Branca

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Matthew D Neal

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Richard H Siggers

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Shipan Dai

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P2860

A frameshift mutation in NOD2 associated with susceptibility to Crohn's disease

Inflammasome components NALP 1 and 3 show distinct but separate expression profiles in human tissues suggesting a site-specific role in the inflammatory response

Function of Nod-like receptors in microbial recognition and host defense

Association of NOD2 leucine-rich repeat variants with susceptibility to Crohn's disease

Smac, a mitochondrial protein that promotes cytochrome c-dependent caspase activation by eliminating IAP inhibition

The roles of bacteria and TLR4 in rat and murine models of necrotizing enterocolitis

Toll-like receptor-4 inhibits enterocyte proliferation via impaired beta-catenin signaling in necrotizing enterocolitis.

Extracellular high mobility group box-1 (HMGB1) inhibits enterocyte migration via activation of Toll-like receptor-4 and increased cell-matrix adhesiveness

Disordered enterocyte signaling and intestinal barrier dysfunction in the pathogenesis of necrotizing enterocolitis.

Muramyl dipeptide activation of nucleotide-binding oligomerization domain 2 protects mice from experimental colitis.

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904-17, 917.e1-6

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10.1053/J.GASTRO.2010.05.038

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20580721

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Toll-like receptor

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