Non-classical proIL-1beta activation during mammary gland infection is pathogen-dependent but caspase-1 independent. - Wikidata - wikimedia - TriplyDB

Non-classical proIL-1beta activation during mammary gland infection is pathogen-dependent but caspase-1 independent.

Non-classical proIL-1beta activation during mammary gland infection is pathogen-dependent but caspase-1 independent.

http://www.wikidata.org/entity/Q34097291

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Mouse models of mastitis - how physiological are they?CD36/SR-B2-TLR2 Dependent Pathways Enhance Porphyromonas gingivalis Mediated Atherosclerosis in the Ldlr KO Mouse Model.Pseudomonas aeruginosa Triggers Macrophage Autophagy To Escape Intracellular Killing by Activation of the NLRP3 Inflammasome.Anti-Inflammatory and Antimicrobial Effects of Estradiol in Bovine Mammary Epithelial Cells during Staphylococcus aureus Internalization.Preconditioning with Lipopolysaccharide or Lipoteichoic Acid Protects against Staphylococcus aureus Mammary Infection in Mice.The Quorum Sensing Inhibitor Hamamelitannin Increases Antibiotic Susceptibility of Staphylococcus aureus Biofilms by Affecting Peptidoglycan Biosynthesis and eDNA Release.Efficacy and Safety of a Bovine-Associated Staphylococcus aureus Phage Cocktail in a Murine Model of Mastitis.Immunomodulation of Host Chitinase 3-Like 1 During a Mammary Pathogenic Escherichia coli Infection.Extracellular Vesicles Elicit an Immunostimulatory Response on the Murine Mammary Gland Anti-inflammatory signaling by mammary tumor cells mediates prometastatic macrophage polarization in an innovative intraductal mouse model for triple-negative breast cancer

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P2860

Non-classical proIL-1beta activation during mammary gland infection is pathogen-dependent but caspase-1 independent.

http://www.wikidata.org/entity/Q34097291

description

2014 nî lūn-bûn

@nan

2014 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի օգոստոսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Non-classical proIL-1beta acti ...... ent but caspase-1 independent.

@ast

Non-classical proIL-1beta acti ...... ent but caspase-1 independent.

@en

Non-classical proIL-1beta acti ...... ent but caspase-1 independent.

@nl

type

label

Non-classical proIL-1beta acti ...... ent but caspase-1 independent.

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Non-classical proIL-1beta acti ...... ent but caspase-1 independent.

@en

Non-classical proIL-1beta acti ...... ent but caspase-1 independent.

@nl

prefLabel

Non-classical proIL-1beta acti ...... ent but caspase-1 independent.

@ast

Non-classical proIL-1beta acti ...... ent but caspase-1 independent.

@en

Non-classical proIL-1beta acti ...... ent but caspase-1 independent.

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JOURNAL.PONE.0105680

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Non-classical proIL-1beta acti ...... ent but caspase-1 independent.

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Dieter Demon

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Evelyne Meyer

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Harald Carlsen

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Koen Breyne

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Kristel Demeyere

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Niek N Sanders

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Steven K Cool

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Wim Van Den Broeck

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P2860

Staphylococcus aureus α-hemolysin mediates virulence in a murine model of severe pneumonia through activation of the NLRP3 inflammasome

Epithelial cells as phagocytes: apoptotic epithelial cells are engulfed by mammary alveolar epithelial cells and repress inflammatory mediator release

Escherichia coli and Staphylococcus aureus elicit differential innate immune responses following intramammary infection.

Essential role of neutrophils but not mammary alveolar macrophages in a murine model of acute Escherichia coli mastitis.

Cells and cytokines in inflammatory secretions of bovine mammary gland.

Muramyl dipeptide synergizes with Staphylococcus aureus lipoteichoic acid to recruit neutrophils in the mammary gland and to stimulate mammary epithelial cells.

Comparative kinetics of Escherichia coli- and Staphylococcus aureus-specific activation of key immune pathways in mammary epithelial cells demonstrates that S. aureus elicits a delayed response dominated by interleukin-6 (IL-6) but not by IL-1A or t

Escherichia coli- and Staphylococcus aureus-induced mastitis differentially modulate transcriptional responses in neighbouring uninfected bovine mammary gland quarters.

The interferon-gamma-induced GTPase, mGBP-2, inhibits tumor necrosis factor alpha (TNF-alpha) induction of matrix metalloproteinase-9 (MMP-9) by inhibiting NF-kappaB and Rac protein.

Comparative expression profiling of E. coli and S. aureus inoculated primary mammary gland cells sampled from cows with different genetic predispositions for somatic cell score.

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e105680

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scholarly article

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10.1371/JOURNAL.PONE.0105680

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8

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9

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Peter Vandenabeele

Tom Vanden Berghe

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2014-08-27T00:00:00Z

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265095072

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25162221

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4146512

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