Increased cell proliferation, but not reduced cell death, induces lymphocytosis in bovine leukemia virus-infected sheep - Wikidata - wikimedia - TriplyDB

Increased cell proliferation, but not reduced cell death, induces lymphocytosis in bovine leukemia virus-infected sheep

Increased cell proliferation, but not reduced cell death, induces lymphocytosis in bovine leukemia virus-infected sheep

http://www.wikidata.org/entity/Q34098939

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Mechanisms of leukemogenesis induced by bovine leukemia virus: prospects for novel anti-retroviral therapies in human Reduced proviral loads during primo-infection of sheep by Bovine Leukemia virus attenuated mutants Recent Advances in BLV Research Modes of Human T Cell Leukemia Virus Type 1 Transmission, Replication and Persistence Vaccination against δ-retroviruses: the bovine leukemia virus paradigm A mechanistic model for bromodeoxyuridine dilution naturally explains labelling data of self-renewing T cell populations.Earlier onset of delta-retrovirus-induced leukemia after splenectomy Memory CD4 T cell subsets are kinetically heterogeneous and replenished from naive T cells at high levels.Reduced cell turnover in bovine leukemia virus-infected, persistently lymphocytotic cattle Naïve T-cell dynamics in human immunodeficiency virus type 1 infection: effects of highly active antiretroviral therapy provide insights into the mechanisms of naive T-cell depletion.Peripheral blood B-cell death compensates for excessive proliferation in lymphoid tissues and maintains homeostasis in bovine leukemia virus-infected sheep Spleen-dependent turnover of CD11b peripheral blood B lymphocytes in bovine leukemia virus-infected sheep.Quantification of T-cell dynamics: from telomeres to DNA labeling.Quantifying T lymphocyte turnover.Reduced levels of reactive oxygen species correlate with inhibition of apoptosis, rise in thioredoxin expression and increased bovine leukemia virus proviral loads.Animal models on HTLV-1 and related viruses: what did we learn?Interaction between Bovine leukemia virus (BLV) infection and age on telomerase misregulation.Reduced cell turnover in lymphocytic monkeys infected by human T-lymphotropic virus type 1.How Does HTLV-1 Undergo Oncogene-Dependent Replication Despite a Strong Immune Response?Quantifying lymphocyte kinetics in vivo using carboxyfluorescein diacetate succinimidyl ester (CFSE).

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Increased cell proliferation, but not reduced cell death, induces lymphocytosis in bovine leukemia virus-infected sheep

http://www.wikidata.org/entity/Q34098939

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2002 nî lūn-bûn

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2002 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2002 թվականի հուլիսին հրատարակված գիտական հոդված

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2002年の論文

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2002年論文

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2002年論文

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2002年論文

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Increased cell proliferation, ...... leukemia virus-infected sheep

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Increased cell proliferation, ...... leukemia virus-infected sheep

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Increased cell proliferation, ...... leukemia virus-infected sheep

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Increased cell proliferation, ...... leukemia virus-infected sheep

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Increased cell proliferation, ...... leukemia virus-infected sheep

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Increased cell proliferation, ...... leukemia virus-infected sheep

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Increased cell proliferation, ...... leukemia virus-infected sheep

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Increased cell proliferation, ...... leukemia virus-infected sheep

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Proceedings of the National Academy of Sciences of the United States of America

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Increased cell proliferation, ...... leukemia virus-infected sheep

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Christophe Debacq

http://www.w3.org/2001/XMLSchema#string

Daniel Portetelle

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Luc Willems

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Pierre Kerkhofs

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Richard Kettmann

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P2860

Human adult T-cell leukemia virus: molecular cloning of the provirus DNA and the unique terminal structure

Regulation of apoptosis by viral gene products

Discordance between bovine leukemia virus tax immortalization in vitro and oncogenicity in vivo.

Bovine leukemia virus-induced persistent lymphocytosis in cattle does not correlate with increased ex vivo survival of B lymphocytes

Measurement of T-cell kinetics: recent methodologic advances.

Human T-cell leukemia virus type 1 Tax and cell cycle progression: role of cyclin D-cdk and p110Rb.

Bovine leukemia virus-induced lymphocytosis and increased cell survival mainly involve the CD11b+ B-lymphocyte subset in sheep.

Suboptimal enhancer sequences are required for efficient bovine leukemia virus propagation in vivo: implications for viral latency

Apoptosis and the pathogenesis of lymphoma.

HTLV-1 infections

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10048-10053

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scholarly article

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10.1073/PNAS.142100999

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15

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99

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