Mitochondrial uncoupling protein-2 protects the immature brain from excitotoxic neuronal death. - Wikidata - wikimedia - TriplyDB

Mitochondrial uncoupling protein-2 protects the immature brain from excitotoxic neuronal death.

Mitochondrial uncoupling protein-2 protects the immature brain from excitotoxic neuronal death.

http://www.wikidata.org/entity/Q34094796

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G2019S leucine-rich repeat kinase 2 causes uncoupling protein-mediated mitochondrial depolarization Seizure-induced oxidative stress in temporal lobe epilepsy Effects of age, dietary, and behavioral enrichment on brain mitochondria in a canine model of human aging Mechanisms of Action of Antiseizure Drugs and the Ketogenic Diet Changes in components of energy regulation in mouse cortex with increases in wakefulness UCP2 induced by natural birth regulates neuronal differentiation of the hippocampus and related adult behavior Cyanide-induced death of dopaminergic cells is mediated by uncoupling protein-2 up-regulation and reduced Bcl-2 expression Role of oxidative stress in epileptic seizures.Antioxidant treatment with coenzyme Q-ter in prevention of gentamycin ototoxicity in an animal model.Brain-derived neurotrophic factor functions as a metabotrophin to mediate the effects of exercise on cognition.Differential cerebral cortex transcriptomes of baboon neonates consuming moderate and high docosahexaenoic acid formulas.Overexpression of UCP2 protects thalamic neurons following global ischemia in the mouse.The ketogenic diet in a pill: is this possible?Dietary curcumin supplementation counteracts reduction in levels of molecules involved in energy homeostasis after brain trauma Degradation of an intramitochondrial protein by the cytosolic proteasome Mitochondrial Uncoupler Prodrug of 2,4-Dinitrophenol, MP201, Prevents Neuronal Damage and Preserves Vision in Experimental Optic Neuritis Acetyl-L-carnitine ameliorates mitochondrial dysfunction following contusion spinal cord injury.Quantification of uncoupling protein 2 reveals its main expression in immune cells and selective up-regulation during T-cell proliferation Cerebrospinal fluid ceramides from patients with multiple sclerosis impair neuronal bioenergetics.The mitochondrial uncoupler 2,4-dinitrophenol attenuates tissue damage and improves mitochondrial homeostasis following transient focal cerebral ischemia.Mitochondrial protection after traumatic brain injury by scavenging lipid peroxyl radicals.Serial MRI after experimental febrile seizures: altered T2 signal without neuronal death Uncoupling proteins, dietary fat and the metabolic syndrome Uncoupling protein 2 and 4 expression pattern during stem cell differentiation provides new insight into their putative function.Aging-related gene expression in hippocampus proper compared with dentate gyrus is selectively associated with metabolic syndrome variables in rhesus monkeys.Post-injury administration of the mitochondrial permeability transition pore inhibitor, NIM811, is neuroprotective and improves cognition after traumatic brain injury in rats A/ASP/VAL allele combination of IGF1R, IRS2, and UCP2 genes is associated with better metabolic profile, preserved energy expenditure parameters, and low mortality rate in longevity.Traumatic brain injury and trichloroethylene exposure interact and produce functional, histological, and mitochondrial deficits Susceptibility to a parkinsonian toxin varies during primate development Ketogenic diets, mitochondria, and neurological diseases.Peroxisome proliferator-activated receptors γ/mitochondrial uncoupling protein 2 signaling protects against seizure-induced neuronal cell death in the hippocampus following experimental status epilepticus.Toward Understanding How Early-Life Stress Reprograms Cognitive and Emotional Brain Networks.Orexin Receptor Antagonism Improves Sleep and Reduces Seizures in Kcna1-null Mice.Trichloroethylene induces dopaminergic neurodegeneration in Fisher 344 rats Analysis of regional brain mitochondrial bioenergetics and susceptibility to mitochondrial inhibition utilizing a microplate based system Human milk cortisol is associated with infant temperament.The brain, seizures and epilepsy throughout life: understanding a moving target.The ketogenic diet and brain metabolism of amino acids: relationship to the anticonvulsant effect Vitamins and nutrients as primary treatments in experimental brain injury: Clinical implications for nutraceutical therapies.Lower respiratory capacity in extraocular muscle mitochondria: evidence for intrinsic differences in mitochondrial composition and function.

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Mitochondrial uncoupling protein-2 protects the immature brain from excitotoxic neuronal death.

http://www.wikidata.org/entity/Q34094796

description

2003 nî lūn-bûn

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2003 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2003年の論文

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Mitochondrial uncoupling prote ...... om excitotoxic neuronal death.

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Mitochondrial uncoupling prote ...... om excitotoxic neuronal death.

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Mitochondrial uncoupling prote ...... om excitotoxic neuronal death.

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Mitochondrial uncoupling prote ...... om excitotoxic neuronal death.

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Mitochondrial uncoupling prote ...... om excitotoxic neuronal death.

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Annals of Neurology

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Mitochondrial uncoupling prote ...... rom excitotoxic neuronal death

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Celine Dubé

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Kristina Dorenbos

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Patrick G Sullivan

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Tallie Z Baram

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P2860

Disruption of the uncoupling protein-2 gene in mice reveals a role in immunity and reactive oxygen species production

Energy metabolism in uncoupling protein 3 gene knockout mice

Calcium-induced cytochrome c release from CNS mitochondria is associated with the permeability transition and rupture of the outer membrane

Superoxide activates mitochondrial uncoupling proteins

Repeated brief seizures induce progressive hippocampal neuron loss and memory deficits

Novel and transient populations of corticotropin-releasing hormone-expressing neurons in developing hippocampus suggest unique functional roles: a quantitative spatiotemporal analysis

Correlation between hypermetabolism and neuronal damage during status epilepticus induced by lithium and pilocarpine in immature and adult rats

Mitochondrial disease in superoxide dismutase 2 mutant mice

Mechanism of uncoupling protein action

Genetic determinants of susceptibility to excitotoxic cell death: implications for gene targeting approaches.

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10.1002/ANA.10543

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6

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53

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10729136

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12783416

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2930774

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